心肌

Application of SOD and /or Hypoxia-preconditioning can prevent myocardiac injury partly by reducing the myocyte apoptosis.

SOD和或缺氧预处理具有抗心肌细胞损伤和减轻心肌细胞凋亡的作用，而且可能是通过降低细胞内钙离子浓度而起作用的。

Objective: To evaluate the significance of myocardial bridge.

目的：通过对53例心肌桥的临床资料进行分析，了解心肌桥的临床意义。

2Myocardial ultrastructure was normal in the control group,myoneme arranged disordered,the structure of typical sarcomere disappeared, mitochondrium intumesced,interstitial substance fiber hyperplasy,capillary vessel endothelial cell swelled in DG group.Myocardial ultrastructure improved slightly in rats of SM and LM groups. 3NF-κB p65 and COX-2 had a small quantity to present in the myocardium tissue of control group,while the quantity increased obviously in the diabetic group.The level of it in the group of treatment was significant lower than that in the diabetic group.There was significant difference between the two groups. Conclusion Metformin can protect against the heart injury in diabetic.

结果 1与对照组比较，DG组体重明显减轻，血糖、心脏质量指数、INS、TG、T-CH及FFA明显升高(P＜0.01)；应用 MT治疗后体重无明显增加，INS有所下降，心脏质量指数、血糖、TG及FFA明显降低(P＜0.01)；DG组心肌组织MDA明显升高，SOD明显降低(P＜0.01)，MT治疗后上述指标明显改善；2超微结构观察单纯糖尿病组肌丝结构明显紊乱，典型肌小节结构消失，线粒体肿胀、间质纤维增生，微血管内皮细胞肿胀等表现，而SM及LM组可见组织结构有所改善；3对照组心肌NF-κB p65及COX-2只有少量表达，单纯糖尿病组表达量明显增多(P＜0.01)，MT治疗后表达量明显减少(P＜0.01)。

In normal myocardium, sorcin extensively co-localized with ryanodine receptors, whereas in myopathic hearts, the degree of co-localization was markedly disrupted.

正常心肌细胞中，sorcin与细胞内钙释放通道RyR能广泛协同定位，而在心肌病的心肌细胞中，这种协同定位的能力被破坏。

How to make necrotic myocardial cell regeneration and diastolic myocardial function is the problem to solve.

如何使坏死心肌或无功能心肌再生，再现其舒缩功能，一直是亟待解决的难题。

Many years ago, it have been noticed that, not all injurise of heart cell were directly proportional to the inflammatary reaction, and this kind of noninflammatory damage may be apoptosis. Kawano has verified first time in 1994 that, the onset and development of VMC accompanied the apoptosis, and the apoptosis probably are significant mechanism from VMC to the dilated cardiomyopathy.

早有人注意到，VMC时心肌损伤并不都与炎症反应成正比，其存在的非炎症性损伤很可能是细胞凋亡。1994年Kawano等首次证实心肌炎伴有心肌细胞凋亡，后者可能是心肌炎发展成扩张型心肌病的重要机制。

But in the old age acute cardiac arrest case of illness,the nontypical angina pectoris disease incidence rate may be 30.2%,does not have the symptom is reaches as high as 42％,but the typical angina pectoriS only accounts for 20%.

一般在急性心肌梗死患者中非典型心绞痛型达1／6～1／3，而在老年急性心肌梗死病例中，非典型心绞痛型的发病率可为30.2％，无症状型更是高达42％，而典型心绞痛型仅占20％。

Reaches 1／6 generally in the acute cardiac arrest patient Central Africa typical angina pectoris～1／3.But in the old age acute cardiac arrest case of illness,the nontypical angina pectoris disease incidence rate may be 30.2%,does not have the symptom is reaches as high as 42％,but the typical angina pectoriS only accounts for 20%.

一般在急性心肌梗死患者中非典型心绞痛型达1／6～1／3，而在老年急性心肌梗死病例中，非典型心绞痛型的发病率可为30.2％，无症状型更是高达42％，而典型心绞痛型仅占20％。

However, it should be notal that the VE content of the KDR diet is not lower than the control's but dosage of Vs needed to prevent the damages is quite large in the case of low Se state (e. g. 200ppm was adde into the KDR diet in our experiments).

但值得注意的是，实验中所用病区粮VE含量不低于甚至高于非病区对照粮，在低Se情况下，所补VE的量需要相当大（如本实验中补充200μg／g）才能较明显地预防心肌细胞和心肌线粒体的损伤。

We generated transgenic mice that overexpress a heat shock protein 27 (Hsp27) and then to determine the effects of heat shock protein 27 on the isoproterenol-induced cardiac hypertrophy in early period.

通过转基因小鼠心肌特异性高表达人热休克蛋白27(Hsp27)模型，探讨人Hsp27对异丙肾上腺素(isoproteronol，ISO)诱导小鼠心脏是否存在早期抑制心肌肥厚的保护作用。

However, as the name（read－only memory）implies, CD disks cannot be written onorchanged in any way.

然而，正如其名字所指出的那样，CD盘不能写，也不能用任何方式改变其内容。

Galvanizes steel pallet is mainly export which suits standard packing of European Union, the North America. galvanizes steel pallet is suitable to heavy rack. Pallet surface can design plate type, corrugated and the gap form, satisfies the different requirements.

镀锌钢托盘多用于出口，替代木托盘，免薰蒸，符合欧盟、北美各国对出口货物包装材料的法令要求；喷涂钢托盘适用于重载上货架之用，托盘表面根据需要制作成平板状、波纹状及间隔形式，满足不同的使用要求。