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The morphological change was observed with transmission electron microscope, compared with the control cells, the irradiated cells have more cytoclasis and cytolysis, the pykonosis and apoptosis corpus were also observed.

电镜下胞浆中空泡形成,细胞溶解、坏死,细胞核固缩,出现典型的细胞凋亡改变,形成凋亡小体。

YiQiHuaYu therapy protected the vascular endothelial cells by decreasing the level of cytotoxin TNFa.

益气化瘀法能降低细胞毒性因子肿瘤坏死因子水平,以减轻对血管内皮细胞的损害。

Why? Your nerves have been deadened fast so that you now feel little pain.

为什么?因为你的神经迅速坏死,所以你较感觉不到痛。

Objective to investigate the effect of mifepristone on the expression of tumor necrosis factor-α and transforming growth factor-βin decidua of early pregnancy.

目的 探讨米非司酮对胚胎生长及蜕膜肿瘤坏死因子-α和转化生长因子-β表达的影响。

Results Punctiform erosion, hemorrhage were observed by nake eyes in each groups. Epithelium cellular necrosis, defluxion, neutrophil being dominant in propria lamina and intact glandular were observed with microscopy. The erosion became severe with the successive stimulate of ethanol. There is no difference between distillate spirit and sodium chloride rats in Guth in 7th day, but Guth in distillate spirit rats was higher than that in sodium chloride rats in 30th day. PGE2, TGF-α contents in gastric mucosa of treated group increased significangly compared with control group. PGE2 showed no difference between each group in 7th and 30th day; TGF-α in gastric mucosa of distillate spirit and sodium chloride rats decreased after 1 month.

结果 茅台组、乙醇组、生理盐水组胃黏膜均肉眼可见程度不同的点状糜烂、出血;光镜下黏膜部分上皮坏死脱落,固有层以中性粒细胞为主炎性细胞浸润,腺体结构基本完整,病变随时间延长有所加重,Guth积分1周茅台组与生理盐水组差异无统计学意义,1月两者相比差异有统计学意义;茅台组、乙醇组胃黏膜组织内PGE2水平与生理盐水组相比明显增高,同时TGF-α表达增强;但PGE2水平各组间1周与1月相比差异无统计学意义;TGF-α表达强度在茅台组、生理盐水组1月后逐渐降低,差异有统计学意义。

Purpose To investigate the effect of LPS and dialyzer membrane on tumor necrosis factor α and soluble tumor necrosis factor receptor p55(TNFsRp55)production in PBMC from uremic patients and healthy controls.

目的 研究不同透析膜和内毒素对正常人和尿毒症患者外周血单个核细胞肿瘤坏死因子α及其可溶性受体p5 5 (TNFsRp5 5 )产生的影响。

AIM To investigate the relationship between plasmic diamine oxidase and gut injury in acute necrotizing pancreatitis.

目的 探讨急性坏死性胰腺炎时血浆二胺氧化酶水平变化及其与肠道损伤的关系。

But the malignant tumor may also disorganize, organ's structure and the function, causes necrosis hemorrhage merge infection, the patient is possible as a result of the organ function failure to die finally.

但恶性肿瘤还可以破坏组织、器官的结构和功能,引起坏死出血合并感染,患者最终可能由于器官功能衰竭而死亡。

The changes of CEA and TNF in the pleural effusion were examined before and after the treatment.

并检测治疗前后胸水中癌胚抗原和肿瘤坏死因子的改变。

Then made blood flow slowly and coagulate untile thrombosis, epithelial cell of vessel wall edema,aseprtic inflammation and necrosis,made the hemorrhoid's vessels atrophy,emphraxis,decrease,diminuting the volume of the vessels,relieving the pressure,at last adherence and fibrosis to gain the ends of hemostasia.

同时血管壁上皮细胞水肿,产生无菌性炎症、坏死。两种作用使痔血管萎缩、闭塞,痔组织发生粘连及纤维化而达到治疗的目的。

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