坏死的

Main clinical symptom of poult gooses were depressed, retardation in reaction, food appetite decreased, alo laxata; and the average death time was 62.6 h Mark processes in dissection were that spleen was intumescenced, gray necrosis appeared; haemorrhage in pancreas, gray necrosis appeared; haemorrhage in intestinal tract, anabrotic; the HA titer of the virus isolated from these samples could reached as high as 2^8, and had the chicken red blood cell agglutination and lys agglutination ability.

雏鹅平均死亡时间为62.6 h。特征性剖检病变为脾脏肿大，有灰白色坏死点；胰脏出血，有灰白色坏死点；肠道出血，溃疡；病料分离培养的病毒液HA价高达28，能凝集鸡红细胞，并出现解凝现象。

Methods: We collected five cases of necrotizing scleritis in the past 2 years. All 5 cases received conjunctival flap advancement as treatment for necrotizing scleritis.

自2004年5月至2005年4月期间内，我们收集5位罹患非感染性坏死性巩膜炎的病人，并采用结膜瓣手术治疗这五位坏死性巩膜炎病人。

Dexamethasone treatment group, the hearts showed cardiac musclewaxy necrosis, bubble apomorphosis, the livers showed a large area of cellularnecrosis, the spleens showed hemorrhagic infarct, the lungs showed hemorrhage andhyperemia, serous fluid exudation, the kidneys showed granular degeneration, thecerebrum showed nerve cell swelling.

地塞米松治疗组的心脏表现为心肌发生蜡样坏死，水泡变性；肝脏表现为肝组织出现大面积坏死；脾脏表现出血性梗死；肺脏表现为出血充血，浆液渗出；肾脏表现为颗粒变性；大脑表现为神经细胞肿胀，变圆。

Pannecrosis was observed after 24 hours. Ischemia-dependenced pathologic change completely fits sigmoidal dose-response curves.

结果：缺血30 min，坏死神经元首先大量出现在缺血侧基底节区，以后逐渐扩展到皮层，至缺血24 h后，缺血中心逐步出现凝固性坏死，缺血时间依赖性病理变化完全符合剂量反应曲线的S-形分布。

The results showed that a large amount of Actinobacillus pleuropneumoniae with gram negative infections could be seen in the secretion of bronchus; There were congestion in liver, necrosis, hemorrhage, fibrosis with neutrophils and macrophage infiltrating in lung tissue, necrotic splenitis in spleen, and hemorrhagic lymphadenitis in lymph node.

结果表明；在支气管分泌物涂片中含有大量呈革兰氏阴性的胸膜肺炎放线杆菌；肝脏淤血，肺组织坏死、出血、显著纤维化及嗜中性粒细胞和巨噬细胞浸润；脾脏呈坏死性脾炎变化，淋巴结呈出血性淋巴结炎变化。

Results: The brain fabric was normal without edema, hemorrhage and necrosis in groupⅠ. The brain parenchyma were loose, perivascular canal became widen, some neurocytes bulk and degenerate in groupⅡ. In groupⅢ, the brain injury aggravateed accompanying the lapse of survival time with extreme cerebral edema at 24 h, and colliquative necrosis in the brain parenchyma aparted at 40 h.

结果：Ⅰ组脑组织结构清晰，无水肿、出血、坏死病灶；Ⅱ组脑实质疏松，小血管周隙增宽，部分神经细胞体积增大变性；Ⅲ组随溺水后存活时间的推移，脑损伤程度逐渐加重，其中24h脑水肿最重，40h个别脑实质小灶性液化性坏死。

American Atlanta conference edited in September 1992 pancreatitis of serious illness acute definition is: AP companion has local complication (cyst of pancreatic and necrotic, false sex, pancreatic abscess) or the organ fails (function of shock, lung not complete, kidney declines, gastrorrhagia, sow medicinal powder the cruor inside sexual blood-vessel and serious metabolization obstacle), or both holds concurrently.

概要： 急性胰腺炎为临床常见急腹症，是由于胰液和溶蛋白酶漏入到胰腺实质和胰腺四周组织而引起。1963年，法国马赛会议以病理形态学为基础，将AP分为水肿性胰腺炎和出血、坏死性胰腺炎。1992年9月美国亚特兰大会议修订重症急性胰腺炎定义为：AP伴有局部并发症(胰腺坏死、假性囊肿、胰腺脓肿)或器官衰竭(休克、肺功能不全、肾衰、胃出血、播散性血管内凝血和严重的代谢障碍)，或二者兼有。

Blinded biopsies were evaluated by a histopathologist and scored according to Knodells histology activity index. Results 53.5%(54/101), 51.5%(52/101) and 31.7%(32/101) patients had a reduction of their total hepatic HAI score, necroinflammation and fibrosis scores by ≥2 points or 1 points at the end of one year of lamivudine therapy, compared with their pretreatment values, respectively.

结果 治疗后53.5%（54/101）患者肝组织学改善（治疗后HAI积分下降≥2），HAI积分由治疗前的8.0±4.7下降至治疗后5.2±3.3（t＝7.358，P.01）；其中51.5%（52/101）患者坏死炎症程度改善(治疗后积分下降≥2)，坏死炎症积分由5.9±3.8下降至3.6±2.5；31.7%（32/101）患者纤维化程度改善（治疗后积分下降≥1），纤维化积分由2.1±1.2下降至1.6±1.2（t＝3.827，P.01。

Labeled perforin cDNA probe in liver biopsy tissues. RESULTS: Perforin positive cells were seen in the whole intralobular areas, especially in inflammatory areas. The amount of positive cells was different, 71.9% in spotty necrosis areas, 31.0% in focal necrosis areas, 9.2% in portal areas and only 4.8% in non????

结果： Perforin阳性细胞在整个肝小叶内均可检见，有炎性细胞浸润区均有perforin阳性细胞分布，浸润阳性细胞数量因病变部位的不同而有较大差异，慢性丙肝点状坏死区阳性率最高达71.9%，其次是片状坏死区(31.0%)，门管区阳性率为9.2%，小叶内非坏死区仅为4.8%。

RESULTS: Perforin positive cells were seen in the whole intralobular areas, especially in inflammatory areas. The amount of positive cells was different, 71.9% in spotty necrosis areas, 31.0% in focal necrosis areas, 9.2% in portal areas and only 4.8% in non necrotic areas. The average amount of perforin cells in the liver lobules of 18 patients had no linear correlation with the ALT level or Knodell's histology activity index.

结果： Perforin阳性细胞在整个肝小叶内均可检见，有炎性细胞浸润区均有perforin阳性细胞分布，浸润阳性细胞数量因病变部位的不同而有较大差异，慢性丙肝点状坏死区阳性率最高达71.9%，其次是片状坏死区(31.0%)，门管区阳性率为9.2%，小叶内非坏死区仅为4.8%。

She gently rebuff ed him, but agreed that they could be friends

她婉言拒绝了，但同意作为朋友相处。

If in the penal farm, you were sure to be criticized.

要是在劳改农场，你等着挨绳子吧！