大脑脑桥的

"The hindbrain comprises the medulla oblongata and the pons, which connects the spinal cord with higher brain levels and transfers information from the cerebral cortex to the cerebellum."

后脑由延髓和桥脑组成，连接脊髓和脑的高级部分，也包含将讯息从大脑皮质传到小脑的神经细胞。

The NOS positive neurons distributed in wide area of rabbit brain including cerebral cortex, cerebellum ,hypothalamus, mesencephalon and pons.

NOS阳性神经元几乎分布于家兔的各个脑区，包括大脑皮质、小脑、丘脑下部、中脑和脑桥。

Meaning : Stem part of the brain, consisting of midbrain, pons, and medulla.

脑干：大脑的连接部分，由中脑、桥脑以及髓质组成。

At G. 32W stable expression of nov mRNA was distributed in pontine abducens nucleus, red nucleus and substantia nigra of midbrain, ventral posterolateral thalamic nucleus and mediodorsal thalamic nucleus.

第32周脑桥展神经核、中脑红核和黑质、丘脑腹外侧核和背内侧核以及第38周大脑纹状体、顶叶皮质显示nov mRNA稳定的表达。

Following brain stem injury,the number of GFAP-positive astrocytes in cerebral cortex and pervent-ricular area,dorsomedial and ventro-medial pafts of midbrain at 30 min antemortem,and in periventricular area,dorsomedial part of pens,dorso-medial and ventro-medial part of medulla oblongata at 60 min antemortem.

采取针刺法造成实验大白鼠脑干损伤，用免疫组织化学LSAB法检测大脑、中脑、桥脑及延髓等不同部位脑组织中胶质原纤维酸性蛋白的改变。结果发现，生前损伤30min，大脑顶部灰质及中脑腹例中央部的GFAP阳性星形胶质细胞数目增多；生前损伤60min，除大脑顶部灰质及中脑腹侧中央部外。

Results The Fos reactive products were distributed in the piriform cortex,accumbens nucleus,bed nucleus of stria terminalis, cingulate gyrus,nucleus of diagonal band, hypothalamic paraventricular nucleus, supraoptic nucleus, suprachiasmatic nucleus, arcuate nucleus, central amygdaloid, subfornical organ, paraventricular thalamic nucleus, lateral habenular nucleus,ventrolateral part of the central gray,lateral parabrachial nucleus,locus ceruleus,medullary visceral zone.

结果 Fos阳性产物分布于梨状皮质、伏核、终纹床核、扣带回、斜角带核、下丘脑室旁核、视上核、视交叉上核、弓状核、中央杏仁核、穹窿下器、丘脑室旁核、外侧缰核、中脑中央灰质腹外侧区、脑桥臂旁外侧核、蓝斑、延髓内脏带等脑区，而在大脑白质及小脑中无明显的密集分布区。

They include cerebellum,cerebral cortex,hypothalamus, mesencephalon, and pons.A few of positive cells could be seen in the medulla oblongata. NADPH-d positive neurons appears blue.Their shapes were diversform such as polygon,fusiform,nummular and oval etc. Karyon hasn't pigmentation. Dendriate and axon's pigmentation are clear.

实验一利用NADPH-d酶组织化学技术，对NOS阳性神经元在兔脑的总体分布规律及其衰老性变化进行了系统研究，结果表明：①NADPH-d阳性神经元分布于兔脑各个部位，几乎涉及所有区域：包括小脑、大脑皮质、丘脑下部、中脑、脑桥和小脑，而延髓分布较少。

Cranial MRI showed round foci of signal change of different sizes in the subcortical white matter of the cerebral hemisphere, in the left paramedian portion of the pons and near the fourth ventricle.

