小脑

Cerebellar abiotrophy is caused by the death of Purkinje cells in the cerebellum.

小脑性生活力缺失是由小脑中浦肯野细胞死亡引起的疾病。

Another carries out input fuzzy quantificat and grading quantificat in the same time, deduces using Takagi-Sugeno fuzzy algorithm, and introduces fuzzy algorithm into weight training of CMAC. It has the characteristics of less quantificat grade and high accuracy of function approximability. It shows good effectivity used for Wiener model identifying. By syncretizing MVL, linear neuron, fuzzy control and CMAC, this paper gives a kind of FCMAC nonlinear control system with the ability of self-learning in line.

将模糊控制和小脑模型神经网络有机的结合在一起，提出了两种模糊小脑模型神经网络，一种是在CMAC基础上进行输出模糊插值，可减小非样本点误差，提高精度和训练速度；一种是对输入进行分级量化的同时进行模糊量化，利用Takagi-Sugeno模糊算法进行推理，并将模糊算法引入CMAC的权值训练，具有输入量化级数少、函数逼近精度高等特点，用于Wiener模型辨识效果好。

In addition, PTEN was detected in rat spinal cord and cerebellar granular cells cultured in vitro. It was also extensively expressed in many sites of adult mouse brain, including olfactory bulb, cortex, basal forebrain, hippocampus, amygdaloid body, and cerebellum.

此外在大鼠脊髓、体外培养的小脑颗粒细胞，以及成年小鼠嗅球、大脑皮层、基底前脑、海马、杏仁体、小脑都可以观察到PTEN的表达，提示PTEN在中枢神经系统(Central nervous system， CNS)中具有重要作用。

Only one part of the brain, in the cerebellum, was more active in women during orgasm.

在女性高潮时，脑只有在小脑的一个区域表现出更高的活性，而小脑通常与协调运动相关。

This approach visualizes the brain stem and the vermis of the cerebellum outlined by the cisterna magna.

这一途径显示脑干和小脑蚓部，其由小脑延髓池描绘出轮廓。

This approach isualizes the brain stem and the ermis of the cerebellum outlined by the cisterna magna.

这一途径显示脑干和小脑蚓部，其由小脑延髓池描绘出轮廓。

A technology of electrolytic lesion was used to destroy the projections of cerebellar FN neurons to hypothalamus in decussation of superior cerebellar peduncle.

电毁损小脑上脚交叉中顶核投射至下丘脑的神经纤维，然后再用生物素化葡聚糖胺顺行追踪观察小脑顶核-下丘脑之间神经元投射。

Hepcidin mRNA was expressed in the mice brain.Total RNA was extracted from choroid plexus, cerebral cortex, diencephalon and mesencephalon, cerebellum, medulla oblongata and pons in mice. RT-PCR analysis demonstrated that hepcidin mRNA was expressed in all areas of mouse brain, in choroid plexus the mRNA expression was highest, although its level is generally lower than that of liver. The PCR products were purified and verified the sequence.

小鼠脑内有hepcidin基因表达提取小鼠脑脉络丛、大脑皮层、间脑及中脑、小脑和脑桥与延髓组织总RNA进行RT-PCR表明，大脑皮层、间脑和中脑、小脑、脑桥与延髓、脉络丛均有hepcidin表达，其中以脉络丛表达较高，但与肝脏相比，表达量较低。

Gene mutations were detected in 25 patients and a 18-year-old girl among 16 families. Trinucleotide repeats of CAG were 73～79. The fragments of abnormal alleles were 380～402bp, and all patients were heterozygous. The copy numbers of normal alleles were 18～40, fragments from 202～270bp. SCV reduction was much obvious compared to MCV, MCV and SCV in lower limb ?ere much more slow than that in upper's. BAEP, VEP were also delayed in latency. The anticipation in parental sex bias were much more obvioius than that in matental's. Cerebellar ataxia was most severe, the next were dysarthria and bulging eyes. Amytrophy was seen only in bed ridden patients. Cerebellar atrophy was more severe than brain stem, cord atrophy was n't observed in all MJD.

结果检出10个家系25例病人及1例症状前18岁女孩有MJD基因突变，CAG三核苷酸重复73～79次，异常等位基因片段长380～402bp，均为杂合子；正常人CAG三核苷酸重复18～40次，等位片段长200～270bp，电生理发现MJD的SCV减慢比MCV明显，而下肢的MCV、SCV又较上肢明显，BAEP、VEP均有不同程度的潜伏期延长或波的异常；MJD的父亲遗传早于母亲，进展也较块，临床以小脑性共济失调为突出症状，其次为构音障碍、突眼等，肌肉萎缩仅见于晚期病人；MRI示小脑萎缩较明显，脑干萎缩并不严重，未见明显的颈髓萎缩。

Mice deficient in CRMP-1 not only showed reduced weight in cerebellum but also lack the normal organic structure in VIb lobe suggested that CRMP-1 also involved in cerebellum development.

另一方面，CRMP-1基因缺乏小鼠小脑发育也受到影响，除了重量较野生型小鼠轻外，小脑十个由颗粒层形成的摺叶中，编号第VIb的摺叶在发育上也有缺损，不明显，推测CRMP-1亦调控小脑的生长与发育。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。