心内膜

Results Of total 11 patients, 10 cases were mirror-image dextrocardia, 1 case was dextroversion. Among 10 patients were diagnosed as mirror-image dextrocardia, 5 patients were found to have concomitant cardiovascular malformations, the major anomalies were VSD,TOF, DORV. 1 case of dextroversion with complex cardiac anomalies: CECD+SA+DORV+PS. Conclusion Congenital dextrocardia may present with complex anomalies.

结果：11例先天性右位心患者中，镜像右位心10例，右旋心1例。10例镜像右位心中5例合并心内畸形，主要畸形为：室间隔缺损2例，法洛四联症2例，右室双出口1例；超声漏诊2例合并双上腔静脉。1例右旋心合并复杂畸形：完全型心内膜垫缺损、单心房、右室双出口、肺动脉瓣重度狭窄。

Then it is used to create a focal lesion on the epicardium or endocardium by delivering temperatures as low as -80oC at its tip.

通过设备的尖端输送-80度的低温到心外膜和内膜产生局灶性损伤。

The degradating and redistributing of ECM were migration of VSMC and vascular remodeling through inhibiting the production of PDGF-BB and the activation of MMPs so to prevent the vascular stenosis.⑤Orthotopic hybridism in situ: The expression of MMP-3 increased after operation, the tendency and intensity were parallel to the expression of NF-κB. Tongxinluo could decrease the expression of MMP-3 and NF-κB. The study showed that the producting and activating of MMP-3 were modulated by NF-κB. The inhibition to the producting and activating of MMp-3 of Tongxinluo was mediated by inhibiting the activating of NF-κB. Conclusion ①The rabbit vascular stenosis model could be established successfully by plain balloon damage, the operation process was easy, economical and practical.

①单纯球囊损伤可成功建立典型家兔血管狭窄模型，操作方法简单，经济实用；②以VSMC增殖、迁移为主的内膜增厚以及以ECM降解、合成与再分布所致的血管重构是导致受损血管狭窄的根本原因；③通心络能明显抑制VSMC的增殖、迁移和ECM降解、合成，阻止血管内膜增生和血管重构，防止受损血管狭窄；该作用可能与其减少和阻止PDGF-BB的产生和活性，以及抑制MMPs的表达和恢复MMPs/TIMPs平衡有关；④通心络能显著抑制球囊损伤后血管内膜增生和血管重构，可能与其保护血管内皮，改善内皮功能，增加血清NO含量有关；⑤通心络抑制内膜增生和血管重构，减轻受损血管狭窄，还可能是通过抑制NF-κB的活化途径而起作用的。

Result The improved mathematical model was applied to sequential echocardiographic images and the experimental results showed that endocardial boundary can be accurately detected.

结果将改进的快速行进模型应用于心超图像中心脏内膜边缘检测，实验结果表明该方法能够准确地检测、跟踪出心脏内膜边缘。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后，血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少，Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少，血小板聚集程度及红细胞聚集指数显著上升，病理切片显示胸主动脉内皮细胞破坏明显，脑心肾组织内的血管进一步增厚甚至几乎阻塞，导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR，脑体重比显著升高；心肌内出现明显的缺血梗塞疤痕灶，心体重比显著升高；肾小球明显萎缩、玻璃样变性，肾小管也变性甚至坏死，使肾硬化程度显著增加呈颗粒性固缩肾，肾体重比也显著减少。

First an advanced snake model based on the gradient vector field was used to segment the contour of the left ventricular and an initial snake line of the contour was got, then optical flow method was used to track the contour of the LV in the successive images. Finally, the Simpson method was applied to calculate the volume of the LV.

首先采用基于梯度向量流的主动轮廓模型对左心室内膜进行分割获取初始轮廓线，再采用光流法跟踪得到心脏序列中后续帧图像的心腔内膜轮廓线，最后采用Simpson方法计算得到左心室容积。

Other conditions that increase the risk of embolism include mitral stenosis (narrowing of a valve in the heart), and endocarditis (swelling of the heart chambers and valves).

其他条件的风险增加栓塞，包括二尖瓣狭窄，以及性心内膜炎（肿胀的心脏，商会和阀门）。

Streptococcus suis type 2 is one of the most important zoonosis, which infect human as well as pigs, horses and cats. It caused permanent deafness or deafness, septicaemia and death.

猪链球菌2型(Streptococcus suis type 2)是重要的人畜共患病的病原，除感染猪、马、猫等动物之外，还可感染人，引起脑膜炎，并可能导致永久性耳聋或复视等后遗症、心内膜炎、败血症及突发性死亡。

And rheumatic valves also accounted for 70 cases (88.5%) of aortic stenosis plus insufficiency. The rheumatic valves was decreased dramatically as the cause of the aortic valve disease, aortic root dilation and degenerative valves increased significantly, while others had no significant changes.

随着社会进步和人口老龄化，风湿性病因所占比例大幅下降，主动脉根部扩张导致的主动脉关闭不全有较大幅度的上升，退行性主动脉瓣有所上升，二叶式主动脉瓣、感染性心内膜炎所致瓣膜损害变化不明显。

The characteristics and changes of etiology were analyzed. All cases with aortic diseases were divided into single aortic stenosis, single aortic insufficiency, and combined aortic stenosis plus insufficiency group.

分别以主动脉瓣狭窄、主动脉瓣关闭不全、主动脉瓣狭窄合并关闭不全3组分析，具体病因包括风湿性、二叶式主动脉、退行性主动脉瓣、主动脉根部扩张、感染性心内膜炎、少见及未确定等6类病因。

We have no common name for a mime of Sophron or Xenarchus and a Socratic Conversation; and we should still be without one even if the imitation in the two instances were in trimeters or elegiacs or some other kind of verse--though it is the way with people to tack on 'poet' to the name of a metre, and talk of elegiac-poets and epic-poets, thinking that they call them poets not by reason of the imitative nature of their work, but indiscriminately by reason of the metre they write in.

索夫农 、森那库斯和苏格拉底式的对话采用的模仿没有一个公共的名称；三音步诗、挽歌体或其他类型的诗的模仿也没有——人们把&诗人&这一名词和格律名称结合到一起，称之为挽歌体诗人或者史诗诗人，他们被称为诗人，似乎只是因为遵守格律写作，而非他们作品的模仿本质。

The relationship between communicative competence and grammar teaching should be that of the ends and the means.

交际能力和语法的关系应该是目标与途径的关系。