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心动过速

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And quiver to the room of fast abidance , the room attacks , reach clonic the sex on the room tachycardia cannot be able to bear or endure the person that suffer , the patient that the room that high room room conducts block sluggish and frequency hair , much source contracts before sexual period , cannot work not only , and should treat actively

而对快速的持续的房颤、房扑,及阵发性室上性心动过速不能耐受者,高度房室传导阻滞及频发、多源的室性期前收缩的患者,不仅不能工作,而且要积极治疗。

In other words, the interruption of reentry induced by RFCA does not indicate the definite histological lesion or enzym injury of the reentrant circle, but probably signifies that the change of ion channels with a large area involves the reentant circle.

也就是说射频消融终止折返性心动过速并不一定是折返环路的组织结构或代谢损伤所致,而可能是射频电流对大范围的心肌细胞膜离子流的影响累到及折返环路所致。

A rapid increase in the concentration of desflurane also induces tachycardia but is not associated with epileptiform EEG.

快速增加地氟醚浓度诱发心动过速不伴有癫痫样脑电图。

Tachycardia during increasing concentrations of D is not associated with epileptiform EEG.

地氟醚浓度增加时伴有心动过速不伴有癫痫样脑电图。

Methods:We ablated the ventricular tachycardia of 12 patients with normal hearts by the catheter, using pace mapping and activation sequence mapping. The former original target was the site where the pacing QRS was entirely consistent with IVT(Idiopathetic Ventricular Tachycardia) attack. And the latter original target was the site of in advance of earliest high frequency and low amplitude local potential on surface electrocardiogram QRS≥20ms, using activation sequence mapping. Results: Among 12 patients, the IVT originated from right ventricle in 2 patients, from left ventricle in 10 patients.

对12例正常心脏室速进行导管消融,采用起搏标测与激动顺序标测,前者以起搏时与室速QRS波型形态完全相同,后者以激动顺序标测法定位,以记录心动过速时较体表心电图QRS提前≥20ms的最早高频低振幅局部电位处为消融靶点,结果:12例患者室速起源部位分别为:右心室2例,左心室10例,疗效100%。

TreatmentGeneral considerations. All patients with UGB should be evaluated for signs and symptoms of hemodynamic instability and active hemorrhage (i.e., hypotension, tachycardia, hematemesis, melena, and a precipitous drop in hemoglobin concentrations).

治疗概论。所有上消化道出血之患者应评估其血液流动不稳定和活耀性出血之讯号和症状(即,低血压,心动过速,呕血,黑便,以及血红蛋白浓度直线下降)。

In SCD survivors, sustained monomorphic ventricular tachycardia is inducible by electrophysiologic testing in 40 to 50% and polymorphic VT in 10 to 20%; in 30 to 50%,no sustained arryhthmia is induced.

在心源性猝死生还者中, 40~50%电生理试验能诱导持续单一型室性心动过速,10~20%能诱导多型的,30~50%不能诱导持续的节律异常。

MRI indices were related to inducibility of sustained monomorphic entricular tachycardia during electrophysiological or deice testing.

在电生理学的或设备的测试期间,MRI显示与持续单一型室性心动过速的可诱导性有关。

In patients with SCD and idiopathic dilated cardiomyopathy, sustained monomorphic VT is rarely induced.

心源性猝死和先天性扩张性心肌病病人中,持续单一型室性心动过速极少能诱导。

Quantification of tissue heterogeneity at the infarct periphery was strongly associated with inducibility for monomorphic ventricular tachycardia (noninducible versus inducible: 13±9 versus 19±8 g, P=0.015) and was the single significant factor in a stepwise logistic regression.

梗死灶周围组织不均一性的定量与单一型室性心动过速的可诱导性强烈相关(不可诱导者与可诱导者相比:13±9 vs 19±8 g, P=0.015),且是逐步logistic回归的唯一显著因素。

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