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心律失常

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PURPOSE: To study the safety and possibility of extraction of teeth in patients with severe bradycardia and pacemaker under ECG monitoring.

目的:探讨安装心脏起搏器的严重缓慢型心律失常患者心电监护下拔牙的可行性。

Objective To approach the safety and efficiency of implantable pacemaker among patients with symptomatic bradycardia in basic-leuel hospitals.

目的 探讨症状性缓慢性心律失常患者在基层医院开展埋藏式心脏起搏器的安全性及有效性。

The tooth of patients with severe bradycardia and pacemaker could be extracted safely under ECG monitoring.

部分安装心脏起搏器的严重缓慢型心律失常患者在心电监护下可以安全拔牙。

METHODS: Twenty-one teeth in 12 patients with severe bradycardia and pacemaker were extracted under ECG and blood pressure monitoring.

在良好的局部麻醉和拔牙过程中密切观察心率、血压及动态心电变化的条件下,对12例伴有严重缓慢型心律失常且已安装心脏起搏器的患者进行拔牙,拔除患牙21颗。

Methods Clinical curative effect and complication of 36 patients with symptomatic bradycardia were evaluated, who were implanted with pacemakers by vein.

36例症状性缓慢性心律失常患者经静脉植入埋藏式心脏起搏器,观察治疗后的临床疗效及并发症。

Results The cardiograph change in varying degrees took place in 98 case among which the incidence of STT change and arrhythmia is relative high.

结果:98例发生不同程度的心电图改变,其中STT改变,心律失常发生率较高。

Results The cardiograph change in varying degrees took place in 98 case among which the incidence of ST T change and arrhythmia is relative high.

结果:98例发生不同程度的心电图改变,其中ST T改变,心律失常发生率较高。

Conclusions— A systematic reiew of published studies indicates that impaired left entricular function, New York Heart Association class III/I, cardiomegaly, and nonsustained entricular tachycardia indicate a poor prognosis in patients with chronic Chagas disease.

对已发表文献的系统回顾分析显示:Chagas病慢性期的病人出现左室功能障碍、NYHA心功能分级、心脏扩大和非持续性的室性心律失常,其预后不佳。

When an excess of food-borne excitotoxins, such as MSG, hydrolyzed protein soy protein isolate and concentrate, natural flavoring, sodium caseinate and aspartate from aspartame, are consumed, these glutamate receptors are over-stimulated, producing cardiac arrhythmias.

当过剩的经由食物传染的excitotoxins ,如味精,水解蛋白大豆分离蛋白和集中,天然调味品,酪蛋白酸钠和天门冬氨酸从阿斯巴甜,消耗,这些谷氨酸受体的过度刺激,产生心律失常

At the same time, the contents of myocardial Ca2+ and MDA were decreased significantly as well as the activities of myocardial SOD and GSH-Px increased markedly. Conclusion: CASI is of protective effect on myocardial ischemia-reperfusion arrhythmia, which may be related to scavenging the oxygen free radicals and Ca2+ overload formed during reperfusion.

CASI对大鼠心肌缺血再灌注心律失常具有明显保护作用,其机制可能与提高心肌组织的抗氧化酶活性,减少自由基及Ca2+超负荷对心肌的氧化损伤有关。

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