坏死的

Objective: To explore the relationship between corneal storage methods and rejection after corneal transplantation, and to study the roles of intercellular adhesion molecule-1 (ICAM-1) and tumor necrosis factor-α in aqueous humor and serum in keratoplasty rejection in rats.

目的：探讨角膜保存时间与术后免疫排斥反应的关系，检测角膜植片的细胞间黏附因子-1（ICAM-1）表达及房水、血清中肿瘤坏死因子-α含量与角膜移植排斥反应的关系。

Tumor Color: general tumor cut surface ash gray or red, depending on the size of its containing blood, with or without hemorrhage, degeneration, necrosis, etc., may be.

肿瘤的颜色：一般肿瘤的切面呈灰白或灰红色，视其含血量的多寡、有无出血、变性、坏死等而定。

When there is extensive caseation and the granulomas involve a larger bronchus, it is possible for much of the soft, necrotic center to drain out and leave behind a cavity.

当存在广泛的干酪性坏死并且肉芽肿累及较大的支气管时，大量的坏死物排出，留下空洞。

Injury to foliage may be visible in a short time and appear as necrotic lesions, or it can develop slowly as a yellowing or chlorosis of the leaf.

对叶子的破坏在短时间内是可见的且呈现出坏死组织，或也表现为叶子的慢慢变黄。

Methods Mimicry and speculation on possible displaced amino acids in proper positions of peptide increased the biological effects of TNFR blocking peptide by compote modeling. The original peptide and its mutant were synthesized. The competitively inhibitory effects on green fluorescence protein-tumor necrosis factor fusion protein with TNFR were compared between original peptide and its mutant (receptor competitively inhibitory binding assays). The inhibitory effects on cytotoxicity were compared between original peptide and its mutant.

通过计算机模建，模拟并推测提高TNFR封闭肽生物学效应需置换的位点和氨基酸；合成原封闭肽和突变肽；受体竞争抑制结合实验比较原肽与突变肽对绿色荧光蛋白－肿瘤坏死因子融合蛋白结合TNFR的竞争抑制效应；细胞毒作用抑制实验比较原肽与突变肽对TNFR的封闭效应。

The overexpression of HO-1 induced by CoPP can inhibit death of the myocardial cells and subsequently alleviate myocardial ischemia/reperfusion injury,whose major mechanism may be related with anti-oxygen free radicals.

CoPP诱导的HO-1过表达可以抑制心肌缺血再灌注损伤后的细胞坏死，从而减轻心肌的再灌注损伤，其主要机制与抗氧自由基有关。

The upregulation of HO1 expression induced by CoPP can inhibit the necrosis of the myocardial cells and subsequently alleviate the myocardial ischemia/reperfusion injury. One of its main mechanisms may be related with antioxygen free radicals.

CoPP诱导的HO1过表达可以抑制心肌缺血-再灌注损伤后的细胞坏死，从而减轻心肌的再灌注损伤，其主要机制与抗氧自由基有关。

Result HO-1 expression was upregulated following pretreatment of CoPP before reperfusion,resulting in both an increase in SOD activity and a decrease in MDA content and thus in a reduction in death of the myocardial cells after ischemia/reperfusion injury.

结果：再灌注前使用CoPP进行预处理，可以诱导HO-1蛋白的表达上调；HO-1蛋白表达上调可以减少缺血再灌注后的心肌细胞坏死，提高心肌组织中SOD含量并降低MDA的含量。

No DNA fragments were observed in electrophoresis in scrotal testis and cryptorchid testis on 3d.

而混合培养的生精细胞，在30min、1h可见明显的DNA ladder小片断，2h及12h则主要以坏死细胞造成的弥散小片断DNA为主。

The panel has also added recommendations for managing intra-abdominal infection in children, particularly where such management differs from that of adults; for appendicitis in patients of all ages; and for necrotizing enterocolitis in neonates.

专家组还增加了管理儿童腹腔内感染的建议，特别是在治疗不同于成人的地区，还增加了所有年龄患者阑尾炎和新生儿坏死性小肠结肠炎的建议。

She gently rebuff ed him, but agreed that they could be friends

她婉言拒绝了，但同意作为朋友相处。

If in the penal farm, you were sure to be criticized.

要是在劳改农场，你等着挨绳子吧！