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All cardiac myocyte are capable of spontaneous rhythmic depolarization and repolarization of their membrane.

所有的心肌细胞都能自发的、有节律的进行细胞膜的去极化和复极化。

We also showed that the putative uncoupler bupivacainedid not depolarize mitochondria in intact ventricular myocytes.

我们同样发现可能的解联体布比卡因并不使完整的心室心肌细胞中的线粒体去极化。

The bone marrow mesenchymal stem cell has highly plastic and can differentiate into specialized tissues such as osteocytes, chondroblasts,derma, myogenic cells, and cardiac cells.

目前认为骨髓间充质干细胞有多向分化潜能,可以在适当的刺激下向骨组织、皮肤、骨骼肌细胞及心肌细胞转化。

METHODS We used a dermatome to obtain thin endocardium, midcardium and epicardium transmural strips from left ventricular free wall of canine (weight 15-18 kg) and standard microelectrode techniques to record and measure the transmembrane action potential duration.

应用标准玻璃微电极技术,在不同频率刺激下,记录应用胺碘酮前后犬左室游离壁内、外膜及中层心肌细胞跨膜动作电位各种参数的变化。

METHODS We used a dermatome to obtain thin endocardium,midcardium and epicardium transmural strips from left ventricular free wall of canine(weight 15-18 kg) and standard microelectrode techniques to record and measure the transmembrane action potential duration.

结果:①胺碘酮延长内、外膜层心肌细胞的动作电位时程,当基础周长为1 000 m s时,使用胺碘酮后心内膜层肌细胞的APD90由(254±38)m s延长为(274±37)m s(P.01),外膜层肌细胞的APD90由(205±37)m s延长为(229±16)m s(P.01);但其明显缩短中层肌细胞的APD,其APD90由(331±50)m s缩短为(290±62)m s(P.01)。

Butanedione monoxime, a cardiac excitation-contraction coupling desensitizer, increased ANP secretion and inhibited atrial stroke volume as well as pulse pressure.

心肌兴奋-收缩耦联脱敏剂2,3-丁二酮单肟促使ANP的分泌,对心房搏出量和心房搏动压呈抑制效应。

Objective To investigate the clinical features and treatment of acute myocardial infarction with diabetes mellitus.

目的探讨急性心肌梗死合并糖尿病的临床特点和治疗。

One of the enginery characters is that it has a longer refractory period, which includes the whole systole and the early period of diastole.

心肌的机能特性之一是具有较长的不应期,整个收缩期和舒张早期都是有效不应期。

Intensity of radio signal also had this variation between in diastole and in systole .

离体心脏的心肌射频信号强度收缩期和舒张期之间也存在这种差异。

"KVP" cardioplegic solution can lower diastole of the ischemic hearts,is beneficial for ischemic myocardial protection.

"钾维普"停搏液停搏的心脏缺血期间基础张力降低,有利于缺血心肌的保护。

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