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On the other hand, we took 7 patients with severe bicuspid valve disease and heart failure into group B1, all these pati...

同期收治风湿性二尖瓣疾病合并全心衰竭患者7 例分为B1组,于二尖瓣置换术中经左、右冠状动脉及心肌内注射干细胞悬液。

The brain and heart (the C-shaped blotch is the heart's muscular wall, called the myocardium) are also big energy users, so they show up too.

大脑和心脏(C形红块是心脏肌肉壁,即心肌层)同样会大量消耗能量,因此它们也会显示出来。

Spontaneous rupture of esophagus also known as Boerhaave's syndrome, is an extremely severe thoracic disease with high mortality, and is difficult to diagnose because of its rarity and frequent confusion with other severe clinical conditions, such as acute myocardial infarction, perforated peptic ulcer and acute pancreatitis.

自发性食管破裂,又称Boerhaave综合征,是临床上一种严重的胸科急症,因临床少见且易与其他疾病,如急性心肌梗死、消化性溃疡穿孔、急性胰腺炎等混淆,临床诊断较困难,死亡率高。

Award BIOS also does not automatically restore the BIOS information, whereas AMI BIOS 'boot block' can automatically trigger recovery process to look for new BIOS content to restore to the 'system block'.

奖的BIOS也不会自动还原BIOS信息,而急性心肌梗死的BIOS '开机区块'可以自动触发复苏的过程中寻找新的BIOS的内容,还原到'系统座'。

Marwick, from the University of Queensland in Brisbane, Australia, said in a statement.

研究结果显示,随着体重的增加,心肌变厚的几率也越来越大。

Intestine, lung, heart, kidney and liver tissue samples were also collected after reperfusion 2 hours, and (1) protein concentration of bronchia alveolus lung fluid ,(2)HSP70 expression in heart and kidney (3) histology pathology and (4) oxidation injury (MDA and GSH-PX) in above tissues were determined.

休克2小时,收集小肠、心肌、肺、肝、肾等组织用于测定(1)支气管肺泡灌洗液的蛋白含量(2)免疫组化检测心脏和肾脏HSP70的表达(3)组织病理切片(4)组织抗氧化指标。

Severe nonspecific airway inflammation is demonstrated in asthmatic patients with sudden death. Lesions of the left ventricle and mucous plug in bronchiole may contribute to the main cause leading sudden death.

结论哮喘猝死患者气道存在明显的非特异性炎症反应,心肌受损和细支气管粘液栓形成为其主要致死原因。

Gotoh Y, Nishida E, Yamashita et al. Microtubule associated protein kinase activated by nerve growth factor and epidermal growth factor in PC12 cells: identity with the mitogen-activated protein kinase of fibroblastic cells. Eur J Biochem, 1990, 349: 661~669.[7]Cook SJ, Beltman J, Cadwallader KA et al.

本文观察到,在AngⅡ介导的心肌细胞肥大反应中,伴随着MAPK的激活, MKP-1蛋白表达增加,若以AT1受体阻断剂CV11974或MAPK激酶特异性抑制剂PD098059抑制MAPK激活,同时也抑制了MKP-1蛋白表达,提示MAPK的激活可能是MKP-1蛋白表达的诱导因素之一。

Methods:To induce 17 515 adult physical exam materials that were collected by our Physica Examination Center from 2001 to April 2007 in LianYunGang cantonal,and analyze the detectable ratios of hypetension,hyperglymia and hyperlipemia and their relation with the detectable ratios of gender, age,occupation and heart abnormity.

归纳本中心2001~2007年4月本市区不同职业成人17 515例体检资料,分析高血压、高血脂、高血糖的检出率与性别、年龄、职业的关系和与左心室肥大和心肌缺血二项心电图异常检出率的关系。

Objective:To study the effects of Myocardial ultrastructure of capillary vessel during intermittent hypoxia training .

目的:探讨间歇低氧训练对大鼠心肌毛细血管的影响,为高原训练方案的制定提供理论依据。

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