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心肌梗死

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Methods Male Sprague Dawley rats were induced by ligating the anterior descending coronary artery and were divided into the 2nd, 4th, 8th, 12th and 21st week groups. Elastic fibers were evaluated in the infarct region using Masson trichrome staining, Uana orcein staining and image analysis. The expressions of TNFα and IL-1β were also detected by immunohistochemistry.

SD大鼠冠状动脉前降支结扎形成心肌梗死模型,于术后2、4、8、12、21周时,用Masson染色、Uana地衣红染色技术和免疫组织化学方法,分别观察梗死区弹性纤维和白细胞介素1β(IL-1β)、肿瘤坏死因子的变化,计算机图像分析系统作半定量分析。

Methods Within 2 weeks after AMI, 35 patients underwent ultasonic tissue characterization from the parasternal papillary short-axis view.

目的 探讨心肌组织背向散射积分和回声强度是否可用于临床急性心肌梗死的诊断及其应用价值。

That scar formation and cardiomyocytes loss result from myocardial infarction is an important cause affecting heart function, and is also the pathobiological basis leading to heart failure and even death.

心肌梗死(myocardial infarction,MI)引起的瘢痕形成和心肌细胞减少是影响心功能的重要因素,也是导致心力衰竭乃至死亡的病理基础。

To the clinical medicine, it is a great development that the acute myocar- dial infarct patients have been cured by coronary artery repatency, thromboclasis, precutaneous transluminal coronary angioplasty and other heart surgery in the last years. The myocardial tissue receives blood reperfusion again due to cardiovascular repatency or reconstructive operation, which can really obtain favorable therapeutic efficacy in multitudinous conditions.

急性心肌梗死病人采用冠脉再通术、溶栓治疗以及其他心脏外科手术是近年来临床医学的一大重要进展,因为缺血后疏通血管或再造血管使心肌组织得到血液的再灌注,在多数情况下确能收到良好的治疗效果。

Methods: Sixscore patients with AMI were divided into control groupand experimental group.All patients recive identifical ba-sal treatment.The patients in received a total of 30ml/d of danhong injecta,change of enzymology in cardiac muscle and percentage of complications were obeserve in 14th,death rate was obeserve too.

120例急性心肌梗死患者随机分为对照组和治疗组,两组给予相同的吸氧,扩冠,抗凝,溶栓,镇静等基础治疗,治疗组给予丹红注射液30 ml/d,观察两组患者治疗2周后的心肌酶谱变化情况,并发症发生率及死亡率。

PET slightly overestimate extent of necrosis myocardium and can't distinguish transmural necrosis from subendocardial necrosis.

PET高估心肌坏死范围,且不能判断心肌梗死的透壁程度。

PET has low spatial resolution which ca not distinguish transmural necrosis from subendocardial necrosis and can't demonstrate wall motion of left ventricular directly.

心肌PET显像空间分辨率低,无法显示心肌梗死的透壁程度,且不能直观显示室壁运动情况。

The myocardium beneath the endocardial surface at the top demonstrates pale fibrosis with collagenization following healing of a subendocardial myocardial infarction.

在心肌层下表面上覆盖一层灰白色胶原纤维,这显示心内膜下心肌梗死愈合。

AIM To investigate the effect of autonomic nerves on the transmural dispersion of effective refarctory period upon acute myocardium infarction in vivo.

目的 为探讨自主神经系统对在体家兔急性心肌梗死后跨室壁3层心肌细胞有效不应期离散度的影响。

OBJECTIVE: The attaching and effacing pathogens enterohemorrhagic E.

间充质干细胞和生长因子混合预处理可加强缝隙连接形成,心肌细胞保护作用和心肌梗死的疗效

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呼气,收缩臀部肌肉;拱起身体,尽量抬起头来,右腿伸直朝向天花板(膝微屈,以避免肌肉紧张)。

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