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Hours after operation, most nerve cells disappeared and the glial cells were degenerated. There were many mono-nucleus cells infiltrating in transplantation group while a few in control group. One week after operation, tissues in the infarction zone were liquescent with many monocaryons and lymphocytes infiltrating around them in control group.

术后48 h神经细胞大片消失,胶质细胞变性,自体骨髓干细胞移植组有大量单个核细胞浸润,对照组相对较少。1周后对照组梗死区脑组织液化变软,坏死灶周围可见较多的单核和淋巴细胞浸润;自体骨髓干细胞移植组大鼠部分坏死灶被新生毛细血管和胶质细胞增生修复,炎性细胞浸润不明显。

Arsenic poisoning have magma role to paralysis small blood vessels,mercapto Jimei inhibit the activity of the liver and fat necrosischange Hepatic Lobules Centre, heart, liver, kidney, intestinecongestion, epithelial cell necrosis, telangiectasia.

砒霜有原浆毒作用,能麻痹细血管,抑制巯基梅的活性,并使肝脏脂变肝小叶中心坏死,心、肝、肾、肠充血,上皮细胞坏死,毛细血管扩张。

Arsenic poisoning have magma role to paralysis small blood vessels, mercapto Jimei inh ibit the activity of the liver and fat necrosis change Hepatic Lobules Centre, heart, liver, kidney, intestine congestion, epithelial cell necrosis, telangiectasia.

砒霜有原浆毒作用,能麻痹细血管,抑制巯基梅的活性,并使肝脏脂变肝小叶中心坏死,心、肝、肾、肠充血,上皮细胞坏死,毛细血管扩张。

AIM To study the long-term toxicity of modified recombinant human tumor necrosis factor rhTNF-NC in Macaca mulatta compared with recombinant human tumor necrosis factor rhTNF.

目的 研究新型重组人肿瘤坏死因子rhTNF-NC iv对恒河猴的长期毒性,并与重组人肿瘤坏死因子rhTNF作比较。

To investigate the effect of tumor necrosis factor-αon lung injury of rats with acute necrotic pancreatitis.

目的 探讨肿瘤坏死因子-α在急性坏死性胰腺炎大鼠肺损伤的作用。

Objective To explore the treatment of ulinastatin in acute necrotic pancreatitis.

目的 探讨乌司他丁治疗急性坏死性胰腺炎的作用,为临床治疗急性坏死性胰腺炎提供参考依据。

The number of DC is more in the cases of those that have more necrotic areas and lymphocytes infiltration.

树突状细胞形态不规则,绝大部分DC分布于坏死灶和汇管区间质中与淋巴细胞伴行,肝细胞内一般不常见,坏死灶越多,淋巴细胞浸润越多的病例中DC细胞也越多。

The first, many tuberculosises in glandular stomach were found besides liver cell degeneration, pneumoncongestion, epithelium cell degeneration of tubuli tenales, necrosis and kidney hemorrhage, cardiac muscle celldegeneration, heart hemorrhage, cerebrum hemorrhage, satellitosis and neuronophagia, cerebellum purkinje cell necrosis.

发现病例1,肝细胞变性,肺淤血,肾小管上皮细胞变性、坏死,间质出血,心肌纤维变性,间质出血,大脑出血,&卫星化&与&噬神经&现象,小脑浦肯野氏细胞坏死,还可见腺胃有大量的结核结节。

Results:(1) In clinical pathology, swelling, denatured, even necrosis and putrescence could be picked out; Collagen fibers in tendon remarkedly swelled, and tissue structure disarranged, and collagen even ruptured, broken and dissolved; Obvious denature, apoptosis and necrosis could be found in tendon cells. There appeared to be a big accumulation of inflammatory cells.

结果 (1)临床病理组织学观察发现:肌腱组织肿胀、变性、甚者坏死、腐败;肌腱内胶原纤维肿胀明显、组织结构紊乱,失去正常排列,甚者出现胶原断裂、破碎、溶解等现象;腱细胞可见明显的变性、凋亡和坏死;病变部位可见大量炎性细胞聚集。

Underlight microscope,we can find edema and few focal necrosis of right nerve cells of neonate rats after 2 to 6 hours of hypoxic-ischemia. We can find clear necrosis in brain tissue after 72 hours of hypoxic-ischemia.

HE染色缺血缺氧后2-6h右侧脑神经细胞水肿及少量局灶性坏死,缺血缺氧后72h右侧脑组织中可见界限清楚的坏死灶。

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