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AIM: To compare the sensitivity of all maps in body surface potential mapping and electrocardiogram for myocardial infarction. A comparative study was made of departure maps, peak potential map, isochrone map, isointegral map in BSPM and ECG in 42 patients with MI using a BSPM-Ⅲmodel computer system. RESULTS: All maps of BSPM in patients with anterior and inferior MI were appeared typical abnormal feature.

目的:比较体表电位标测各种图形与心电图诊断心肌梗死的敏感性,方法采用BSPM-Ⅲ型体表电位标测微机系统,对42例MI 患者和健康人的体表差电位图、峰电位图、等时间图、等积分图及ECG进行了对比研究。

Diagnostic value of departure maps for myocardial infarction;2. A comparative study was made of departure maps , peak potential map, isochrone map, isointegral map in BSPM and ECG in 42 patients with MI using a BSPM Ⅲmodel computer system.

目的 :比较体表电位标测各种图形与心电图诊断心肌梗死的敏感性,方法采用 BSPM-型体表电位标测微机系统,对 42例 MI患者和健康人的体表差电位图、峰电位图、等时间图、等积分图及 ECG进行了对比研究。

A comparative study was made of departure maps, peak potential map, isochrone map, isointegral map in BSPM and ECG in 42 patients with MI using a BSPM-Ⅲmodel computer system. RESULTS: All maps of BSPM in patients with anterior and inferior MI were appeared typical abnormal feature.

目的:比较体表电位标测各种图形与心电图诊断心肌梗死的敏感性,方法采用BSPM-Ⅲ型体表电位标测微机系统,对42例MI 患者和健康人的体表差电位图、峰电位图、等时间图、等积分图及ECG进行了对比研究。

Methods48 Wistar male rats were randomly divided into control, I/R group, high-, medium- and low-dose PLF groups, and verapamil group. The isolated perfused rat hearts were set up by Langendorff-perfusion system.

方法48只Wistar雄性大鼠随机分为正常对照组,心肌缺血再灌注组,PLF高、中、低剂量组,阳性药对照组。

The isolated working heart model was set up, high-performance liquid chromatography were used to measure myocardial adenosine triphosphate,total adenine nucleotides, while left ventricular end-diastolic pressure,left ventricular developed pressure, and rate of change of left ventricular pressure were determined before and after preservation.

对照组用St Thomas停搏液停搏并保存大鼠心脏6小时;实验组在St Thomas液中加入前列腺素E1(5 μg/L)停搏并冷保存6小时,利用离体鼠心非循环式Langendorff灌流功能测定模型,测定左心室舒张期末压、左心室发展压和左心室压力变化率,并检测心肌细胞线粒体三磷酸腺苷、腺嘌呤核苷酸总量。

RESULTS: Under isotonic conditions, no single channel activities were observed in most patches.

目的:研究急性分离小鼠心肌细胞肿胀激活氯离子通道的单通道特征。

The size of myocardial infarction caused by coronary artery ligation was decreased by Jat ( 0.75mg/kg iv. 1 h before and 6 h after operation) in rabbits.

耳静脉注射Jat(每次0.75mg/kg)还能使家兔冠脉结扎所致的心肌梗死范围缩小(n=6;P.01)。

Objective To explore the effect of pathological jaundice on neonatal myocardium and its clinical significance.

目的 探讨病理性黄对新生儿心肌组织的影响。

As the ischemic time was prolonged, amorphous dense bodies were found in swellen mitochondria, and the nucleus progressed gradually from early pyknosis to karyswelling, karyorhexis and karyolysis. The myocardium eosin staining became pale. Cell membrane was disrupted. This indicates that it entered an irreversible swelling stage.

随着缺血时间的延长,缺血1小时后,肿胀的心肌细胞,线粒体可见不定形颗粒,细胞核逐渐由早期固缩到肿胀、破裂及溶解,细胞伊红染色变淡,细胞膜破裂,表明此时的细胞已进入不可逆性肿胀阶段。

We also have observed some organs pathological changes of juvenile Jian Carp, such as pancreas acinous gland and zymogen granules of acinous gland cell disappearing, intercellular substance hyperplasia and inflammatory cells soakage, hepatatrophia, liver cell granular or vacuolar degeneration and necrosis, karyolysis or pyknosis, glycogen granules decreasing, metanephros atrophy, metanephric canaliculus epithelium granular or vacuolar degeneration and necrosis, mitochondrion swelling and mitochondrion cristae disappearing, karyolysis, distal convolutal tubule microvilli desquamating, spleen marrow cell degeneration and necrosis, intercellular substance of spleen hyperplasia, spleen atresia, blood corpuscle disappearing.

后肾土黄色、淡褐色或苍白色,肾小管上皮细胞肿胀、颗粒或水泡变性、坏死,细胞内有大量血细胞流出,线粒体肿胀,嵴结构消失,细胞核溶解,肾间质甲状腺滤泡和拟淋巴细胞增生,远曲小管微绒毛脱落、管道细胞界限不清。心脏肌纤维肿胀、颗粒或空泡变性,严重的肌纤维溶解、变细或断裂,肌纤维间水肿、炎性细胞浸润,部分心肌细胞核浓缩。脾脏暗褐色,脾髓质细胞变性、坏死,拟淋巴细胞明显减少,淋巴细胞岛少见、岛中细胞成份减少,黑素巨噬细胞中心减少、体积缩小,脾脏网状基质水肿,脾窦闭锁,血细胞减少。

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呼气,收缩臀部肌肉;拱起身体,尽量抬起头来,右腿伸直朝向天花板(膝微屈,以避免肌肉紧张)。

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