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RESULTS Cardiac muscles of left atriums extended onto pulmonary veins.

P样细胞散在分布于心肌袖内,主要分布于心肌袖的中间部,并且几乎都是在上肺静脉中。

Objective To evaluate the effect of calcitonin gene-related peptide intravenous on myocardial injury and the oxygen free radicals in rats.

目的 研究静脉用降钙素基因相关肽对大鼠心肌缺血损伤的氧自由基影响及心肌保护作用。

Results Clofibrate normalized plasma free fatty acid and cholesterol and reduced plasma triglyceride and insulin in obese rats. However, clofibrate led to accumulation of heart TG and increased myocardial long-chain unsaturated and monounsaturated fatty acids in obese rats. Conclusion Clofibrate aggravates myocardial triglyceride accumulation in female obese Zucker rats.

结果 氯苯丁酯可使肥胖大鼠血浆游离脂肪酸和胆固醇浓度恢复正常并可降低血浆甘油三酯和胰岛素浓度,可增加肥胖大鼠心肌甘油三酯的积聚,且心肌长链不饱和脂肪酸和单不饱和脂肪酸含量也相应增加。

W. . The levels of myocardial troponin I, creatin kinase, aspartate transamnaseand lactatede hydrogenasewere detected before and per 30 mins during the experiment, and the changes of rabbit myocardial pathology were analyzed.

分别于实验前和实验中每隔30分钟抽取血液,以肝素抗凝并分析心肌肌钙蛋白I浓度,肌酸激酶、门冬氨酸氨基转移酶和乳酸脱氢酶活性,同时观察心肌组织的病理学变化。

Results At the 10th week after constriction,the structure of cadherin,desmosome and gap junction in intercalated discs were destroyed,gap of intercellular space widened.Pan-cadherin,Cx43 and desmoplakin distributed disorderly on the surface of cardiomyocytes.

结果 术后10周手术组心肌闰盘的粘着膜、桥粒、间隙连接结构模糊,连接间隙增宽;Pan-cadherin,Cx43,desmoplakin 在心肌细胞表面呈无序分布。

The digestions should be less than 6 times, and every digestion should last for less than 20 min. Two-hour differential adherence could improve the purity of myocardial cells. By this protocol, 94.6% of cultured cells were live myocardial cells.

分离细胞时消化次数最好控制在不多于6次,每次消化时间不超过20min.2h差速贴壁可以提高心肌细胞纯度,经肌动蛋白鉴定,心肌细胞达94.6%。

Thomas II, M group (Mexibetine 70μmol/L+ st. Thomas II), HM group (HOE642 20μmol/L+ Mexibetine 70μmol/L+ st. Thomas II). The percent recovery of heart function and water content ofmyocardial were determined after I/R both in group A and B. C was composed of 4 groups (n=24, 6 in each group) as B but the dugs were added at the earlier period of reperfusion.

中,心肌进行缺血/再灌注后检测其心功能恢复率和心肌水含量。B组(n=24,每组6只)分为对照组,H20组(HOE642 20μmol/L组),M组(慢心律70μmol/L组),HM组(HOE642 20μmol/L组+慢心律70μmol/L组),加入心脏停搏液

Cardiomyocyte hypertrophic response induced by ET-1 is mediated by ETA receptor coupled to PTX-sensitive G-protein, which involves at least two signalling pathways: PKC-mediated increase of [Ca2+]i , and PKC-independent activation of ERKs.

ET-1主要通过ETA受体并经PTX敏感的G-蛋白介导心肌细胞肥大反应,该作用至少涉及两条途径:①通过PKC介导的心肌[Ca2+]i浓度增加;②不通过 PKC介导的ERKs激活。

Results:The levels of inflammatory cells infiltration,myocardial fibro injury and CVB 3-RNA in group with interferon and shuanghuanglian were significantly lighter than control group.

结果 :干扰素和双黄连联合治疗组的炎症细胞浸润程度、心肌细胞损伤程度、心肌内病毒核酸含量显著低于病毒对照组(P 0 5 )。

The increased workload can put a strain on the heart, which can lead to heart abnormalities that are usually first seen as enlarged heart muscle.

这种工作负载的增加会导致心肌劳损,最后心脏异常的初发症状就是心肌变大。

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