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It seems that just by preventing the infarction from growing and by artificially thickening the heart wall in the infracted area , the graft could have helped prevent the usual remodeling of the ventricle

它的效果看起来比较像避免梗塞部位的扩大,并且增加了梗塞心脏的心室壁厚度,只能说,移植的组织可能防止了心室重塑。

The restrictive filling pattern is distinguished from overfilling of a normal ventricle by a reduced and delayed E' and a shortened E deceleration time that reflect slow relaxation and increased LV stiffness.

限制性充盈模式是区别二尖瓣环流速峰值减少和延缓导致的正常心室过度充盈与更短的二尖瓣血流E峰减速时间导致的正常心室过度充盈,其反映了舒张功能变得迟缓和左室室腔僵直程度的增加。

To investigate the changes and clinical significance of myocyte in right ventricle myocardial remodeling, a special technique connecting the arteria carotis communnis and venae jugularis externa was developed, and an artificial shunt of left to right was made resulting in volume and pressure overload in rat models.

目的:通过人工套接大鼠右侧颈动静脉,造成左向右分流,建立右心室容量、压力负荷增加的大鼠模型,观察右心室心肌细胞在此模型中的改变及其可能的临床意义。

Methods 20 patients with complete left bundle branch block and 20 normal subjects were studied.

方法对20例完全性左束支传导阻滞患者和20例正常人解剖M型在心室波群区观察室间隔与左室后壁运动;运用频谱多普勒成像技术测量左、右心室射血前期时间(LVPET、RVPET),计算两者之差PETd;采用定量组织速度成像技术测量右室侧壁基底段和左室各壁基底段达收缩期峰值速度时限和达舒张早期峰值速度的时限。

When in the short axis view with the papillary muscles and chordae tendineae in view place the M. mode cursor between the papillary muscles and obtain an M-mode trace of cardiac function.

心室乳头肌与其腱索呈现在短轴图像时,将M模式的屏幕游标放在心室的乳头肌之间,同时藉由M模式探测心脏的功能。

Methods Enzyme digestion was used to isolate single ventricular myocyte and whole cell patch clamp technique was used to record potassium current.

方法酶解法分离豚鼠单个心室肌细胞,应用全细胞膜片钳技术记录延胡索乙素对豚鼠单个心室肌细胞钾电流的影响。

PQDS maybe prevent and treat ventricular remodeling with improving systolic and diastolic function of cor sinistrum in ventricular remodeling rats.

PQDS可能通过改善心室重构大鼠的左心收缩和舒张功能发挥防治心室重构的作用。

ConclusionG-Rb has protective effects on ventricular remodeling through improving systolic and diastolic function of cor sinistrum in ventricular remodeling rats,increasing anti-oxidase activity, reducing the damage of free radicals and vasoactive substance in myocardium and correcting disequilibrium of PGI2/TXA2 in ventricular remodeling etc.

结论G-Rb对大鼠心室重构具有保护作用,可能与其改善心室重构大鼠的左心收缩和舒张功能,增强抗氧化酶活性,减少自由基及缩血管活性物质对心肌的损伤,纠正PGI2/TXA2失衡等机制有关。

METHODS: Open chest operation was performed on the anesthetized rats to take out their hearts. The remaining blood was washed, and the large vessels, atriums and right ventricle were removed. The left myocardium was cut into two parts, then put in the DMEM tube containing 600 mL/L of glycerol, frozen at 4℃ for 30 minutes, at -20℃ for 30 minutes, and at80℃ for 30 minutes. Then they were stored in the liquid nitrogen at196℃.

麻醉大鼠开胸后,迅速取出心脏,置于冷Krebs-Henseleit液中,洗去残存的血液,弃去大血管、心房和右心室,将左心室肌剪成两块,然后置于含600 mL/L甘油的DMEM的冻存管中,程序冷冻,即4℃30min,-20℃30 min,-80℃30 min,最后保存于-196℃的液氮中备用。

This might be the fault of LA-Ao assist with the pump, To remedy the fault, it may be taken to insert cannulae into LV through left atrial, which might improve the effect of LHA.

本研究证实,采用气动泵行LA-Ao左心辅助并不能有效地使左心室减压、使心肌氧耗量减少,即不能使左心室得到充分休息。

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