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At the same time, the numbers of activated lymphocytes, macrophages, DNES cells and the expression of Lysozyme also up-regulated in some subfields of sacculus rotundus.

结果如下:家兔感染巴氏杆菌、大肠杆菌后,圆小囊各部位中SP和VIP的表达量均出现了明显的变化,相应部位的淋巴细胞活化分子、巨噬细胞及DNES细胞的数量也呈现出显著的变化。

However, IL-18 expression w a s more abundant in the thyroid tissues of HT group than in those of ST grou Th e expression was mainly localized in thyroid follicular cells from HT patients a nd in the mononuclear-macrophages within sarcoid from ST patients .

IL-18在HT组甲状腺组织中的表达明显高于ST组,前者 IL-18表达主要定位于甲状腺滤泡细胞,而后者则主要定位于肉芽肿内单核-巨噬细胞

Abstract] Objective To study effects of recombinant BCG-Sj26GST vaccine of schistosome on NO concentration of serum in mice.

NO是一种免疫效应分子,在巨噬细胞杀伤血吸虫童虫过程中起重要作用,它引起靶细胞酶的铁丢失,从而杀伤童虫[1]。

Results Compared to the controlled groups,the lungs in cerebral hemorrhage groups existed hemorrhage,serosity exudation and inflammation infiltration(including macrophage and leucocyte),and the number of macrophage an...

结果与对照组比较,脑出血组肺组织存在明显渗出、出血及大量白细胞、巨噬细胞浸润,炎症细胞记数有显著意义P.05。结论脑出血可以通过引发炎症反应导致急性肺损伤;炎症反应在3~5d时最明显。

Meanwhile, pulmonary epithelial cells and pulmonary macrophages play an important role in the development or silicosis.

同时,肺上皮细胞和肺巨噬细胞对矽肺发生发展起到不可忽视的地位。

Aim To study the effect of phosphalipids on the cholesterol efflux from rat macrophages mediated by HDL3. Methods Measure the cholesterol content, cellular phospholipid content, and the balance between free cholesterol and cholesteryl ester when phosphatidylcholine or sphingomyelin was added to the mediun.

为了研究卵磷脂和鞘磷脂对高密度脂蛋白3介导大鼠腹腔巨噬细胞内胆固醇流出的影响,测定了细胞内胆固醇含量、磷脂含量及游离胆固醇-胆固醇酯平衡的变化。

ABSTRACT Aim To study the effect of phosphalipids on the cholesterol efflux from rat macrophages mediated by HDL3. Methods Measure the cholesterol content, cellular phospholipid content, and the balance between free cholesterol and cholesteryl ester when phosphatidylcholine or sphingomyelin was added to the mediun.

摘 要]为了研究卵磷脂和鞘磷脂对高密度脂蛋白3介导大鼠腹腔巨噬细胞内胆固醇流出的影响,测定了细胞内胆固醇含量、磷脂含量及游离胆固醇-胆固醇酯平衡的变化。

Objective:To study molecular mechanism of suppressive effect of macrophages from traumatized mice on T cell functions.

目的:研究创伤后巨噬细胞抑制T细胞功能的分子机制。

Objective: To study molecular mechanism of suppressive effect of macrophages from traumatized mice on T cell functions.

摘 要 目的:研究创伤后巨噬细胞抑制T细胞功能的分子机制。

Methods Freshly isolated eosinophils were cultured in vitro for 24 hours with granulocyte-macrophage colony factor to induce HLA-DR expression, and then exposed to different concentrations of tetanus toxoid. The capacity of HLA-DR+ eosinophils to function as APC was evaluated with the response of T cell proliferation induced by eosinophils.

新分离的EOS经人重组粒细胞-巨噬细胞集落刺激因子刺激24小时,以诱导HLA-DR的表达,然后将其暴露于不同浓度的TT,检测HLA-DR+EOS对自身T 细胞增殖反应的影响,以评价EOS呈递抗原的能力。

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And Pharaoh spoke to Joseph, saying, Your father and your brothers have come to you.

47:5 法老对约瑟说,你父亲和你弟兄们到你这里来了。

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