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Resuscitations for 29 cases were successful and another 6 cases had developed cardiogenic sudden death.

总结恶性心律失常病人的急救护理措施。回顾性分析35例恶性心律失常病人的临床资料。

The incidence rates and duration of ventricular dilation-induced arrhythmias in each of verapamil, chelerythrine or glibenclamide group were similar to those in control group. Conclusion Ventricular dilation may induce arrhythmias in isolated rat hearts, which is inhibited by ischemic preconditioning. The transient increase in [Ca(superscript 2+)] appeared to be a triggering factor in the process of ischemic preconditioning. K channel PKC might also play an important roles in this process.

对大鼠心室进行膨胀可诱发心律失常;缺血预适应对心律失常的发生具有抑制作用;细胞内钙离子浓度的短暂升高可能是缺血预适应发挥抑制作用的驱动因子;ATP敏感性钾通道及蛋白激酶C在缺血预适应过程中可能发挥了重要作用。

CVB-D+ verapamil group:verapamil of 0.1 μmol/L was added into the bath firstly,10 minues later AP was recorded,then CVB-D of different concentrations (3×10 -8,1×10 -7,3×10 -7,1×10 -6,3×10 -6,1×10 -5)mol/L was added into the bath cumulately and AP was recorded every five minutes.

环维黄杨星D(cyclovirobuxine D ,CVB D)是从黄杨科植物小叶黄杨中提取的甾体生物碱,具有良好的抗心律失常、抗心肌缺血和强心作用,临床上广泛用于治疗心律失常、心绞痛和冠心病[1,2 ] 。

Objective This study is to observe the effect of carvedilol on Cx43 of infarcted rabbits and its relationship with ventricular arrhythmia.

目的卡维地洛是一种新型的抗心律失常药物,对β1、β2、α1受体都有阻滞作用,而Cx43是心室肌心电传导的基础,本组观察卡维地洛对心肌梗死边缘区Cx43分布的影响及与室性心律失常的关系。

Conclusions— BM cell injection is able to improve cardiac function in ischemic chronic heart failure but has a risk of arrhythmia occurrence when the intramyocardial route is used.

结论——对于慢性缺血性心衰骨髓细胞注射能改善心脏功能,但是直接心肌注射有引起心律失常的风险,而经冠脉注射则可减少心律失常的发生率。

Conclusions— BM cell injection is able to improe cardiac function in ischemic chronic heart failure but has a risk of arrhythmia occurrence when the intramyocardial route is used.

结论——对于慢性缺血性心衰骨髓细胞注射能改善心脏功能,但是直接心肌注射有引起心律失常的风险,而经冠脉注射则可减少心律失常的发生率。

These results define the atrial antiarrhythmic profile of a mechanistically noel antiarrhythmic drug and suggest that gap junction dysfunction may be more important in ischemic AF than in ATP remodeling or congestie heart failure substrates.

这些结果明确了这种新型抗心律失常药物的心房抗心律失常的特点,提示与快速心房起搏和充血性心力衰竭这两种房颤病理基础相比,缝隙连接功能失调可能在缺血性房颤中发挥更为重要的作用。

Results During isoprenaline administration the heart rate of arrhythmia in6cases went faster gradually and They were diagnosed as chronic sustained junctional tachycardia(3cases)and chronic atrial tachycardia(2cases)and sinoatrial reentrant tachycardia(1case)respectively;while the arrhythmias in other9cases disappered and turned to sinus tachycardiac and several minites after isoprenaline administration the arrhythmia in5cases recurred again,As a reˉsult,they were diagnosed non-paroxysmal ventricular(3cases)and non-paroxysmal junctional tachycardia(6cases)respectively.

结果 6例患者心动过速的频率逐渐增快,分别诊断为慢性持续性交界性心动过速(3例)、慢性房性心动过速(2例)和窦房结折返性心动过速(1例);另9例异位心律失常消失,转变为窦性心动过速,停止滴注后数分钟5例再次出现原心律失常,诊断为非阵发性室性心动过速(3例)和非阵发性交界性心动过速(6例)。

Results During isoprenaline administration the heart rate of arrhythmia in6cases went faster gradually and They were diagnosed as chronic sustained junctional tachycardia(3cases)and chronic atrial tachycardia(2cases)and sinoatrial reentrant tachycardia(1case)respectively;while the arrhythmias in other9cases disappered and turned to sinus tachycardiac and several minites after isoprenaline administration the arrhythmia in5cases recurred again,As a reˉsult,they were diagnosed non-paroxysmal ventricular(3cases)and non-paroxysmal junctional tachycardia(6cases)respectively.

结果 6例患者心动过速的频率逐渐增快,分别诊断慢性持续性交界性心动过速(3例)、慢性房性心动过速(2例)和窦房结折返性心动过速(1例);另9例异位心律失常消失,转变为窦性心动过速,停止滴注后分钟5例再次出现原心律失常,诊断为非阵发性室性心动过速(3例)和非阵发性交界性心动过速(6例)。

Methods: Using LAG during TEAP in 385 patients with cardiac arrhythmias or suspiciouscardiac arrhythmias.

采用润滑止痛胶法,对385例心律失常或可疑心律失常患者,行食管心房调搏检查。

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