小脑

Pial basement membrane breaches and glia limitans disruptions are the underlying causes of cerebellar granule neuron ectopia in POMGnT1 knockout mice and that migration into the IGL is not required for their differentiation.

1。在POMGnT-1基因敲除小鼠中，导致小脑颗粒细胞异位的原因是软脑膜基底膜层和胶质细胞的界膜的破裂，而颗粒细胞迁移到IGL并非是分化所必需。

Ray will show the associated skeletal anomalies, such as skull base invagination, scoliosis, kyphosis, cervical ribs, and many other skeletal anomalies. Axial images with CT may demonstrate an obliterated cisterna magna, tonsillar ectopia, and a normal fourth ventricle.

线可以显示出伴随的骨骼异常，如颅底凹陷，脊椎侧弯，脊椎后突，颈肋，以及许多其他的骨骼异常，CT轴位图像可以显示一个闭塞的小脑延髓池，扁桃体异位，以及正常的四脑室。

The imaging findings are characterized by sagging of the posterior fossa with low-lying cerebellar tonsils (which may be mistaken for a Chiari I malformation), elongation of the fourth ventricle, and effacement of the prepontine cistern.

其特征性的影像表现是颅后窝的下降，表现为小脑扁桃体的低位（可能被误以为是ChiariI型畸形），四脑室的延长、桥前池的消失。

Investigations in vivo and in vitro have elucidated the following: IL-2 and IL-2R have been detected in the brain; IL-2 promotes survival and neurite extension of cultured neurons, stimulates oligodendrocyte proliferation and maturation, affects the hypothalamic-pituitary function and produces behavioral and electrocorticogram spectrum changes.

研究表明IL-2在脑中主要存在于海马、下丘脑、小脑、前额皮层、纹状体、膈以及蓝斑等脑区。

The chicks show an ataxia not unlike the ataxia of vitamin E deficiency known as encephalomalacia or crazy chick disease.

雏鸡显示共济失调，但与维生素E缺乏的称为小脑软化症或疯狂病不同。

The chick s show an ataxia not unlike the ataxia of vitamin E deficiency known as encephalomalacia or crazy chick disease.

雏鸡显示共济失调，但与维生素E缺乏的称为小脑软化症或疯狂病不同。

The protein and phospholipid constituent in membrane of neurocyte in brain of chicken with encephalomalacia was separated and studied with the method of SDS-PAGE electrophoresis and the method of high performance-thin layer chromatography respectively The fluidity of neurocyte membrane of chicken with encephalomalacia was determined for the first time The content of Ca and trace elements were determined in chicken encephalomalacia systematically and the status of Fe ion in cerebellum was observed. The activity of enzymes that were relative to metabolism of energy and neuro-medium in brain were checked systematically with the method of enzyme-histochemistry. Pathological examination was carried out by microscopic and ultramicroscopic observation. The apoptosis of neurocyte in brain was checked.

在进行临床观察、定期剖杀并检测体内V〓含量、体重和脑重的基础上，重点研究了与神经损伤有关的以下几个方面的内容：系统地检测了体内自由基代谢和抗氧化系统功能的变化；首次采用SDS-PAGE电泳法分离并检测了雏鸡脑软化症神经细胞膜蛋白组分，并以高效薄层层析法检测神经细胞膜磷脂组分；测定了脑软化症雏鸡神经细胞膜的膜流动性；首次系统检测了脑软化症雏鸡体内钙及微量元素含量，并进行了小脑组织总铁离子状态观察；采用酶组织化学方法系统地观察了脑组织中与脑能量代谢和神经递质有关的酶的活性；对相关组织进行了病理组织学和超微结构观察；并对脑组织中神经细胞的凋亡进行了检测。

First, the non-brain structures removal was addressed to obtain the mask of encephalon, then the brain stem and cerebellum were separated respectively from the encephalon mask, and finally, two hemispheres were separated.

整个分割过程主要包括：首先从头部MRI图像中去除非脑组织，得到整个脑模板；然后分别从脑模板中分割提取脑干和小脑；最后分离左右两个脑半球。

First, the non-brain structures removal is addressed to obtain the mask of encephalon, then separate the brain stem and cerebellum respectively from the encephalon mask, and finally, two hemispheres are separated.

整个分割过程包括四个主要部分：首先从头部MRI图像中去除非脑组织，得到整个脑模板；再分别从脑模板中分割提取脑干和小脑；最后分离左右两个脑半球。

The results showed that two types of cNOS express and distribute expansively in cells of body.

小脑组织中bNOSmRNA含量和eNOS mRNA含量只在记忆减退时下降，老年正常组与青年组之间无差异。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。