存活率

The frogssurvival ratios at room temperature, 1℃Cand -2℃were observed.

试验结果表明：室温组和1℃组黑龙江林蛙72h的存活率为100％，-2℃组24h的存活率为84.6％。

The toxicity of trichlorphon would become degradation after a week under the same conditions. For example, the survival rate of Daphnia pulex reached to 100 percent in the concentration of 200μg/L after 144h and 65 percent in the concentration of 400μg/L after 168h.

在相同的实验条件下，敌百虫溶液的毒性可在一周后降解：如200μg/L的敌百虫溶液在放置144h后，蚤状溞的存活率就达到100％；168h后，400μg/L的存活率为65％。

Result When nimodipint dosage was 1×10^(-7)g/L^(-1), the survival rate of the neural stem cell was obviously higher than glutaminic acid in the group (P.01); With gradual increased of the nimodipint dosage, the cell survival rate of the neural stem cell damage model was obviously improved.

结果 当尼莫地平剂量为1×10^(-7)g/L^(-1)时，神经干细胞的存活率明显高于谷氨酸组(P.01)；以后随着尼莫地平剂量的逐渐增加，神经干细胞的存活率明显提高。

Separate when feeding on the leaves of MON810 and Bt11 at the 11th day, compared with 0 mortality in non-transgenic isogenic hybrid.

取食的第11天4龄幼虫全部死亡，对照幼虫存活率均为100％；在Bt11和MON810叶片上取食的粘虫5龄幼虫也不能存活，分别在取食的第11天和12天全部死亡，对照存活率分别为96.00％和98.00％。

To test the hypothesis, neontal rat cadiomyocytes Treat with heat shock(HS,42℃,2h) to induce the expression of HSP70,then teated with 0.5mmol/L H2O2. We first found H2O2 can reduced cadiomyocytes apoptosis and HSP70 only over-expression in the HS group, other two group only express HSP70 a little in the test of expression of HSP70.Then we tested the concentration of Ca2+, the [Ca2+ ] of CON was 192.224+6.654, the [Ca2+ ] of H2O2 group was 290.6918+8.922and the [Ca2+]of HS group was 214.2633+4.484.To further determine how HSP70 repaired the Ca2+ homestasis, we measured calcium transient of each group.

实验中应用MTT和流式细胞技术检测细胞的凋亡，发现损伤组的存活率明显低于其他2组，热休克组(Heat shock group，HS group)细胞存活率略低于正常组细胞；免疫组化结果表明只有在HS组才检测到阳性反应，即是HSP70的表达，其他2组均为弱阳性结果；应用离子成像技术对细胞内钙离子浓度进行测定，发现损伤组290.6918+8.922明显高于正常组192.224+6.654和HS组214.2633+4.484，HS组细胞内的钙离子浓度略高于正常组；为了进一步探讨HSP70对于ROS引起的心肌凋亡过程中钙离子的调控机制，又对各组的心肌细胞进行了钙瞬变的测定。

Kangdai Ⅰ has protective function to the damaged neurons and astrocytes: Main results:(1) It has direct protective function to the damaged neurons. It can increase the activity and survival rate, decrease the mortality and the transudation rate of LDH in cultured medium and the strong positive cell count of NOS expression of injured neurons.(2) It has also directly protective function to the damaged astrocytes. It can increase the activity and survival rate and protein content in conditioned medium.(3) It can strengthen the ability of BDNF, GDNF, bFGF, HSP and IL-6expression in damaged astrocytes.(4) It can also strengthen obviously the expressions of NSE, bFGF-receptor and bc1-2, lower the expression of bax and caspase-3.(5) It can indirectly protect and restore the damaged neurons by astrocytes. Because the effect of ACMK (ACM interfered by Kangdai Ⅰ) is stronger than ACM+K (ACM associated with Kangdai Ⅰ).

抗呆Ⅰ号对受损的神经元和星形胶质细胞均具有保护作用：主要表现为：(1)对受损神经元具有直接的保护作用，可提高受损神经元的活性和存活率，降低细胞培养液LDH的漏出率、细胞死亡率和NOS染色强阳性细胞的表达量；(2)对受损的星形胶质细胞也有直接的保护作用，可提高其活性、存活率以及培养液蛋白质的含量；(3)能增强受损星形胶质细胞分泌BDNF、GDNF、bFGF、HSP和IL-6的能力；(4)可明显增强受损神经元对NSE、bFGF的受体和bc1-2的表达，降低受损神经元对bax和caspase-3的表达；(5)抗呆Ⅰ号可通过星形胶质细胞间接地保护和修复受损的神经元，因为在多数实验组中经抗呆Ⅰ号作用的ACM的作用远大于ACM与抗呆Ⅰ号联合应用的作用，统计学上具有显著性差异。

Hochman, MD, from the New York University School of Medicine in New York City, and colleagues for the Should We Emergently Revascularize Occluded Coronaries for Cardiogenic Shock Investigators."Although survival in patients with cardiogenic shock complicating acute MI has been shown to be significantly higher at 1 year in those receiving early revascularization vs initial medical stabilization, data demonstrating long-term survival are lacking."

