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Objective To investigate the characteristics of atrioventricular nodal reentrant tachycardia and its classification.

目的 探讨典型的房室结双径路引起的房室结折返性心动过速可激动间隙的特点及其分区。

Background Slowly activating delayed rectifier potassium currentI_(Ks is important in therepolarization of human cardiomyocyte action potential,especially in forming andmaintaining plateau of action potential. I_ channel is combined byαandβsubunits,which are coded by KCNQ1 and KCNE1 respectively. Mutations ofhKCNQ1/hKCNE1 genes,chronic myocardial hypertrophy and chronic heartfailure will lead to dysfunction of I_ and ingenital or acquired LQTS which areimportant factor in hazardous ventricular arrhythmia such as Torsade depoints.

实验背景缓慢激活延迟整流钾电流I_(Ks是参与人心肌细胞动作电位复极,特别是平台期形成与维持的重要外向离子流。I_通道由KCNQ1基因编码的α亚单位及KCNE1基因编码的β亚单位组成。hKCNQ1/hKCNE1基因突变或慢性心肌肥厚、慢性心力衰竭等病理过程均可导致I_功能异常,引起遗传性或获得性长QT综合征,LQTS易引起尖端扭转型室性心动过速等心律失常,从而危及病人生命。

Methods The experimental arrhythmia models were induced with barium chloride (BaCl2),uabaina and aconitine in rats and cobayaes.The incidence rate and mortality rate of arrhythmia,the emergence time and the duration of arrhythmia induced with BaCl2 were recorded in rats pretreated with CASI (in dosages of 25,50, and 100 mg·kg-1 sublingual vein injection).At the same time,the dosages of uabaina and aconitine were observed when the ventricular premature contraction,ventricular tachycardia,ventricular fibrillation and cardiac asystole were appeard in rats and cobayaes pretreated with CASI (in dosages of 25,50, and 100 mg·kg-1 sublingual vein injection).

制备氯化钡(BaCl2)、哇巴因及乌头碱诱导的大鼠及豚鼠实验性心律失常模型,CASI按25、50、100 mg·kg-1分为3个剂量组,舌下静脉给药1次,不给CASI作为对照组,记录大鼠给BaCl2后心律失常的发生率、出现时间、持续时间及动物的死亡率;观察豚鼠及大鼠出现室性早搏、室性心动过速、心室颤动及心脏停搏时哇巴因及乌头碱的用量。

And quiver to the room of fast abidance , the room attacks , reach clonic the sex on the room tachycardia cannot be able to bear or endure the person that suffer , the patient that the room that high room room conducts block sluggish and frequency hair , much source contracts before sexual period , cannot work not only , and should treat actively

而对快速的持续的房颤、房扑,及阵发性室上性心动过速不能耐受者,高度房室传导阻滞及频发、多源的室性期前收缩的患者,不仅不能工作,而且要积极治疗。

Objective To compare the therapeutic effect and side effect of propafenone and adenosine triphosphateon paroxysmal supraventricular tachycardia.

目的 对比普罗帕酮与ATP对阵发性室上性心动过速(PSVT的治疗效果及各自的不良反应。

Objective To compare the therapeutic effect and side effect of propafenone and adenosine triphosphateon paroxysmal supraventricular tachycardia.

目的 对比普罗帕酮与ATP对阵发性室上性心动过速(PSVT的效果及各自的不良反应。

Methods 53 patients with paroxysmal supraventricular tachycardia were given incremental doses of adenosine intravenously during sinus rhythm before electrophysiological study.

对53例阵发性室上性心动过速患者在窦性心律下经外周静脉推注递增剂量的腺苷后观察体表心电图上房室传导时间的变化。

Serotonin syndrome and paraneoplastic syndrome were likely diagnoses and adenosine is an appropriate drug for a supraventricular tachycardia.

医疗方面也不全是糟糕的,羟色胺综合征和副肿瘤综合征都是可能的诊断,腺苷确实可以用来治疗室上性心动过速。

Here we show that transplantation of embryonic cardiomyocytes in myocardial infarcts protects against the induction of ventricular tachycardia in mice.

我们的研究显示心肌梗死大鼠移植胚胎心肌细胞能够对诱发的室性心动过速起保护作用。

If someone were to take a higher than expected dose of Dextroamphetamine Sulfate, then the risk of adverse effects known to be associated with the drug such as tachycardia, hypertension, tremors, decreased appetite, headache, insomnia, dizziness, blurred vision, stomach upset, and dry mouth may be increased.

如果服用硫酸右苯丙胺高于推荐剂量,可能引起已知的药物不良反应的风险,如心动过速,高血压,震颤,食欲下降,头痛,失眠,头晕,视力模糊,肠胃不适,和口干。

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