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The overexpression of HO-1 induced by CoPP can inhibit death of the myocardial cells and subsequently alleviate myocardial ischemia/reperfusion injury,whose major mechanism may be related with anti-oxygen free radicals.

CoPP诱导的HO-1过表达可以抑制心肌缺血再灌注损伤后的细胞坏死,从而减轻心肌的再灌注损伤,其主要机制与抗氧自由基有关。

The upregulation of HO1 expression induced by CoPP can inhibit the necrosis of the myocardial cells and subsequently alleviate the myocardial ischemia/reperfusion injury. One of its main mechanisms may be related with antioxygen free radicals.

CoPP诱导的HO1过表达可以抑制心肌缺血-再灌注损伤后的细胞坏死,从而减轻心肌的再灌注损伤,其主要机制与抗氧自由基有关。

Result HO-1 expression was upregulated following pretreatment of CoPP before reperfusion,resulting in both an increase in SOD activity and a decrease in MDA content and thus in a reduction in death of the myocardial cells after ischemia/reperfusion injury.

结果:再灌注前使用CoPP进行预处理,可以诱导HO-1蛋白的表达上调;HO-1蛋白表达上调可以减少缺血再灌注后的心肌细胞坏死,提高心肌组织中SOD含量并降低MDA的含量。

The garlic in HeiLongJiang was widely infected by complex viruses,through investigating.Natural infected garlic crops exhibited diversiform symptoms such as masaic、crimple、leaf roll、abnormality、etiolation、putrescence and so on.

通过调查发现黑龙江省大蒜普遍受到病毒的复合侵染,侵染后的田间症状多样,主要有花叶、皱缩、卷叶、畸形、黄化、坏死和矮化等。

If the toxic effects of viral gene therapy can be overcome, Melton's work may provide a basis for treating certain types of diabetes, in which cells in the pancreas that produce insulin die and are not replaced.

如果能使病毒基因疗法不产生毒性效果,那么梅尔顿从事的工作可为治疗因分泌胰岛素的胰腺细胞坏死后没有得到替代的糖尿病打下基础。

Necrosis was observed in some of the 30 Gy-3-month-period group. Different levels of morphological changes in blood vessels,neuroglia cells and neurons were found in rats of all other groups.

在30 Gy照射后3个月组中有坏死灶的存在,而其他组别中可以看到血管、胶质细胞和神经元等组织的形态学异常。

The symptoms such as cough, sputum and stridor etc of both groups who were treated by point application for two weeks were observed. the serum levels of il 2, tnf α both of the admission of hospital and after being treated by elisa method were measured in these cases.

采用酶联免疫吸附方法测定70例慢阻肺急性发作期患者入院初及治疗后血清中的白细胞介素 2(il 2)、肿瘤坏死因子α的水平,同时测定肺功能(fev 1%pre、fev1/fvc、mvv。

To explore the change and its significance of the expression of vascular endothelial growth factor messenger RNA and bone morphogenic protein-2(BMP-2) of the local femoral head in the nontraumatic osteonecrosis of femoral head.

探讨非创伤性股骨头发生坏死后局部的血管内皮生长因子(vascular endothelial growth factor VEGF)mRNA与骨形态发生蛋白-2(bone morphogenic proteins,BMP-2)表达的变化及其意义。

An animal model of DM was established byinjecting a little dosage ofSTZ in high fat-fed rats. When the model was establishedsuccessfully, corresponding medicine were supplied for 12 weeks and then the ratsweight、biochemical criterions, blood glucose, GSP and the content of NO, ET, hs-CRP,AR, IL-6, TNF-a in rats serum were mensurated.

给药12周后,观察冬连胶囊对大鼠生长状态、体重、生化指标、血糖、糖化血清蛋白、血浆一氧化氮、内皮素、血清高敏C—反应蛋白、醛糖还原酶、白细胞介素—6(IL-6)、肿瘤坏死因子的影响,及大鼠视网膜微血管形态学、病理改变。

These animals were killed after 24 hours of treatments and instillation of meconiums. The measurements included lung tissue wet/dry ratio,lung injury score,the expression and level of AngⅡ,lung myeoperoxidase activity and TNF-α level.

模型制备后24h取材,观察其肺组织病理损伤并检测湿/干重比及进行病理评分;检测AngⅡ在肺组织的表达情况及用图像分析方法检测其含量;测定肺组织中肿瘤坏死因子-α的含量及肺匀浆髓过氧化物酶活性。

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