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Methods the caput femoris necrosis models were established in 23 rabbits with bilateral caput femoris necrosis, and 45 necrotic caput femoris were randomly divided into 3 groups with 15 necrotic caput femoris in each group.

23 只新西兰白兔,用液氮冷冻的方法,建立双侧股骨头坏死模型,取其中45 个坏死股骨头,随机分组,分别为对照组,实验1 组,实验2 组。

CONCLUSION摘要: DWI and ADC value are helpful to differentiate capsule formation stage of abscess from cystic/necrotic tumor, but not helpful to distinguish evolution stage/cerebritis stage of abscess from cystic/necrotic tumor.

结论摘要:DWI及ADC值有助于鉴别包膜期脑脓肿和坏死囊变脑转移瘤,对吸收期脑脓肿和坏死囊变脑转移瘤的鉴别诊断无明显意义。

METHODS摘要: Fourteen patients proved pathologically as brain abscess and 17 cystic/necrotic metastatic tumor underwent conventional MRI and DWI examinations. DWI signal and ADC value were evaluated in cerebritis stage, capsule formation stage, and evolution stage of brain abscess and compared with those of cystic/necrotic metastatic tumor. Chisquared test and t test were performed for statistic analysis.

方法摘要:病理及临床证实脑脓肿14例,坏死囊变脑转移瘤17例,经常规磁共振检查及弥散加权检查,在脑脓肿脑炎期、包膜期及吸收期进行弥散信号及ADC值的测量,并和坏死囊变脑转移瘤相比较,统计学分析采用χ2检验和t检验。

METHODS: Fourteen patients proved pathologically as brain abscess and 17 cystic/necrotic metastatic tumor underwent conventional MRI and DWI examinations. DWI signal and ADC value were evaluated in cerebritis stage, capsule formation stage, and evolution stage of brain abscess and compared with those of cystic/necrotic metastatic tumor. Chisquared test and t test were performed for statistic analysis.

病理及临床证实脑脓肿14例,坏死囊变脑转移瘤17例,经常规磁共振检查及弥散加权检查,在脑脓肿脑炎期、包膜期及吸收期进行弥散信号及ADC值的测量,并与坏死囊变脑转移瘤相比较,统计学分析采用χ2检验和t检验。

METHODS: Fourteen patients proved pathologically as brain abscess and 17 cystic/necrotic metastatic tumor underwent conventional MRI and DWI examinations. DWI signal and ADC value were evaluated in cerebritis stage, capsule formation stage, and evolution stage of brain abscess and compared with those of cystic/necrotic metastatic tumor.

病理及临床证实脑脓肿14例,坏死囊变脑转移瘤17例,经常规磁共振检查及弥散加权检查,在脑脓肿脑炎期、包膜期及吸收期进行弥散信号及ADC值的测量,并与坏死囊变脑转移瘤相比较,统计学分析采用χ2检验和t检验。

Results According to CT findings,they were divided into edematous type (n=46) and necrotic type (n=42).Forteen cases were complicated with infection.Swelling in 75 cases,exudation in 68 cases,necrosis in 42 cases,splenomegaly in 62 cases.

结果 水肿型46例,坏死型42例,其中合并感染14例,均有较典型的CT表现,胰腺肿胀75例,胰周渗液68例,胰腺坏死42例,脾大62例。

Necrosis and apoptosis that gradually increase and eventually result in enervation and death of chicken with selenium-penia. The effect of selenium on the lipid peroxidation of the cellular membrane system of tissues is the main mechanism that selenium induces apoptosis of tissues.

雏鸡硒缺乏时,甲状腺、肝脏、胸部肌肉、胰腺、胸腺等组织细胞凋亡增加,证明硒缺乏雏鸡体内发生两种细胞死亡,即坏死和细胞凋亡,由于细胞坏死和凋亡的加重与增多,致使硒缺乏雏鸡走向衰竭,最后死亡。

Cut off the inactive skin, after 20 days changing of dressing, wound closed up.conclusion 2 of them esthesia decreased of the wounded finger.

结果 13例一期闭合伤口的患者中2例出现伤口部分皮肤坏死,清除坏死皮肤经十余天换药后伤口闭合。

We also have observed some organs pathological changes of juvenile Jian Carp, such as pancreas acinous gland and zymogen granules of acinous gland cell disappearing, intercellular substance hyperplasia and inflammatory cells soakage, hepatatrophia, liver cell granular or vacuolar degeneration and necrosis, karyolysis or pyknosis, glycogen granules decreasing, metanephros atrophy, metanephric canaliculus epithelium granular or vacuolar degeneration and necrosis, mitochondrion swelling and mitochondrion cristae disappearing, karyolysis, distal convolutal tubule microvilli desquamating, spleen marrow cell degeneration and necrosis, intercellular substance of spleen hyperplasia, spleen atresia, blood corpuscle disappearing.

后肾土黄色、淡褐色或苍白色,肾小管上皮细胞肿胀、颗粒或水泡变性、坏死,细胞内有大量血细胞流出,线粒体肿胀,嵴结构消失,细胞核溶解,肾间质甲状腺滤泡和拟淋巴细胞增生,远曲小管微绒毛脱落、管道细胞界限不清。心脏肌纤维肿胀、颗粒或空泡变性,严重的肌纤维溶解、变细或断裂,肌纤维间水肿、炎性细胞浸润,部分心肌细胞核浓缩。脾脏暗褐色,脾髓质细胞变性、坏死,拟淋巴细胞明显减少,淋巴细胞岛少见、岛中细胞成份减少,黑素巨噬细胞中心减少、体积缩小,脾脏网状基质水肿,脾窦闭锁,血细胞减少。

The pathogen of gas gangrene mainly exist and live in local place, and seldom enter blood system to cause blood poisoning, but than can form lots of toxins, this toxins can harm blood system, kidney system, and also the tissue can putrescence and the wound will enlarge step by step, all tissue putrescence and toxin can make these bacteria easier to live and reproduce, all these situation if not being control in time will lead a serious situation that multiple organs dysfunction and then died

气性坏疽的病原菌主要在伤口内生长繁殖很少侵入血液循环引起败血症。产气夹膜杆菌产生α毒素、胶原酶透明质酸酶、溶纤维酶和脱氧核糖核酸酶等,红细胞破坏引起溶血血红蛋白尿、尿少、肾组织坏死水肿、液化,肌肉大片坏死使病变迅速扩散、恶化。糖类分解产生大量气体使组织膨胀;蛋白质的分解和明胶的液化,产生硫化氢,使伤口发生恶臭由于局部缺血,血浆渗出,及各种毒素的作用伤口内的组织和肌肉,进一步坏死和腐化,更利于细菌的繁殖使病变更为恶化。

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