尿毒症

Intermittent hemodialysis produces a dramatic alkalinization, frequently followed by a rebound of acidosis; however, CRRT slowly generates a steady-state concentration of both uremic solutes and organic acids in blood.

间歇性治疗会产生显著的碱血症，随后常常导致酸中毒的反弹。而CRRT则是缓慢地产生稳定浓度的尿毒症溶质和有机酸。

Results: All patients with serum Cr and BUN to fall back to normal 24 cases, 7 cases of azotemia, uremia in 5 cases, and no case died after treatment.

结果 所有患者血Cr及BUN进行下降，恢复正常24例，氮质血症7例，尿毒症5例，无1例治疗后死亡。

Methods: 30 control subjects and 79 patients were involved in this study, 79 patients were divided into azotemia group and uremia group by serum crestitine, serum LN and α1 - MG concentrations were detected by radioimmunoassay.

79例慢性肾功能不全患者以血肌酐水平分为氮质血症组和尿毒症组，用放免分析法对全部患者血清LN和α1—MG进行检测。

Methods: Separated 120 cases with glomerular diseases into chronic glomerular nephritis group (70 cases, including 33 cases with normal serum creatinine, 17 cases with azotemia, 20 cases with uremia, 33 cases accompanied with hypertension), acute nephritis group (12 cases), nephritic syndrome group (38 cases), and selected 30 healthy cases as control group. Detected concentration of NO in serum with nitrate reductase method, and made comparison among groups.

120例肾小球疾病患者分为慢性肾小球肾炎组（70例，其中血清肌酐正常33例，氮质血症17例，尿毒症20例，伴有高血压33例）、急性肾炎组（12例）、肾病综合征组（38例），同期选择30例健康体检者作为对照组，采用稍酸还原酶法测定肾小球疾病患者及对照组血浆NO浓度，并对各组NO浓度进行比较。

Results The reference range of serum Cys C in healthy people was ≤1.02 mg/L in our laboratory. The correlation coefficient between Cr and Cys C was 0.734 6. The serum level of Cys C in patient with DM was higher than that in healthy people. The level of Cys C in 2.18-3.15 mg/L and ≥3.16 mg/L could be defined as the limits of azotemia and uremia.

结果 本实验室健康人群Cys C参考范围为≤1.02 mg/L，Cys C与Cr的相关系数为0.734 6，Cys C水平在2.18~3.15 mg/L和≥3.16 mg/L可分别作为界定肾功能不全氮质血症期和尿毒症期的衡量标准，糖尿病患者的Cys C水平明显高于一般人群。

The outcome factor was recovery of renal function (serum creatinine ≤116 μmol/L) in azotemia patients or a 50% reduction of serum creatinine and discontinuation of dialysis in uremic patients,improved for at least three months.

结局因素：氮质血症者血肌酐下降至116 μmol/L以下，尿毒症者血肌酐下降50%以上，并脱离透析3个月以上为阳性结局，死亡者因其肾功能不可能再恢复，因此删失时间为28周。

Herein, we report the cases of 2 cirrhotic patients and 1 uremic patient, all with delayed presentations of Boerhaave's syndrome who survived with intensive conservative management.

因此，我们报告两个肝硬化、一个尿毒症，且延迟就医之Boerhaave氏症候群的病患，成功地以保守疗法挽救其生命。

We can improve the survival rate and quality of life by receiving hemodialysis early, reinforcing hemodialysis individually, receiving more nutrition, preventing and treating complications, doing psychological canalize of the old patients.

早期充分透析，加强个体化透析，增加营养物质的摄入，积极防治并发症，做好心理疏导，可提高老年慢性肾衰尿毒症患者血液透析的存活率和生存质量。

This type of pericarditis is typical of uremia with renal failure, underlying myocardial infarction, and acute rheumatic carditis.

此种类型的心包炎在尿毒症肾功能衰竭的患者较为典型，也可能由心肌梗死和急性风湿性心脏病引起。

Neuropathy is a common complication of uremia and is one important cause of death.

神经病变是尿毒症常见的并发症之一，也是死亡的重要原因。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。