Hochman 医师和其同僚进行「阻塞之冠状动脉应否紧急执行血管再形成术以免发生心因性休克」研究，他指出，心因性休克依旧是急性心肌梗塞住院病患的主要致死原因，虽然心因性休克并发急性心肌梗塞之病患接受血管再形成术之一年存活率高於其他使用药物者，但是缺乏长期存活率之数据。

Albopictus showed remarkable tolerance at 10oC and the LT50 was estimated to be 526 hours, which is five times to that of Ae. aegypti. For fourth instars, Ae.aegypti had greater tolerance than Ae.

在四龄幼虫中，埃及斑蚊在2.5 及5oC的处理后存活率比白线斑蚊稍高，而白线斑蚊则是在10oC的处理后有较高的存活率。

These were raised underinducing condition to test if any morphology and structure changes. Twenty two Arabidopsismutants had various changes in morphology and anatomical structures. Twenty mutants hadlow germination rate from 10% to 60%, of which 2 had survival rates at 0% and 50%. Fivemutants occurred structure changes in the hypocotyls or stem. One of these lines, namedarris-stem, showed some unique changes: slower growth rate in comparison with the wildtype from germination to florescence; serrated margin of leaf blades, spiral rosette; morebranch in the bottom part of the stem, shorter nodes, twisted stem and branch. There were oneor several arrises along the stem. Across sections of the arrises showed one or several compactcells lumps which were round, made up of several layers of cells, looked like vascular-bundle.

在诱导条件下，这些突变体表现出不同的形态和结构的变化，共有22个表型和结构发生变化。20个突变系发芽率或存活率较低，发芽率由10％—60％，其中2个突变系存活率分别为0％和50％。5个突变系在茎或下胚轴的结构上发生变化，其中一个突变系arris-stem发生如下表型变化：从真叶出现至开花结实的整个生长期，生长速度要明显比野生型拟南芥缓慢；叶缘有明显锯齿，莲座叶呈螺旋状排列；茎的基部有较多侧枝，侧枝间距明显缩短，茎有明显扭曲；突变体茎侧面有1至数条棱形突起，内部存在一至数个排列紧密的细胞团，由多层细胞呈环形排列，细胞壁明显加厚，内部存在管状分子，推测为维管组织。

Results By incubating cardiomyocytes with bFGF, ERKs was activated and the cellular viability 〔(70.0±4.6)%in bFGF 10 ng group vs.(53.0±4.5)%in H/R group, P＜0.01） and the ATP content 〔(23.1±2.3) nmol/106 cells in bFGF 10 ng group vs.(12.3±2.1) nmol/106 cells in H/R group, P＜0.01〕 were increased, also the LDH leakage 〔(257.3±51.0) U/L in bFGF 10 ng group vs.(372.5±69.2) U/L in H/R group, P＜0.05〕, was decreased, then attenuation of H/R injury to cardiomyocytes was observed. PD098059, an inhibitor of upstream kinase of ERKs completely abolished the protective effects 〔viability (57.0±5.8)%,ATP content (15.1±2.6) nmol/106 cells,LDH activity in medium (325.5±59.0)U/L in PD098059 group vs. bFGF 10 ng group, P＜0.01, respectively〕.

结果 bFGF激活ERKs，并呈剂量依赖性减轻H/R所致心肌细胞损伤，表现为细胞存活率〔bFGF 10 ng组(70.0±4.6)%，H/R组(53.0±4.5)%，P＜0.01〕和细胞内ATP含量〔bFGF 10 ng组(23.1±2.3)nmol/106细胞，H/R组(12.3±2.1)nmol/106细胞，P＜0.01〕升高，细胞浆酶LDH漏出〔bFGF 10 ng组（257.3±51.0）U/L，H/R组（372.5±69.2）U/L，P＜0.05〕减少；ERKs上游激酶抑制剂PD098059完全消除上述保护作用〔PD098059组细胞存活率（57.0±5.8）%，ATP含量（15.1±2.6）nmol/106细胞，培养液中LDH活性（325.5±59.0）U/L，与bFGF 10 ng组比较均为P＜0.01〕。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。