基膜

Second, Electroreduction of Carbon Dioxide on Nanocrystalline TiO2-CNT Complex Film Electrode in tetrabutylammonium bromide solution and fixation of Carbon Dioxide to organic compounds.

第二部分纳米TiO_2/碳纳米管复合膜电极在四丁基溴化胺水溶液中对二氧化碳的电催化还原及电解固定CO_2为有机化合物的研究 1。

Objective To study the relationship of endangium proliferation and the expression of metalloproteinases and tissue inhibitors of metalloproteinases after angioplasty of iliac artery in rabbits to investigate the probable mechanism of composite Danshen pill in prevention and treatment of restenosisMethods 30 white male rabbits of Japan （the average body weight was 25～30 kg）were randomly divided into 3 groups:normal control group,model group (intima destroyed by balloon and given hypercholesterol diet),CDP group (intima destroyed by balloon and given hypercholesterol diet plus drug CDP 150 mg/d),10 ones each groupResults The results of iliac artery angiogram and the analysis of pathology:there were lumens stenosis,thinner intima,smaller intima area,in CDP group compared with those in model group,and ratio of intima and tunica media thickness and area among each group (P＜001)Immunohistochemistry analysis:the MMP2,MMP9,TIMP1,TIMP2 in model group significantly increased than those in control group (P＜001)The CDP group had lower MMP2 and MMP9 expression,higher TIMP1 and TIMP2 expression,and increased MMP2/TIMP1,MMP9/TIMP2 than model group (P＜001)The thickening of vascular neointima and stenosis degree of vascular lumen were relevant to MMP2 and MMP9 (r=0896,P＜001)Conclusions MMP and TIMP play the very important role in the restenosis process of arteryCDP could inhibit the thickness of vascular neointima after balloon injury,the probable mechanism of which may be inhibiting collagen formation,smooth muscle immigration and decreasing hyperplasia of intima by interfering expression of MMPs and TIMPs

目的 研究兔髂动脉成形术后血管内膜增生和基质金属蛋白酶及其抑制物表达之间的关系，探讨复方丹参滴丸预防再狭窄的可能机制。方法健康成年二级雄性日本大耳白兔30只，平均体重25～30 kg。随机分为3组：正常对照组10只，模型组10只（球囊内膜剥脱加高胆固醇饮食），治疗组10只（球囊内膜剥脱加高胆固醇饮食以及复方丹参滴丸150 mg/d）。结果兔髂动脉造影、血管病理图像分析检测结果：①复方丹参滴丸组较模型组血管造影示管腔直径狭窄、内膜厚度减少、内膜面积减少、内膜厚度和中膜厚度比、面积比，各组之间有显著性差异(P＜001)。②免疫组化分析：模型组MMP2、MMP9、TIMP1、TIMP2表达均高于对照组（P＜001）；复方丹参滴丸组MMP2、MMP9的表达低于模型组，而TIMP1、TIMP2的表达高于模型组；TIMP1/MMP2、TIMP2/MMP9明显增加，差异有显著性（P＜001）。③内膜的增生以及管腔的狭窄程度与MMP2、MMP9有很好的相关性(r=0896，P＜001)。结论 MMP和TIMP在再狭窄形成中起重要作用；复方丹参滴丸能够明显抑制血管损伤后的内膜增生，可能的机制是通过影响金属蛋白酶MMP2、MMP9及其抑制物TIMP1、TIMP2的表达从而抑制胶原的生成、平滑肌的迁移、增生，而减少内膜的增生。

Other features of the peripheral red blood cells are protein and lipid dysglycosylation and endoplasmic reticulum double-membrane remnants.

另外的特征是外周红血细胞蛋白和脂质糖基化不良以及残留的双膜内质网，其它造血系统是正常的。

The pinwheel's core contains the apical endings of B1 cells and in its periphery two types of ependymal cells: multiciliated (E1) and a type (E2) characterized by only two cilia and extraordinarily complex basal bodies.

其中风车的中心含有B1细胞的顶端末梢，而在风车周围则包含两种室管膜细胞：其中一种是多纤毛的E1细胞，另一种则是具有2纤毛以及非常复杂基体的E2细胞。

The pinwheel's core contains the apical endings of B1 cells and in its periphery two types of ependymal cells: multiciliated (E1) and a type (E2) characterized by only two cilia and extraordinarily complex basal bodies.

其车的中心含有B1细胞的顶端末梢，而在风车周围则包含两种室管膜细胞：其中一种是多纤毛的E1细胞，另一种则是具有2纤毛以及非常复杂基体的E2细胞。

The changes of free radicals metabolism and membrane ingredients and morphology of mammary gland epithetical cells in cow suffered from Alcohol Positive Milk were studied in this paper for studying the molecular mechanism of APM/s occurring further.

为了进一步研究酒精阳性乳的发病机理，本研究从乳腺上皮细胞自由基代谢、膜组分改变和形态学变化的角度对酒精阳性乳的发病机理进行了探讨。

Leaves 2(or 3), basal, long petiolate; petiole erect, equitant, forming a pseudostem, proximally enveloped by several cylindric, membranous sheaths.

叶2(或3)，基部，具长叶柄；叶柄直立，套折，形成假茎，下部被数个圆筒状，膜质鞘包围。

Recent studies have demonstrated that free radicals, excitatory amino acids, and membrane permeability to calcium are all important factors in ischemic brain injury.

最近研究表明，自由基、兴奋性氨基酸和膜对钙的通透性是缺血性脑损伤的重要因素。

The process contains the proliferation of cells and the accumulation of extracellular matrix components.

这种增生膜的形成主要包括细胞的增生和细胞外基质的沉积。

To regulate tumor cells proliferation and metastasis. In many malignant tumors, CD44 has shed by MT1-MMP extracellularly, and causes the release of the soluble CD44, which facilitate the tumor cells motility.

在多种恶性肿瘤中，CD44分子胞外结构域可被膜结合型基质金属蛋白酶1(MT1-MMP)剪切，导致&可溶性&CD44分子的释放，此过程在细胞迁移中发挥重要作用。

Do you know, i need you to come back

你知道吗，我需要你回来

Yang yinshu、Wang xiangsheng、Li decang,The first discovery of haemaphysalis conicinna.

1〕 杨银书，王祥生，李德昌。安徽省首次发现嗜群血蜱。