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外神经胶质

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Besides hemal sex migraine, cause the cause that have a headache a lot of, if abscess of all sorts of sexual disease, encephalitis, meningitis, head is affected inside skull; All sorts of cerebral blood-vessel pathological changes, if cerebral block, cerebral hemorrhage, arachnoid haemorrhage finishing speech, ventricle bleeds, Hypertensive Cerebral disease; A pathological change is taken inside skull, if colloid tumour mixes meningeal tumour, head,transfer tumor; All sorts of heads are traumatic if defeat of cerebral concussion, head cracks injury, putamen,issue haematoma; The waist is worn cause low skull pressure head painful; All sorts of neuralgic; The headache of disease of facial features division and infection, toxic, heatstroke and nerve function all can be caused mussily have a headache.

除了血管性偏头痛外,引起头痛的原因很多,如颅内感染性疾病、各种脑炎、脑膜炎、脑脓肿;各种脑血管病变,如脑梗塞、脑出血、蛛网膜下腔出血、脑室出血、高血压脑病等;颅内占位病变,如脑膜瘤、脑胶质瘤和转移肿瘤;各种脑外伤如脑震荡、脑挫裂伤、硬膜下血肿;腰穿引起低颅压头痛;各种神经痛;五官科疾病的头痛以及感染、中毒、中暑和神经功能紊乱均可引起头痛。

Results The etiology of exophthalmos in children includes two aspects:(1)the outside of ocular diseases,mainly including Langerhans cell histiocytosis,chloroma,neuroblastoma etc.(2)ocu-lar diseases,mainly including retioblastoma,rhabdomyosarcoma of the orbit,glioma of optic nerve and angioma of the orbit and so on.

结果 提示小儿突眼病因主要包括两方面:(1)眼外疾患,主要包括有郎格罕组织细胞增生症、绿色瘤、神经母细胞瘤等;(2)眼部疾患,主要包括视网膜母细胞瘤、眶横纹肌肉瘤、视神经胶质瘤、眶内血管瘤等。

A clear cause-and-effect relationship between excessive myocardial MMP-1 activity, increased mysial collagen degradation, and progression to systolic HF has been shown experimentally through the use of transgenic models and the use of pharmacological MMP activators.53,54 In addition, experimental evidence has been provided showing that following chronic neurohormonal activation, increased synthesis and release of MMPs into the local extracellular matrix of the cardiomyocyte occurs,55 which in turn could contribute to endomysial and perimysial collagen degradation and disruption.

一个明显的因果过多的心肌MMP-1关系活动, mysial增加胶原蛋白的降解和进展收缩性心力衰竭已被证明试验,通过使用转基因模型和使用的MMP药理作用此外,activators.53,54实验证据下面被提供显示慢性神经激活时,增加的MMPs合成和释放到当地的细胞外基质的心肌细胞发生,55反过来有助于人肌内膜和 perimysial胶原退化和破坏。

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