小脑的

Results The clinical manifestations of TOBS comprised transient unconsciousness, vertigo, dyskinesia of eyeball, abnormality of pupils, dyskinesia of limbs, hemianopsia or cortical blindness, ataxia and memory impairment. MRI showed the local infarction in thalami, cerebellum, midbrain, pons, occipital lobe, temporal inner surface. The features of DSA were occlusion or stenosis in basilar artery and vertebral artery.

结果 TOBS的临床主要表现为一过性意识障碍、眩晕、眼球运动障碍及瞳孔的变化、肢体运动障碍、偏盲或皮质盲、共济失调及记忆功能损害；磁共振显示梗死灶在丘脑、小脑、中脑、脑桥、枕叶、颞叶内侧面；数字减影血管造影显示为基底动脉及椎动脉闭塞或狭窄。

In the brain stem,there were very dense orexin B immunoreactive fibers in the superior colliculus,inferior colliculus,interpeduncular nucleus,dorsal raphe nucleus and central gray.

此外，在小脑、脊髓和垂体柄及垂体后叶也有少量的阳性纤维的分布。

In addition, the cytoarchitecture of the cerebelli and the medulla oblongata of Taihe silky fowl and the medulla oblongata of African ostrich were studied by the HE staining method and compared with Pekin duck using comparative neuroanatomical method.

同时采用HE染色技术，对泰和乌鸡延髓和小脑及非洲鸵鸟延髓神经核团的细胞构筑进行了研究，并与北京鸭的相关研究结果进行了比较神经解剖学研究。

The cytoarchitectonic organization ofprojections to the insular cortex and the amygdala from the parvicellular ventroposterior nucleus of the thalamus andtheparabrachial nucleus in the rat was investigated by means of horseradish peroxidase -tracing technigue; The relationship of origins of afferents to the insular cortex and the amygdala from the PBN was studied by using the retrograde transport of two fluorescent tracers; Thesynaptic connections of axonal terminals with the neuronsof the PBN were observed by means of electron microscopy.

GI主要位于岛皮质的尾侧部，DI和AI主要位于首侧部；VPpc是位于丘脑腹后核和内侧丘系之间的带状区，所含细胞较小(平均14u)，其内侧部称为腹后内侧核小细胞部，其外侧部称为腹后外侧核小细胞部；PBN位于小脑上脚周围，由三部分组成：外侧亚核、内侧亚核和Kollikor-Fuse核，其中，PBl被分为7个区、PBm被分为3-4个区；杏仁核主要由三个细胞群组成：皮质内侧核群、基底外侧核群和中央核，中央核又有内侧部、外侧部和外囊部之分。

A single point mutation in intervening sequence 2 (IVS2) of human mitochondrial arginyl-tRNA synthetase gene causes abnormal cleavage of its transcript, resulting in pontocerebellar hypoplasia.

人线粒体精氨酰-tRNA合成酶基因2号内含子中的一个单点突变导致该基因的转录本被异常剪接，造成脑桥小脑发育不全。

One set of projects in the lab explores a curious feature of the main output neurons of the cerebellum, the Purkinje cells.

我们研究组的一个研究方向是小脑中的一个主要输出神经元，浦肯野细胞。

RNA interference experiences impliedthat cerebellum Purkinje cell after RNA interference have obviously apoptosis.

RNA干扰实验表明，RNA干扰后的小鼠小脑神经元出现明显的凋亡现象。

Somatotopic localization of the sensorimotor functions in the cerebellar cortex of a monkey.

猴子的小脑皮质区与特定感觉运动功能的局部化。

Machado-Joseph disease is an autosomal dominant spinocerebellar degeneration characterized by a wide range of clinical manifestations.

Machado-Joseph 疾病是晚发性的体染色体显性遗传疾病，一种小脑脊髓渐进性神经退化性疾病，此疾病的徵状在临床上变化很多，显现范围很广。

Spinocerebellar ataxias are a clinically and genetically heterogeneous group of neurodegenerative disorders.

第一部分 SCA家系致病基因定位第一章一个新的SCA家系致病基因定位脊髓小脑型共济失调(spinocerebellar ataxias，SCA)是一种具有高度临床和遗传异质性的神经系统退行性疾病，多呈常染色体显性遗传。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。