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CAC was identified in 2003 as a cardiovascular disease risk factor and an independent predictor of mortality. Investigators have known since at least 1996 that CAC is from 2 to 5 times more extensive in people on hemodialysis than in people with coronary artery disease but not kidney disease, said Dr. Asci, a nephrologist at the Ege University School of Medicine in Izmir, Turkey. Subsequent studies have shown an association between CAC and left ventricular hypertrophy and coronary abnormalities, including atherosclerosis, stiffness, and perfusion problems.

土耳其Lzmir Ege大学医学院肾脏科医师Asci医师表示,CAC在2003年即被确认为是心脏血管疾病的危险因子,同时也是独立的死亡率预测因子;研究者早在1996年就知道,相较於无肾脏疾病之冠状动脉疾病患者,透析病患CAC的情形普遍2~5倍;接下来的研究结果显示,CAC与左心室肥大与冠状动脉血管异常,包括粥状动脉硬化、血管硬化、与灌流异常是有关连的。

Increased lung olume, seere emphysematous change, dilatation of the right entricle, and mild pulmonary hypertension were obsered in (more than 15-wk-old) Tg mice.

转基因鼠中能观察到肺容积的增加,严重的肺气肿的改变,右心室扩张,在15周龄以上的鼠中还能观察到肺动脉压的轻度升高。

Objective To study the change of cardiac functions in hypertensive patients complicated with obˉstructive sleep apnea syndromeand those without OSAS.Methods To observe30hypertensive patients comˉplicated with OSAS and28hypertensive patients without OSAS.24h dynamic blood pressure monitoring,left ventricular function by echocardiography and polysomnography were measured.The left ventricular function was compared between two groups.

目的 通过超声心动图检查高血压并阻塞性睡眠呼吸暂停综合征患者的左心室功能,并与单纯高血压患者的左心功能比较,以探讨高血压并OSAS患者的心功能的改变方法选择30例高血压合并OSAS患者和28例单纯高血压患者,全部病人完成动态血压、心脏B超及睡眠呼吸监测等检查,并进行两组资料对比分析。

The complexity of the distribution of Purkinje fibers has led to great difficulty in ECG simulation research.

由于Purkinje纤维在心室内膜上分布的复杂性,给心电仿真的研究带来很大的困难,本文对此进行了很有意义的探索,即不仅把心肌单元按动作电位分类,又将其按传导速度分类。

Because definitie myocardial regeneration remains undemonstrated, we propose that cardiac changes obsered from implanted materials may result from altered mechanisms of the entricle.

因为发育成熟的心肌再生能力仍然不明确,我们计划观察由于植入材料心脏的变化导致心室工作的机制的变化。

The morphological and spontaneous contractile properties of isolated cell appeared to be typical embryonic cardiomyocytes when assessed by phase-contrast microscope.

心肌肥大会使胚胎时期出现过的基因重新表达[5],所以在胚胎心脏上曾经存在的电流可能会在病理情况下重新表现,从而导致某些心脏病的发生,事实上,在自发性高血压大鼠和人体衰竭心脏的心室肌细胞上都记录到了if [1, 2]。

Right ventricular outflow tract reconstruction for congenital heart disease or restoration of continuity after pulmonary autograft transfer in the Ross procedure is generally performed with pulmonary homografts, the current gold standard.

由于作为心脏外科临床进行右心室流出道重建材料的同种带瓣血管日渐匮乏,其广泛的临床应用受到很大限制,尤其是在需要小尺寸同种瓣的新生儿及婴幼儿右心系统复杂畸形的外科矫治方面更加突出。

They found that fish consumption was also associated with trends toward lower incidence of subclinical infarcts.

他们发现,吃鱼也与降低无临床症状的梗塞有关,鱼与较佳的白质等级也有关,但是对於脑病变的标记-脑回和心室等级无关;研究者观察发现,吃炸制鱼类与任何无临床症状的脑部异常无显著关联。

They found that fish consumption was also associated with trends toward lower incidence of subclinical infarcts.

他们发现,吃鱼也与降低无临床症状的梗塞有关,鱼与较佳的白质等级也有关,但是对于脑病变的标记-脑回和心室等级无关;研究者观察发现,吃炸制鱼类与任何无临床症状的脑部异常无显著关联。

Methods: Twenty-four male SD rats were randomly divided into normal control (NC, n=6), protein C activator (PCA, n=6), lipopolysaccharide (LPS, n=6) and LPS+PCA group (n=6). NC group were given normal saline and PCA group were injected protein C activator (0.4 mg/kg) through vein of the tail. LPS group were administered lipopolysaccharide (10 mg/kg, 4h) by intraperitoneal injection method to make the model of septic shock rats. After the animal model was made, LPS+PCA group were injected protein C activator (0.4 mg/kg) through vein of the tail. 30 minutes after the injection, each rat was tested by Medlab biosignal analyzing system for observing mean arterial pressure and left ventricle function, the activity of LDH and iNOS in myocardium and the cuttings of myocardial tissues.

取SD雄性大鼠24只随机分成对照组、PCA组、LPS组和LPS+PCA组四组,对照组尾静脉注射生理盐水,PCA组尾静脉注射蝮蛇毒蛋白C激活物,LPS组腹腔注射LPS (10 mg/kg, 4h)的方法建立败血症休克的实验动物模型,在建立败血症的实验动物模型的基础上,尾静脉注射PCA (0.4 mg/kg)为LPS+PCA组,每只大鼠均于药后30 min时运用Medlab生物信号采集处理系统记录平均动脉压和左心室功能指标,并测定心肌中乳酸脱氢酶活性、诱导性一氧化氮合酶活性及心肌病理切片。

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