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Methods:9 patients including 8 male and 1 female were included in this study.2 patients suffered from both side of ANFH,5 were righe side and 2 were left side.X-ray,CT and MRI were performed before the surgery.4 hips were classified as ARCO stage 1,7 hips were stage 2.All the patients were treated as follows:the area of NFH and cystis degeneration were detected and bouche decompression was done through X-ray with C-arm,and the necrotic bone was cleared for the pathologic diagnosis under the monitoring of arthroscopy.And then the non-cell based tissue engineering bone, which was the complex of autologous red bone marrow and osteoinductice absorbing material containing BMP,was transplanted into the body.

本组9例,男性8例,女性1例;双侧股骨头坏死2例,右单侧股骨头坏死5例,左单侧股骨头坏死2例;术前均行X线摄片、CT、MRI 检查,ARCO分期:Ⅰ期4髋,Ⅱ期7髋;C型臂X线透视下行股骨头坏死区和囊性变区定位、髓芯减压,关节镜监视下,刮除坏死骨送病理;植入由自体红骨髓与含BMP的骨诱导活性材料复合成的非细胞型组织工程骨,并用空心钛钉支撑股骨头软骨下骨质。

Was presented in 12 cases (24%) by bacterial isolation. By in-situ hybridization, PCV2 signals were chiefly distributed in interstitial and necrotic lesions, and the positive signals could be found in macrophages-like cells and necrotic debris. PRRSV signals were mainly located at interstitial lesions and alveolar wall, and the positive signals could be found in macrophages-like cells and epithelial cells of alveoli. Swine influenza virus could be found in interstitial and necrotizing lesions, and the signals could be found in macrophages-like cells, and epithelial cells of terminal bronchiole and alveoli.

另运用原位杂交法对PCV2、PRRSV及猪流行性感冒病毒进行检测,结果发现PCV2主要分布於PNP的间质及坏死病灶区,并能於肺泡及终末细支气管上皮细胞、巨噬细胞与坏死细胞碎片皆可见有病毒核酸;PRRSV则多分布於间质病灶区及肺泡壁处,能於巨噬细胞与肺泡上皮细胞见到病毒核酸;SIV则存在於间质及坏死病灶区,且能於肺泡及终末细支气管上皮细胞与巨噬细胞见有病毒核酸。

Herein, we describe a patient who developed extensive necrotizing fasciitis of the abdominalwall after intestinal resection for necrotizing enterocolitis .

在此,作者报道1例因坏死性小肠结肠炎而接受肠切除的新生儿发生的腹壁广泛性坏死性筋膜炎病例。坏死性筋膜炎为新生儿坏死性小肠结肠

There were hemosiderin or multiphase intramedullary haem-orrhagein 52% of the femoral heads.

出血灶位于坏死区内或与坏死骨相间。坏死越严重,IH的发生率越高。

In treatment group, there was no progressive necrosis in stasis zone, and at 24 h post injury, capillary dilation, cell edema and inflammatory infiltration were lessened significantly. In control group, 2 wounds had progressive necrosis (accounted for 10% of the total). Cell edema, inflammatory infiltration and capillary thrombosis were serious, Until 72 hr post injury, cell edema did not subside and necrosis of the dermis worsened. 2. Pathological assessment of the stasis zone tissue: As compared with that in control group, in treatment group the dermis structure in stasis zone was intact and the collagenous fiber bundle was normal.

结果:①创面细胞形态学改变:伤后8h毛细血管扩张、细胞水肿,炎性浸润最明显;治疗组中央淤滞区无进行性坏死,伤后24h后毛细血管扩张、细胞水肿、炎性浸润减轻;对照组2个创面呈进行性坏死(占10%),中央淤滞区细胞水肿、炎性浸润明显,毛细血管血栓形成,伤后72h水肿仍明显,真皮坏死加重;②淤滞区组织病理学评分:治疗组与对照组相比,治疗侧淤滞区的表皮结构相对完整、胶原纤维束相对正常,粒细胞浸润小于5个/400倍视野。

Using acupotomology incise and separate the hip joint articular capsule and ligament and the periosteum on the collum ossis femoris and the soft tissue nearby the joint can cause slight hurt and haemorrhage. The hurt factor is release from the local tissue. Human self-rebuilding system is activated, Building bone function of osteoblast is accelerated, new bone is built quickly. New blood vessel and nerve ending is rebirth. Local tissue's blood supply and nerve nourishment is improved. The developing of bone necrosis is prevented. The necrotic part is re-blood vessel. The conditions of the necrotic's absorbing tissue and the new bone's replacing are established.