脑部MRI见大脑半球皮质下白质的不同部位、左侧脑桥旁正中结构靠近第四脑室处散在许多圆形异常信号病灶。

There were no statistically differences between the 8th~ 14thday, the 15th - 21th day post-BC and the control group. During the rt~24tkday post-BC , pycnosis degeneration or necrosis neurons of the locus cerebral cortex, dorsal hippocampus, dentate fornix were significantly increased, then decreased gradually, but recovered to normal by the 24thday after BC, and especially in parietal cortex and piriform cortex the necrosis neurons were significantly increased than temporal cortex , and there were no statistically difference berween the left and the right side. Pycnosis degeneration or necrosis neurons in the brainstem reticular formation were markedly increased in the 4thday after BC, and there were no statistically difference among the other groups and the control group.

与对照组相比，BC后在大脑皮质、背侧海马和齿状回部位，固缩变性和不完全坏死细胞数先显著增加(P＜0.05)，然后逐渐减少，至24d基本恢复正常；顶叶、梨状皮质比颞叶皮质变性坏死细胞多，差异有高度显著性(P＜0.01)；大脑左侧比右侧变性坏死细胞稍少但差异无显著性；在BC后4d，脑桥核、斜方体核平面的脑干网状结构中固缩变性和不完全坏死细胞数明显增多(P＜0.05)，其它组间无显著性差异。

While the tissue spaces surrounding a few blood vessels wasAl and Fg positive,no Al or Fg positive cells were observed.In antemortem injurygroup,diffuse subarachnoid hemorrhage,cerebral edema,swelling or pyknotic neu-rons could be observed.The axons showed irregular swelling and disconnection at1～3h,marked swelling and disconnection at 6h,and retraction ball at 15h whichwas more remarkable at 24h after injury.The space between myelin sheaths andaxons was increased at 3～6h after injury.Tortuous and wavelike myelin sheathswhich adhered on axons incompletely,or even peeled off could be found from 15hto 24h after injury.Perinuclear lysis of Nissl bodies began at 24h after injury.Thenumber of GFAP positive cells in cerebrum and brain-stem increased significantlyfollowed by decrease,and then increased again,but the time courses of the changesin different areas of brain were not same.Al and Fg positive neural cells,mainlysurrounded blood vessels,with diffuse or peripherally distributed positive matter incytoplasm could be observed at 0.5h after injury.The number of Al or Fg positivecells and the intensity of immunoreaction increased with the time of injury.The areaof SYN positivity in medulla oblongata and pons decreased notably 3～6h afterinjury,then return to normal levels and continued to 24h after injury.

生前损伤组，可见广泛蛛网膜下腔出血，脑组织水肿，神经细胞肿胀，晚期神经元固缩；伤后1～3h见部分神经轴突不规则增粗、断裂，伤后6h断端膨大，伤后15h可见收缩球，至伤后24h更为明显；伤后3～6h可见部分神经髓鞘与轴突之间的间隙增宽，伤后15h髓鞘明显曲折，不完全附着在轴突两侧，甚至剥脱，持续到伤后24h；核周尼氏体减少在伤后24h才开始出现；同一部位的GFAP阳性细胞数目随损伤时间发生改变，先增多（最早在伤后0.5h），达到高峰后减少，其后又有增多趋势，但不同部位的GFAP阳性细胞数目增减的时间过程不尽相同，同时，大脑中的GFAP阳性细胞数目也有改变；伤后0.5h，可在脑干组织中见到Al和Fg阳性神经细胞，主要位于血管周围，阳性物在胞浆中呈弥散性分布，但部分细胞的阳性物仅分布于靠近胞膜的胞浆中而呈环状，随损伤时间延长，阳性细胞数目增多，反应强度增加；伤后3～6h，延髓及桥脑中的SYN阳性物面积减少，其后恢复到正常水平，并持续到伤后24h。

For a big chunk of credit-card losses; the number of filings (and thus charge-off rates) would be rising again, whether

年美国个人破产法的一个改动使得破产登记急速下降，而后引起了信用卡大规模的亏损。

Eph. 4:23 And that you be renewed in the spirit of your mind

弗四23 而在你们心思的灵里得以更新