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In the experiment, an approach was made to the cause of DRP and the mechanism of it's prevention and treatmend by acupuncture. The experimental results indicated that in rat with diabetes,(1) plasma TXB2 and the ratio of TXB2 to 6-keto-PGF1α increased obviously, suggesting hyperfunction of platelet and injury to endothelial function, which tend to result in thrombosis;(2) erythrocytic filtration index increased remarkably, suggesting erythrocytic transforming and oxygen carring hypofunction, which is one of the factors leading to retinal anoxia;(3) hyperglycemia persisted, which is related to the thickening of basilar membrane of and the decrease of mural cell of retinal blood vessels;(4) serum growth hormone rose notably, which is not only one of the important factors causing basilar membrane thickening but also promotes the synthesis or Von Willebrand Factor that participates in a tendency to thrombosis.

本实验对糖尿病性视网膜病变(Diabetic Retinopathy,DRP)的成因以及针刺防治DRP的作用机理进行了探讨,实验结果表明:糖尿病大鼠(1)血浆TXB2及TXB2/6-ket0-PGF1α比值均显著升高,提示其血小板机能亢进,内皮细胞功能损伤,具有血栓形成倾向;(2)红细胞滤过指数(Filtration Index,IF)显著增高,提示其红细胞变形能力低下,携氧运行功能降低,是造成视网膜缺氧状态的因素之一;(3)呈持续高血糖状态,与视网膜血管基底膜增厚及管壁细胞减少有关;(4)血清生长激素(Growth Hormone,GH)水平明显升高,既是导致基底膜增厚的重要因素之一,又促进VWF合成,参与血栓形成倾向。

Results The diagnostic results are following:vitric umbra in 7 cases thicken interlobular spetum in b cases,camber umbro under pleural membrane in 6 cases,abnormal imaging of bronchi arteries in 7' cases,thicken pleural membrane in 7 cases,verticillate or alveolate umbra in 7 cases.which were distributed two sides of and lower field of pulmonary and pleural membrane.

结果 磨玻璃状阴影7例;小叶间隔增厚6例,胸膜下弧线影6例,支气管血管束影像异常改变7例,胸膜增厚7例,轮状或蜂窝状阴影5例,主要分布于两下肺野及胸膜下区。

Results Stenosis or occlusion of bronchotracheal tree were observed in 5 patients with localized amyloidosis; Among 4 patients with systemic amyloidosis, 3 cases of pulmonary interstitial involvement, 4 cases of pleural thickening, 4 cases of pleural effusion, 3 cases of interventricular septum and ventricular walls thickening , 1 case of pericardial effusion were observed.

结果 5例局限性病变患者均有气管支气管树的狭窄或闭塞,其中2例分别合并左舌叶与右上叶阻塞性肺不张;4例全身性病变患者中肺间质病变3例,胸膜增厚4例,胸腔积液4例,室间隔与左心室肌增厚3例,心包积液1例。

The rate of pleural effusion to diagnose is 88.9% under thoracoscopy:tuberculous pleurisy displays mainly pleura hairy nubble and diffuse white millet nubs and pleural thickeningthe trabe-form conglutination can been found; and metastatic tumor of pleura shows gray tuberculum impar and inequality of size nodosities with diffused pleural congestive and edema in the CPA and disphragmatic muscle.

结核性胸膜炎主要表现为胸膜多发结节,部分病灶出现弥漫性白色粟粒样结节和胸膜增厚,可见条索状粘连;而转移性胸膜肿瘤主要表现在肋膈角、横膈处的灰白色单结节和大小不等的多结节;非特性慢性炎症性胸膜炎主要为胸膜充血水肿,胸膜增厚纤维增生或粘连,少见有单个或多个结节增生。

The rate of pleural effusion to diagnose is 88.9% under thoracoscopy:tuberculous pleurisy displays mainly pleura hairy nubble and diffuse white millet nubs and pleural thickening,the trabe-form conglutination can been found; and metastatic tumor of pleura shows gray tuberculum impar and inequality of size nodosities with diffused pleural congestive and edema in the CPA and disphragmatic muscle.

结核性胸膜炎主要表现为胸膜多发结节,部分病灶出现弥漫性白色粟粒样结节和胸膜增厚,可见条索状粘连;而转移性胸膜肿瘤主要表现在肋膈角、横膈处的灰白色单结节和大小不等的多结节;非特性慢性炎症性胸膜炎主要为胸膜充血水肿,胸膜增厚纤维增生或粘连,少见有单个或多个结节增生。

However, the cell wall cultured at high concentration of Cu2+(10~200 mg/L) was clearly thickened and became multiple layers, and the pigment decreased and the pyrenoid became one or disappeared. Low concentration of Cu2+(0.01 mg/L, 0.1 mg/L) did not affect the growth of Chlorococcum sp., but the growth became slow and even negative beyond the concentration of 0.1 mg/L.

