心肌病的

Objective To study the surgical treatment of the acromegalic cardiomyopathy.

目的探讨肢端肥大症性心肌病的神经外科手术治疗和疗效。

Objective To explore etiology and pathogenesis of viral myocarditis and dilated cardiomyopathy.

目的 探讨病毒性心肌炎和扩张性心肌病的发病机制及相互关系。

the upregulation of tsp ⅰ in the left ventricle myocardium in diabetic model may relate to pathologic changes of diabetic heart disease, such as hypertrophy, fibrosis of myocardial cell and microvessel dissepiment thickening.

tsp ⅰ可能参与了心肌细胞肥大和纤维化以及心肌微血管病变等糖尿病心肌病的特征性病理改变。

Background— Impairment of intracellular Ca2+ homeostasis and mitochondrial function has been implicated in the development of cardiomyopathy.

背景：细胞内Ca2+稳态失衡和线粒体功能受损与心肌病的发展有关。

Objective: To explore the pathogenic mechanism of diabetic cardiomyopathy.

目的：探讨糖尿病性心肌病的发病机制。

We speculated that mutations in sarcomere protein genes known to cause hypertrophic cardiomyopathy and dilated cardiomyopathy may be associated with left ventricular noncompaction.

我们推测：已知的可导致肥厚性心肌病和扩张性心肌病的肌节蛋白基因突变，可能和左室心肌致密化不全相关。

Background: Left ventricular noncompaction constitutes a primary cardiomyopathy characterized by a severely thickened, 2-layered myocardium, numerous prominent trabeculations, and deep intertrabecular recesses.

背景：左室心肌致密化不全，原发性心肌病的一种类型，以&心肌严重增厚成双层状、异常粗大肌小梁多见、小梁间隐窝深陷&为特征。

Background— The hallmarks of diabetic cardiomyopathy are cardiac oxidative stress, intramyocardial inflammation, cardiac fibrosis, and cardiac apoptosis.

背景——糖尿病性心肌病的标志是心脏氧化应激、心肌内炎症、心脏纤维化、心脏凋亡。

objective trimetazidine has shown anti-ischemic properties to improve exercise tolerance without haemodynamic effects and direct cyto protective effect on the myocardium.however,it is not clear whether trimetazidine has beneficially effect on severe ischemic cardiomyopathy.methods 19patients（68±9）years with postnecrotic left ventricular dysfunction（ejection fraction31.7%±5.4%）were studied to examine the benefit of adding60mg a day of trimetazidine for3months to the classical therapy.the assessment included clinical evaluation,electrocardiography and echocardiography.results after adding60mg a day of trimetazidine for3months,attack of angina was relieved and dyspnea was improved（p.05）.but heart rate,systolic blood pressure,rate pressure product,ejection fraction,left ventricular volume and global wall motion index were not changed.conclusion trimetazidine seems to improve clinical status while can not change oxygen supply-to-demand ratio without adverse effects in patients on severe ischemic carˉdiomyopathy.

目的 曲美他嗪是一种新型的抗心肌缺血的药物，能够改善患者的运动耐量和心肌的直接保护作用同时并没有血流动力学效应。但目前对于严重缺血性心肌病患者的临床治疗价值还需要进一步观察。方法 19例（68±9）岁确诊为缺血性心肌病（左心室射血分数0%）的患者在常规治疗的基础上加用每日60mg曲美他嗪治疗3个月，随诊观察患者临床情况以及超声心动图指标的变化。结果 19例患者均可耐受在常规治疗基础上加用每日60mg曲美他嗪的治疗，随诊发现患者心绞痛发作明显减少，运动耐量明显改善。同时患者心率、收缩压、心率血压乘积、左心室射血分数、左心室容量以及整体室壁运动指数无明显改变。结论缺血性心肌病的患者在常规治疗基础上加用曲美他嗪是安全的，同时其能明显改善患者的临床状态，而这种作用并不改变心肌氧的供求比值。

Conclusions— Among healthy relaties of DCM patients, AHAs are independent predictors of disease deelopment within 5 years.

扩张性心肌病患者健康的亲属中心脏自身抗体阳性是预测未来5年内发展为扩张性心肌病的独立预测因子。

Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气，收缩臀部肌肉；拱起身体，尽量抬起头来，右腿伸直朝向天花板（膝微屈，以避免肌肉紧张）。

The cost of moving grain food products was unchanged from May, but year over year are up 8%.

粮食产品的运输费用与5月份相比没有变化，但却比去年同期高8％。