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the positive expression of pcna in the myocardium after myocardial infarction is a sort of repairing response to the injury. the injured cardiomyocytes with pcna positive expression have decreased contractile function.

心肌梗死后部分心肌细胞表达pcna是对缺血损伤的反应,pcna阳性表达的心肌细胞其收缩功能降低。

Results: Compared with control dogs, sections from AF dogs showed cellular hypertrophy, disarranged cell bundles and increasing connective tissue.

结果:与对照犬相比,AF犬肺静脉肌袖内心肌细胞增大、排列紊乱;细胞核大小不甚规则,核异型性明显,细胞内可见肌纤维断裂;心肌纤维之间连接组织积聚,使心肌细胞之间的间隔增宽。

Myocardialβ3-AR density up-regulation obviously(-5.16±0.004 vs -5.13±0.011, P.05)in rats treated with dexamethasone compared with rats treated with saline. 6. Myocardialα1-AR density up-regulation obviously(-5.16±0.017 vs -5.14±0.003, P.01)in rats with CHF compared with normal rats, buffy particle increased in myocardium slice. Gray scale value grew down, and i.e.α1-AR density greatened.

地塞米松治疗的心衰大鼠与注射生理盐水的心衰大鼠相比,心肌的β3-AR明显上调(-5.16±0.004 vs -5.13±0.011, P.05)。6大鼠心衰时,心肌的α1-AR密度明显上调(-5.16±0.017 vs -5.14±0.003, P.01),免疫组化采集的图像显示,心衰大鼠的心肌切片棕黄色颗粒明显增多,灰度值变小,密度变大。

METHODS: The model of acute myocardial ischemia of rats was created by ligaturing the left coronary artery and followed with recording changes of EGG, the range of myocardial infarct and the levels of the AST, CK, LDH, SOD and MDA enzymes in blood serum to study the effects of gleditsioside on myocardial ischemia in rats.

采用结扎大鼠左冠状动脉的方法,造成急性心肌缺血模型,通过对大鼠标准Ⅱ导联心电图,心肌梗死面积及血清中天冬氨酸转氨酶、肌酸激酶、乳酸脱氢酶、超氧化物歧化酶和丙二醛的测定,探讨皂荚皂苷对大鼠心肌缺血的影响。

Methods: The model of acute myocardial ischemia in rats was developed by ligaturing the left descending anterior coronary artery. Forty male Wister rats were divided into normal control group, ischemia model group, Xuesetong group and Verapamil group. The effect of Xuesetong on the CPK, LDH, SOD and MDA in serum and myocardium and myocardial infarct range was observed.

采用结扎大鼠冠状动脉左前降支复制出大鼠心肌缺血再灌注损伤模型。40只Wister大鼠分为正常对照组、模型组、血塞通组及维拉帕米组,观察血塞通对大鼠血清磷酸肌酸激酶、乳酸脱氢酶、血清及心肌组织超氧化物歧化酶和脂质过氧化物丙二醛及对心肌梗死范围的影响。

aim: to reveal the significance of positive expression of proliferating cell nuclear antigen in myocardia of rabbits with myocardial infarction and the relationship between pcna positive expression and left ventricular function.

目的:阐明兔实验性心肌梗死模型中心肌增殖细胞核抗原(proliferating cell nuclear antigen, pcna)阳性表达的意义及其与心功能和心肌缺血的关系。

Enzyme immunoassay was used to detect Apelin in plasma and myocardia. RT-PCR was used to analyze the relative amount of mRNA of Apelin and APJ in the myocardia.

酶联免疫法测定血浆和心肌中Apelin含量,RT-PCR法测定心肌组织Apelin、APJ mRNA水平,Western blot方法测定心肌组织中APJ蛋白水平。

The result of the serum biochemistry detection indicate that CK and LDH activity step up obviously compare to the normal control group (P.01), to which the taurine treat group appear to decrease slightly contrasting (P.05) 4 The staining of HE showed the model group rat's myocardial fiber soluted, degenerated and necrosed partly, while the taurine treat group decrease obveriously the above-mention pathological changes.

4通过HE染色观察心肌组织发现,模型组大鼠的心肌组织有明显的纤维溶解、断裂及变性,而牛磺酸给药组能明显降低心肌组织的上述改变。

Ultrasonic tissue characterazation can assess pathological changes of myocardial tissue based on detecting and quantitating the acoustic properties of myocardium .

超声心肌组织定征技术(ultrasonic tissue characterization,UTC)是通过对心肌物理状态的变化即声学特征改变的检出和量化来判断心肌细微组织病理变化的方法。

Objective To evaluate the diagnostic value of"cTnⅠ+H-FABP" in acute myocardial infarction, and the effect of heart- type fatty acid-binding protein on elevating the sensitivity of the composite diagnostic scheme.

目的 探讨心肌型脂肪酸结合蛋白与肌钙蛋白Ⅰ联合检测对于急性心肌梗死诊断的价值及H- FABP在增加心肌标志物组合诊断效能方面的作用。

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呼气,收缩臀部肌肉;拱起身体,尽量抬起头来,右腿伸直朝向天花板(膝微屈,以避免肌肉紧张)。

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