与对照BG11培养的绿球藻比较,0.01~1 mg/L Cu2+浓度下培养的绿球藻,细胞壁无明显增厚,色素没有多大变化,但蛋白核由一个变为多个;而在高浓度(10~200 mg/L Cu2+)下,细胞壁明显增厚为多层,色素减少,蛋白核减少并回复到1个或消失。

On the background of collision between India plate and Eurasia plate and uplifting of Tibet, the viscosity lower crustal flow induced crust thicken extensively, and driven upper middleup crust break into some big and small subblocks. The continental deformation features in SYR, including remotion of rigid subblock through the movement of huge slip faults and extensive crustal thickening, are not fit "Crustal thickening" mode or "Continental extrusion" mode.

川滇地区的大陆变形特征既存在广泛的地壳增厚现象,同时在下地壳塑性流动的驱动下,中、上地壳破碎成大小不一的块体,变形特征表现出整体刚性,因而不能简单地套用"地壳增厚"或"大陆逃逸"模式。

Results: MR demonstrated capsule wall and papillary solid lesions in one case with cystic and solid lesions of hepatobiliary cystadenoma. Poly cystic form tan if different sine in sew case, capsule wall and septation was thick, part of capsule wall was obviously thickened and nodes form, intrahepatic bile duct was dilated, large poly-antrum cystic keen form structure in one case, capsule wall and septation were smooth, part of capsule wall was uniformly thicken, capsule wall, septation, mural nodus form and solid lesions in three cases wee all slightly hyperintense on T2WI and slightly hypointense on T1WI. Part of cyst fluid was hypointense on T2WI the diameter if tumor ranged from 3.4cm to 13.5cm. Part of capsule wall, septation, solid lesions and mural nodus west obviously enhanced in arterial phase in three cases, the extent of enhancement showed a little degrade in port vein phase, balance phase and delay phase, but dense to liver parenchyma at synchronization.

结果:3例肝内胆管囊腺瘤中1例呈囊实性结构,可见囊壁及乳头状实性肿块;1例为多个大小不等的多房囊状结构,囊壁及分隔光滑,囊壁局部明显增厚,有壁结节,周围见轻度的肝内胆管扩张;1例为巨大的多房囊状结构,囊壁及分隔光滑,局部均匀增厚;3例中的囊壁、分隔、壁结节及实性部分均呈T2WI稍高信号、T1WI稍低信号,囊内液性部分均呈T2WI高信号、T1WI低信号;肿瘤瘤体大小径线范围3.4~13.5cm.3例肝内胆管囊腺瘤中囊壁、分隔、实性肿块和壁结节动脉期均明显强化,门脉期、平衡期及延迟期强化程度略减低,强化程度均高于同期肝实质。

Result showed that the epidermis dominant the morphogenesis changes. Mesenchymal is subdominant.β-catenin expression begin from horny placode epidermis stage and go up increasingly to peak value and maintain in hair follicle. The range of mesenchymal change is less.

结果表明,牦牛角的形态发生按照角基毛囊和角质外鞘发生与否分为5个阶段:角原基前期(Ed≤60)表皮增厚,无毛囊发生;角原基中期(Ed60~140)表皮持续增厚,有少量毛囊发生;角原基后期(Ed140~250)表皮变薄,大量毛囊发生;肉角期(≤0.5 a)真皮乳头发生;成角期(≥0.5 a)形成角质外鞘。

Results CT displayed the lesions located in the bladder neck and triangle area in 7 cases, in the right anterior wall in 1 cases, involve the entire bladder wall in 2 cases. Eight cases showed the local thickening or nodular or flat dune-like shape thickening of the bladder wall, the size of the lesions were 1.0cm × 0.8cm ~ 4.3cm × 5.9cm, two cases showed the diffuse thickening of the bladder wall. Cystal low density were demonstrated in 2 lesions.The bladder exine were smooth in 9 cases.

结果 10例CG中CT表现为膀胱颈部和三角区7例,右前壁1例,累及整个膀胱壁2例。8例CT平扫表现为膀胱壁局限性增厚或呈结节状、扁丘状突向膀胱腔内的软组织密度影,大小1.0cm×0.8cm~4.3cm×5.9cm,2例膀胱壁弥漫性增厚,边缘光整。2例病灶内可见囊性低密度区。9例膀胱外壁光滑。

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