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The Th1/Th2 ratio was decreased markedly in uremic patients (P<0.05). Conclusions Uremic patients showed Th2 predominance. LYmphopenia and Th1/Th2 imbalance may contribute to the immuno-insufficiency in HD patients.

尿毒症患者T细胞亚群表现为Th2优势,透析患者存在B淋巴细胞减少,淋巴细胞亚群的这些改变可能参与了血液透析患者免疫功能下降的发病机理。

Methods The clinical results of 28 cases of polycystic kidney receiving kidney transplantation were analyzed.

尿毒症是先天性多囊肾疾病发展的最终结果,肾移植是其最理想的治疗方法。

Before dialysis there were 19 patients with overhydrated heart failure, 23 with moderate or severe hypertension, 8 with uremio pericarditis or hemorrhagio perioardial tamponade, and 14 patients associated with serious psychoneurological disturbances.

它的疗效持续、稳定,操作简便,可在家庭施行透析,特别适合于尿毒症患者伴有心包炎、严重贫血、心血管功能不稳定的老年病例和不宜血液透析者。

To observe upper digestive tract mucosa lesions of uremic patients and to analysis the as- sociation among mucosa lesions and clinical symptom and dialysis.

目的 观察尿毒症患者上消化道黏膜病变的情况并分析其与临床症状和透析的关系。

Methods The present study included twelve undialyzed ESRD patients with anemia.The sera from the uremic patients were added to CFU-E and BFU-E culture in the concentrations ranging from 1.25% to 5%.In vitro CFU-E and BFU-E growth in the presence of sera from ESRD patients was compared with in the presence normal human subjects with the use of normal mice bone marrows.

选择12例尿毒症肾性贫血未透析患者为试验组,另选正常健康体检者12例为正常对照组,将实验组、正常对照组血清分别稀释成不同终浓度,采用甲基纤维素细胞培养的方法对小鼠骨髓细胞进行培养,对比不同浓度血清组红系集落形成单位、红系爆式形成单位的集落数。

Hp infection can degrade gastric mucosa's resistance, but it isn't the main cause of uraemic gastric mucosal lesion.

Hp感染可以降低胃黏膜的抵抗力,但Hp感染不是尿毒症胃黏膜病变的主要原因。

Serum gastrin obviotisly uprosed when mucosal lesions with erosion, ulcer, polyp, ect. However, Hypergastrinemia may be the main cause of uraemic gastric mucosal lesion.

胃黏膜糜烂、溃疡、息肉/息肉样病变患者的血清胃泌素水平明显升高,提示高胃泌素血症可能是尿毒症胃黏膜病变的主要病因。

Moreoer, our data suggest that uremic men may be more susceptible than are women to inflammation-induced anorexia.

并且我们的数据还显示,尿毒症男性患者对炎症诱发的食欲减退较女性更为易感。

METHODS: Six patients with urinemia underwent renal transplantation with donor kidney of brain death patients from May 2006 to November 2008 at the Organ Transplantation Center, 309th Hospital of Chinese PLA, were selected, including 2 patients receiving kidneys from a brain death patient, 4 patients receiving kidneys from 3 brain death patients.

选择解放军第三○九医院全军器官移植中心2006-05/2008-11尿毒症患者6例,其中2例接受1例院内脑死亡患者的无偿供肾,4例患者接受3例院外脑死亡患者的无偿供肾。

Materials and Methods: 40 Sprague-Dawley Rats were divided into five groups at random. 8 rats served as normal control group. The remaining rats were given 5/6 subtotal nephrectomies to establish the model of CRF.

目的:观测慢性肾功能衰竭(Chronic Renal Failure, CRF)肾功能、肾脏病理和血浆丙二醛、超氧化物歧化酶,以及肾、心、小肠线粒体的改变,初步探讨CRF&肾小管高代谢学说&和&尿毒症毒素致病学说&的深层机理。

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