针刀治疗股骨头无菌性坏死是通过针刀对髋关节囊、关节囊韧带、股骨颈的骨膜和髋关节相邻周遍组织切割分离铲拨松解可以产生轻微创伤、出血、局部组织释放创伤因子,激发人体自我修复重建反应,激活成骨细胞造骨功能,加速新骨生成,促进新血管和神经终端组织再生与重建,使局部组织血流加速,改善血液供应和神经营养状况,阻止坏死进一步发展,使股骨头颈坏死区域再血管化,为坏死组织吸收和为新骨再生爬行替代过程创造条件。

Pneumatosis intestinalis and portomesenteric venous gas are alarming radiological findings that signify bowel ischemia. The management of PI and PMVG remain a challenging task because clinicians must balance the potential morbidity associated with unnecessary surgery with inevitable mortality if the necrotic bowel is not resected.

肠气囊肿病和门脉肠系静脉积气在影像诊断学上是缺血性肠道疾病非常重要的表现,当病人的影像出现肠气囊肿病或是门脉肠系静脉积气时,临床医师要如何作最适当的处置,依然是一项富挑战性任务,因为如果坏死的肠组织没有被切除,无可避免的会造成病人死亡,但是多余的手术亦会有潜在的并发症。

There were no statistically differences between the 8th~ 14thday, the 15th - 21th day post-BC and the control group. During the rt~24tkday post-BC , pycnosis degeneration or necrosis neurons of the locus cerebral cortex, dorsal hippocampus, dentate fornix were significantly increased, then decreased gradually, but recovered to normal by the 24thday after BC, and especially in parietal cortex and piriform cortex the necrosis neurons were significantly increased than temporal cortex , and there were no statistically difference berween the left and the right side. Pycnosis degeneration or necrosis neurons in the brainstem reticular formation were markedly increased in the 4thday after BC, and there were no statistically difference among the other groups and the control group.

与对照组相比,BC后在大脑皮质、背侧海马和齿状回部位,固缩变性和不完全坏死细胞数先显著增加(P<0.05),然后逐渐减少,至24d基本恢复正常;顶叶、梨状皮质比颞叶皮质变性坏死细胞多,差异有高度显著性(P<0.01);大脑左侧比右侧变性坏死细胞稍少但差异无显著性;在BC后4d,脑桥核、斜方体核平面的脑干网状结构中固缩变性和不完全坏死细胞数明显增多(P<0.05),其它组间无显著性差异。

In the clinical use of the pre-expanded axial-pattern flap, we noticed that partial necrosis of transferred flap might develop following over pre-expansion.

在预扩张轴型皮瓣的临床应用中我们观察到大量预扩张的轴型皮瓣转移后可能出现皮瓣局部坏死的现象,但目前这方面的研究还比较局限,缺少对轴型皮瓣扩张的安全限度的了解和对皮瓣血液循环立体结构影响的观察。

The cancer therapy and composition for using the same of the present invention involves administering a therapeutically effective and non-lethal amount of a pharmaceutical composition to mammals in need of such a therapy and more specifically humans suffering from cancerous neoplasms and to prevent the formation and elimination of the hostile cancer viability zone and more specifically the tumor micro-environment which can help treat metastatic tumors and at sites other than a primary tumor site with site specificity for malignant cells that is not restricted by the type of cancer cell, including damaged or necrotized cells and tissues.

使用本发明制剂的抗癌疗法和制剂可以在需要此类治疗的哺乳动物,更确切一点,受癌瘤折磨的人身上,使用疗效显著、剂量不致命的医药制剂。可以组织敌对癌瘤生存区的形成,并加以消除,更确切一点,消除癌瘤微环境,从而有助于治疗病毒转移性癌瘤;消除当场有针对性地治疗不受癌细胞类型限制的恶性细胞,如受损或坏死的细胞和组织。

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