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To that purpose, we studied the effects of carrageenan, bupivacaine, or both on the production of tumor necrosis factor-alpha, interleukin-1beta, and IL-10 by whole blood cultured in the presence of lipopolysaccharide and of heat-killed Staphylococcus Aureus Cowan.

我们研究了角叉菜胶、布比卡因或二者对脂多糖和热处理后金黄色葡萄球菌 Cowan 株培养的全血а-肿瘤坏死因子、白介素-1β, IL-10产生的作用。

Microscopically, the ulcer here is sharply demarcated, with normal gastric mucosa on the left falling away into a deep ulcer whose base contains infamed, necrotic debris.

显微镜下,溃疡边界清楚,左边正常的胃黏膜脱落形成一个深的溃疡,其基底部有炎症和坏死碎片。

Objective To study the role of tumor necrosis factor in the mechanisms of liver injury and the relationship between LPS-induced apoptosis in hepatocyte, kupffer cell, and hepatic damage.

目的 探讨内毒素、肿瘤坏死因子在诱导枯否细胞及肝细胞凋亡过程中的作用及其与肝脏损伤的关系。

TP and EGCG can protect liver against necrosis and inflammation induced by alcohol, and the mechanism may involve its effect on the reduction of plasma endotoxia, and inhibition of Kupffer cell activity and proinflammatory cytokine expression and secretion.

茶多酚及EGCG可减轻酒精性肝损伤大鼠肝脏的炎症与坏死,其可能机制包括降低内毒素血症,抑制Kupffer细胞活性与促炎细胞因子的表达与分泌。

These animals were killed after 24 hours of treatments and instillation of meconiums. The measurements included lung tissue wet/dry ratio,lung injury score,the expression and level of AngⅡ,lung myeoperoxidase activity and TNF-α level.

模型制备后24h取材,观察其肺组织病理损伤并检测湿/干重比及进行病理评分;检测AngⅡ在肺组织的表达情况及用图像分析方法检测其含量;测定肺组织中肿瘤坏死因子-α的含量及肺匀浆髓过氧化物酶活性。

The compositions of fatty acids in mid-gut gland of cultured healthy Penaeus monodon, healthy Penaeus japonicus, P. monodon slightly infected by MBV, P. monodon severely infected by MBV and P.

应用气相色谱法分别检测了人工养殖的健康斑节对虾、健康日本对虾、斑节对虾杆状病毒轻度感染斑节对虾、MBV重度感染斑节对虾及患中肠腺坏死杆状病毒病日本对虾中肠腺脂肪酸的组成及含量。

To study the effects of leptin on tumor necrosis factor α secretion in murine peritoneal macrophages and the role of mitogen activated protein kinases in this process.

研究瘦素诱导小鼠腹腔巨噬细胞分泌肿瘤坏死因子α的影响,以及丝裂原活化蛋白激酶信号通路在这一过程中的作用。

In order to study the effects of pre-heating on the sensitivity of cells to tumor necrosis factor-α, the K562 cells were pre-heated at 40℃ for different times to induce heat shock protein 70(HSP70). To observe the effects of TNF-α concentration on the sensitivity of pre-heated cells, cells were pre-heated for 60 min and treated with TNF-α(50,100,200 and 400U/ml) for 16 hours, and the cells vitalities were detected by MTT.

为探讨预热对肿瘤细胞肿瘤坏死因子α敏感性的影响及其机理,将白血病患者胸腔渗出细胞系(K562细胞)在40℃预热不同时间(0、30、60、90、120、150和180min),使细胞热应激蛋白70(HSP70)高表达;然后将预热60min细胞和未预热细胞分别暴露于50、100、200和400U/ml的TNF-α16小时,用MTT比色法检测细胞活力。

Results demonstrated that serum sICAM 1 was significantly increased in each stage of severe hepa titis and in each type of chronic hepatitis,compared with healthy donors and AsC.

表明:从AsC至重肝早期,血清sICAM-1水平随着患者肝损害的加重而升高,能较好反映肝组织炎症坏死程度,重肝患者血清sICAM-1水平随着肝衰竭的加重而降低。

Similarly, there was a graded increase in risk of stroke across quartiles of inflammatory markers in the placebo patients only.

与此相似,卒中的风险随着炎症和心肌坏死四分位数的升高而逐级增加。

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