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Acute onset visual agnosia points to a stroke or brain tumor.

急性发作的视觉失认指向中风或脑瘤

Visual agnosia and hyper-sexuality are the key symptoms.

主要症状为视觉失认和性欲高涨

Can lead poisoning cause visual agnosia?

铅中毒会引起视觉失认吗?

Results: the protocol has been applied at present to 29 cases, from a woman of 84 yo. diagnosed as alzheimer phase IV in 2002 with aphasia, agnosia, apraxia, continuos repetitive oscillating and like-parkinson movements, dysfunction of all sensorial recognition system, without sphincters control, to a 49 yo.

结果:这个干预方案被应用于29例病人,这其中包括年龄84岁的老年女性,该患者在2002年被诊断为"阿尔采木病4期",有失语、失认、共济失调、持续的帕金森样的震颤运动、整体感觉系统功能障碍以及括约肌功能障碍。

After comparing the clinical symptoms and physical signs between the analogous apoplexy group and apoplexy one, using American SAS statistical analysis soft ware to take logistic regression analysis on 2206 cases, combining with clinical practice, bibliography investigation and specialists experience, the main symptoms of analogous apoplexy were defined as: vertigo, sensory disturbance, severe headache, visual abnormality, involuntary movement, mental aberration, epileptic attack, agnosia, alexia and agraphia, etc.

运用类中风组与中风组临床症状体征对照的研究方法,采用美国SAS统计分析软件,对2206例调研资料进行logistic回归分析等多因素分析。参考回归结果,并结合临床实际、文献调查、专家经验,从而确定了类中风的主症为:眩晕,身体感觉障碍,剧烈头痛,视物异常,不随意运动,精神障碍,癫痫样发作,失认失读失写等。

After comparing the clinical symptoms and physical signs between the analogous apoplexy group and apoplexy one, using American SAS statistical analysis soft ware to take logistic regression analysis on 2206 cases, combining with clinical practice, bibliography investigation and specialists experience, the main symptoms of analogous apoplexy were defined as vertigo, sensory disturbance, severe headache, visual abnormality, involuntary movement, mental aberration, epileptic attack, agnosia, alexia and agraphia, etc.

将临床上不以传统中风的突然昏仆、半身不遂、口舌歪斜、语言謇涩或不语、偏身麻木为主要临床表现的西医脑卒中统归为类中风进行研究运用类中风组与中风组临床症状体征对照的研究方法,采用美国SAS统计分析软件,对2206例调研资料进行logistic回归分析等多因素分析参考回归结果,并结合临床实际、文献调查、专家经验,从而确定了类中风的主症为眩晕,身体感觉障碍,剧烈头痛,视物异常,不随意运动,精神障碍,癫痫样发作,失认失读失写等为类中风的进一步研究奠定了基础

Others include prosopagnosia (the inability to recognise faces; until recently it was thought this condition only arose through injury), dyscalculia, dyslexia, amusia and specific language impairment.

其他病症,包括面容失认症(不能辨认面容;直到近期才发现这种病症只由外伤引起)、计算障碍症、读写障碍症、失乐症和特别的语言损伤。

Normal BOLD activation in VOTC is thus insufficient to subserve intact face recognition, and disrupted information propagation between VOTC and the extended face processing network may explain the functional impairment in congenital prosopagnosia.

VOTC正常的BOLD活化不足以帮助完整面部识别,VOTC和扩展面部处理网络之间信息传递的破坏可以解释先天性面容失认症的功能损害。

To clarify the mechanisms of face recognition is not only important for theoretical psychology and physiology, but also necessary for understanding the pathogenesis of diseases such as prosopagnosia and name anomia, as well as developing feasible therapies for them.

揭示面孔识别的认知过程和脑机制不仅对面孔加工识别的研究具有重要的理论意义,而且有助于提高对面孔失认症、物体失认症和命名不能症等症候群发病机制的认识,为这类症候群病人的康复治疗提供理论指导。

Using the fMR adaptation paradigm with famous and unknown faces, we show that individuals with congenital prosopagnosia evince normal adaptation effects in VOTC, indicating sensitivity to facial identity, but show no differential activation for familiar versus unknown faces outside VOTC, particularly in the precuneus/posterior cingulate cortex and the anterior paracingulate cortex.

让患者识别知名的和未知的面孔,同时进行功能磁共振适应模式成像,我们发现,先天性面容失认症患者VOTC表现出正常的适应性,表明具有面部识别的敏感性;但是VOTC区以外区域,特别是楔前叶/后扣带皮层和旁扣带回皮层后部,识别熟悉和未知面部时的活化无区别。

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