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The other signs of AMI included bowel wall edema and thickening、massive fluid in the bowel and ascites in all 5 cases, mesenteric edema and pneumatosis in 2 cases respectively.

5例均见小肠病变肠管壁明显增厚、肿胀,肠管积液扩张及腹水,2例见肠系膜水肿,2例并肠壁积气。

Cases of superior mesenteric venous thrombosis were found, in one case, the SMV was occlusion completely. MSCTA showed the SMV and portal venous unopacification, in the other case, the filling defect was seen in SMV in portal venous, and the strip low density was seen in MSCTA.. The other signs of AMI included bowel wall edema and thickening、massive fluid in the bowel and ascites in all 5 cases, mesenteric edema and pneumatosis in 2 cases respectively.

肠系膜上静脉梗塞2例,其中1例SMV完全闭塞,管腔内无对比剂充盈,MSCTA示SMV和门静脉均未显影;另1例SMV内见充盈缺损影,MSCTA示SMV内见条状低密度影。。5例均见小肠病变肠管壁明显增厚、肿胀,肠管积液扩张及腹水,2例见肠系膜水肿,2例并肠壁积气。

The effect above is also a collective result of decreasing Ca2 and Mg2 contents and improving structure of cement paste by pozzuolanic reaction.

矿物掺合料的上述作用效果,是其降低Ca_2由水泥基材料内部向表层的扩散速度和表层Mg_2薄膜的增厚速度及改善水泥石孔结构的综合作用结果。

CT Findings were as follows : pancreas enlargement in 24 cases , focal and diffused hypodense in 39 cases , hemorrhage and necrosis of pancreas in 3 cases, peripancreatic effusion in 39 cases , prerenal fascia thickening in 39 cases , pleural effusion in 24 cases , with thrombosis in portal and SMV in 1 case . A case of pancreas head enlargement misdiagnosed for tumor,a case pancreatitis effusion was through superficial inguinal canal into scrotum.

CT影像学征象如下:胰腺体积增大24例;局限性及弥漫性密度减低39例;胰体及尾部出血坏死3例;胰周间隙渗出39例;肾旁筋膜增厚及脾肾间隙模糊39例,特别是左侧肾前筋膜均累及;少量胸腔积液24例;出血性胰腺炎伴门脉及肠系膜上静脉血栓1例;炎性渗出物通过腹股沟管蔓延到阴囊1例;单纯胰头增大,误诊为胰头肿瘤1例;本组40例中合并胆石12例。

Pseudomembranous colitis also demonstrates marked wall thickening and, occasionally, skip areas but is associated with broad-spectrum antibiotic treatment or chemotherapy.

伪膜性肠炎也表现为肠壁显著增厚,偶尔会有跳跃征,这跟广谱抗生素的应用及化学治疗有关系

Results The BMC surrounding tumor mass and strands showed multi-layered or dissolution, pseudopodia were seen extended from tumor cells, and contained numerous microvesicles.

瘤细胞伸出伪足样突起,且胞浆内含大量微泡,该处基膜消失,胶原原纤维溶解,肿瘤细胞围绕血管部位的血管基膜增厚,纤维组织增生。

Methods 8cases severe adherence acute cholecystitis,10cases repeatedly broken out,the gallbladder recrease fibross,2cases of Mirizzi sydrome the chololith occlude the neck of the cyst,the total chole-cystectomy is difficult,partly retain the gallbladder back wall of cyst,the mucosa is damaged by electrotore,partly anat-omize the cyst duct,close the duct in the proper postion.

目的 总结20例腹腔镜胆囊次全切除术的临床应用经验方法 8例粘连严重处于炎症急性期,10例反复发作胆囊壁纤维增厚,2例Mirizzi综合征结石嵌顿在胆囊颈部,强行完整切除胆囊困难,采取保留部分胆囊后壁,残留黏膜予以破坏,部分游离胆囊管,在适当位置关闭胆囊管。

Enhanced edge degradation results in increasededge oxide thickness during the poly reoxidation step.

实验发现,栅边缘退化越强,多晶硅再氧化步骤中氧化层增厚越多。

A lot of organelles such as mitochondria, rough endoplasmic reticula and Golgi bodies were observed in fiber cell. Then, double karyotheca disappeared, and the organelles disintegrated. Multivesicular bodies appeared in fiber cytoplasm. With the further development of fiber, fiber wall underwent continual thickening with aging, and polylaminate structure gradually appeared. While the agglutinated nucleus, transfer vesicles, plasma membrane and plasmodesmata still presented.

研究发现,次生壁形成早期,细胞核具有双层核膜,染色质凝聚,可见大量的线粒体、粗面内质网和高尔基体等细胞器存在于纤维细胞中;随后,双层核膜消失,细胞器将逐渐解体,多泡体开始出现在纤维细胞的细胞质;随着年龄的增加,纤维细胞壁逐渐增厚,并出现多层结构现象,而运输小泡、细胞膜、胞间连丝和凝聚的染色质将持续存在。

In electron microscopy endothelial cell of capillary and mitochondrial was edematous obviously, the basement membranes were thickened. RGCs were vacuolated, chromatin of nucleus aggregated marginally decreased and densification. Some of RGCs were necrosis, nucleus were dissolved, mitochondria was edematous, the rough endoplasmic reticula decreased obviously.

电镜下视网膜毛细血管内皮细胞明显肿胀,胞体变圆,突向管腔,线粒体肿胀,空泡化,基底膜增厚,视网膜神经节细胞胞体变形,核明显固缩,核膜消失,胞浆空泡化,线粒体肿胀,细胞表面突起减少,多聚核糖体及粗面内质网显著减少。

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