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From the dehydration of acetic acid in industry, novel membranes used for this purpose were prepared by modifying Cardo polyetherketone (which is solvent resistance and hydrophobic) for the first time.

基于醋酸脱水的重要的工业意义,本论文首次以耐化学溶剂性能优异的亲油性材料酚酞侧基聚醚酮为膜材料,通过对其进行改性,制备醋酸渗透蒸发脱水膜。

It is postulated that IP induces the release of endogenous protective molecules that interact with cognate receptors to activate PLC through mediation of the chincough virus sensitive proteins. PLC hydrolyzes membrane phospholipid and produces diacylglyccerol,that,by working together with calcium hydronium,translocates PKC from cytoplasm to cell membrane and activates PKC.

推测缺血预处理细胞保护作用机制是:缺血预处理引起内源性保护物质释放,作用于细胞膜上的相应受体,通过百日咳病毒敏感性G蛋白的介导而激活磷脂酶,磷脂酶水解膜磷脂,产生二酰基甘油(diacylglyccerol,DAG),DAG和Ca2+协同作用于PKC,使其从胞浆转位到细胞膜并且被活化,PKC在膜上使底物蛋白磷酸化,引发一系列相关信号转导通路的激活,调控保护性基因的表达,从而提高细胞对缺血再灌注损伤的耐受性。

One method of preparation new membrane materials was presented with putting multi-wall carbon nanotube and carboxylic multi-wall carbon nanotube in collodion solution.

分别将碳纳米管和羧基化碳纳米管均匀分散在火棉胶溶液中,采用流涎法制备出碳纳米管复合膜;并用分光光度法对样液、滤液中的间苯二酚和茜素红的吸光度进行了对比测定,以考察膜的吸附性能。

The results show that:Changing the substituents of the phenyl rings,on the one hand,will influence the capacity of conjugation of the phenyl rings through S→N dative bond and furthermore influence the weak acid-dissociation of phenolic hydroxyl,on the other hand,will influence the geometric conformation of the compound and furthermore influence to form structure matched with membrane and water/membrane.

结果表明:改变药物化合物两边苯环上的取代基,一方面,将影响苯环通过S←N授受键的共轭能力,进一步影响酚羟基的弱酸离解常数;另一方面,将影响C-S,S-N键自由旋转的难易程度,进一步影响到能否形成与膜/水膜界面相匹配的结构。

To get rid of the usual assumption that temperature field in diamond water of substrate is uniform or linear so as to make the simulation results closer to the actual conditions under which the diamond water can be fabricared, the plasma jet from a plasma torch controlled by DC electromagnetic field was introduced in simulation.

为了使模拟更接近于金刚石膜的真实制备环境,本文取消了以往对金刚石膜或基体内的温度场常作的均匀或线性的人为假设,而采用对从磁控直流等离子体炬内喷射出的射流仿真计算结果。

The reasons of RA focus on disbalance of immunity net, the effect of so many cytokines and MMPs , and the tumor-like proliferation of fibroblast-like synovicytes in joints, though the cause of developing disease is unknow.

RA的发病过程中,免疫网络失衡,加上成纤维样滑膜细胞(fibroblast-like synoviocytes,FLS)的类肿瘤样生长及所分泌的大量促炎性细胞因子、趋化因子和金属蛋白酶,是导致关节内炎性细胞聚集,滑膜细胞增生和向周围组织侵蚀及基质降解和破坏的主要因素。

The thickness of diamond-like film deposited on monocrystalline silicon substrate was measured by elliptically polarized light. According to the result, the film distributes over the whole surface of substrate randomly.

用椭圆偏振法测量了单晶硅基体上沉积的类金刚石薄膜,同时从所测得的膜厚值中,发现运用该沉积装置所沉积的类金刚石膜在基片表面上的厚度值呈随机分布状态。

Objective To explore the invading mechanism of amebae in lamina porpria and observe the interaction between the cysteine proteinase of Entamoeba histolytica and laminin.

目的 探索溶组织内阿米巴通过基底膜进入固有膜的机制,了解其半胱氨酸蛋白酶(cysteine pro-teinase,CP)与胞外基质的相互作用。

Acellular Extracellular Matrixis three dimensional mesh stent which mainly composed by vessel of basilar membrane and coated by epineurium. As a bioactive nerve graft,it own natural construction,lower immunogenicity.

脱细胞神经细胞外基质材料是以SC基底膜管为主、以神经外膜为外套的完整的三维网状支架;它作为有生物活性的神经替代物,具有良好的仿生性,较低的免疫原性。

Beijingensis under three droughttreatments, normal, moderate and severe stresses. The results showed:(1) the canker disease ofboth cultivars was serious gradually with increased drought;(2) the bark tissue cells sufferedplasmolysis, more evident with the severity of drought stress;(3) under the drought andinoculation with B. dothidea, cells of two cultivars damaged at different degree, mailyrepresented in the changes of organelles, such as chloroplast swollen and distorted, number ofmitochondria increased and membrane system indistinct; then organelles suffered furtherdamagement with inoculation time, thinned mitochondrias stroma, decreased cristae, crumpledand partly broken membrane of chloroplasts with stroma exosmosis. At last, the chloroplastspartly disorganized;(4) the hyphae growed mainly intercellular in resistant cultivar and notonly intercellular but also intracellular in susceptible cultivar, which directly caused thenecrosis of cells;(5) under the severe drought, the damage of cells enhanced the infection ofpathogen and drought and pathogen stressed together and promoted the disease development;the damage from pathogen on cells was more serious than that from drought.

结果显示:(1)随着干旱胁迫程度的增加,2种杨树溃疡病害发生渐趋严重;(2)干旱胁迫下,杨树树皮组织细胞发生质壁分离,并随胁迫程度的增加而严重;(3)干旱胁迫下接种病原菌,2种杨树细胞发生不同程度的损伤,主要表现为细胞器发生较大变化,如出现叶绿体肿胀变形、线粒体数量增多,质膜模糊不清等现象;随接种时间的延长,细胞器受到进一步损伤,叶绿体被膜折皱,严重时局部破裂,基质外渗,并部分最终解体;(4)毛白杨中的菌丝主要在细胞间隙中穿行,而北京杨的菌丝除在细胞间隙中生长之外,侵入细胞内部也较多,直接导致细胞的解体;(5)干旱胁迫下细胞的损伤促进了病原菌的侵染,干旱和病原菌的双重胁迫加剧了病害的发生程度,并且病原菌侵染对细胞的破坏程度大于水分胁迫。

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