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Cyclophilin A is a important member of a large family of ubiquitous peptidyl-prolyl cis-trans isomerases that regulate protein folding .

Cyclopnilin A是生物界普遍存在的肽基脯氨酸顺/反异构酶家族的主要成员,能调节细胞内蛋白质折叠。

Cyclophilin A is a important member of a large family of ubiquitous peptidyl-prolyl cis-trans isomerases...

Cyclopnilin A是生物界普遍存在的肽基脯氨酸顺/反异构酶家族的主要成员,能调节细胞内蛋白质折叠。

Objective To investigate the distributionand the chimerism features after infusion of allogeneic bone marrow cells in rats.

目的 研究大鼠异基因骨髓细胞输注后在受者器官的嵌合状态及分布规律。

Transgenic expression of the Ascl2 transcription factor throughout the intestinal epithelium induces crypt hyperplasia and ectopic crypts on villi.

遍及小肠上皮细胞的Ascl2转录因子的转基因表达,能够引起小肠绒毛上小囊的增生和异位。

There were many irregular vascular lumens , dells and proliferated endothelial cell masses with large atypical n .

多数不规则的血管腔,裂隙及增生的内皮细胞团块,细胞核大,有明显异形。

Bitter compounds in saliva activate particular T2R/TRB isoforms activate gustducin heterotimers, activated α-gustducin stimulates PDE to hydrolyze cAMP, the decreased cAMP may disinhibit cyclic nucleotide-inhibited channels to elevate intracellular Ca2+.

唾液中的苦味化合物激活T2R/TRB同型体,这种激活导致味导素异聚体的激活,被激活的α-味导素又激活PDE,PDE水解cAMP,cAMP水平的降低使环核苷酸抑制的阳离子通道去抑制,从而使细胞内Ca2+升高。

Salty and sour directly transduction by apical channels that are non-gated channel or chemically-gated channel. Bitter compounds in saliva activate particular T2R/TRB isoforms which activate gustducin heterotimers, activated α-gustducin stimulates PDE to hydrolyze cAMP, the decreased cAMP may disinhibit cyclic nucleotide-inhibited channels to elevate intracellular Ca2+.

咸和酸直接通过顶部非门控或化学门控离子通道转导唾液中的苦味化合物激活T2R/TRB同型体,激活导致味导素异聚体的激活,被激活的α-味导素又激活PDE,PDE水解cAMP,cAMP水平的降低使环核苷酸抑制的阳离子通道去抑制,从而使细胞内Ca2+升高。

Bitter compounds in saliva activate particular T2R/TRB isoforms which activate gustducin heterotimers, activated α-gustducin stimulates PDE to hydrolyze cAMP, the decreased cAMP may disinhibit cyclic nucleotide-inhibited channels to elevate intracellular Ca2+.

唾液中的苦味化合物激活T2R/TRB同型体,这种激活导致味导素异聚体的激活,被激活的α-味导素又激活PDE,PDE水解cAMP,cAMP水平的降低使环核苷酸抑制的阳离子通道去抑制,从而使细胞内Ca2+升高。

Such conditions can occur in sickle cell disease, fat embolization, or air embolization from dysbaric phenomena.

这种状况可能发生在镰状细胞病,脂肪栓塞或空气栓塞从异压性骨现象。

Isoflurane and halothane cause electroencephalographicdepression and neuronal depression in the reticular formation,a site critical to consciousness.

异氟烷和氟烷在网状细胞结构(决定意识状态的关键之处)能产生脑电抑制和神经细胞抑制。

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