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Objective: To investigate the effects of curcumine on the expresion of leptin and leptin receptor in rats with hepotic fibrosis.

目的:探讨姜黄素对实验性肝纤维化大鼠肝脏组织中瘦素以及瘦素受体表达的影响。

Methods The mesangial cells were cultured in the high glucose with or without curcumine.

体外培养大鼠肾系膜细胞,同步后采用高浓度的葡萄糖(30 mmol/l)联合不同浓度姜黄素进行干预处理。

Objective To investigate the effect of high glucose on proliferation and the production of LPA3(edg-7)in rat mesangial cells and the intervention of curcumine.

目的 探讨高糖对大鼠肾小球系膜细胞增殖及其LPA3(edg-7)表达和姜黄素的干预作用。

ObjectiveTo probe into the hyperplasy effect of different curcumine administered approaches for the splenic lymphocyte of mice.

目的探讨姜黄素3种不同给药途径对小鼠脾淋巴细胞增殖的影响。

Results ① The inhibitory rate was 68.32% in the curcumine group,70.43% in the positive controlled group .

结果①姜黄素组的抑瘤率为68.32%,阳性对照组的抑瘤率为70.43%,与空白对照组比较,两组的抑瘤率均明显升高(P.01)。

The effect of curcumine on airway inflammation in asthmatic rats may be correlated with its inhibition on over expression of GM-CSF in lung tissue.

姜黄素可抑制哮喘大鼠的气道炎症,其作用机制可能是通过抑制GM-CSF的表达而导致嗜酸性粒细胞生成减少及存活时间缩短来实现。

The survival and proliferation of the cells was quantified by MTT assay before treatment and after the treatment of curcumine.

应用MTT测量高糖及不同浓度姜黄素干预后肾小球系膜细胞增殖活力。

Curcumine could suppress the growth of androgen-dependent and androgen-indenpent prostate cancer cell lines and promote their apoptosis by different mechanism.

对于LNCaP细胞,姜黄素则不是通过活化IκBa的表达从而发挥诱导细胞凋亡的作用。

The ultimate regulation of inflammatory factor was involved, at least in part, in the mechanism of action of Curcumine and TyrphostinA1 effects.

本研究结果表明,姜黄素和TyrphostinA1能抑制油酸诱导的肺损伤,其作用机制可能与调节细胞因子(TNF-α、IL-6和IL-10)的合成与释放有关。

MethodsThe effect of curcumine on the proliferation of spleen lymphocytes through three different administration routes were determined with MTT test.

方法通过噻唑蓝比色法比较姜黄素3种给药途径对小鼠脾淋巴细胞增殖的影响。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

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双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。