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Background: The neuron death resulting from traumatic brain injury can be classified into 3 types:(1)neuron death due to primary injury;(2)neuron necrosis due to secondary injury such as hemorrhage, compression, anoxemia and ischemia;(3)neuron apoptosis due to inflammatory factor, cytokine,, excitatory transmitter, Ca~(2+) overload and oxygen radical et al.

引言创伤性脑损伤所致的神经元死亡主要有三种:①原发性损伤所致的神经元直接死亡;②外伤后出血、压迫、缺血缺氧等继发性损伤因素所引起的神经元坏死;③炎性介质、细胞因子、兴奋性递质、Ca~(2+)超载、氧自由基等因素所诱发的神经元凋亡。

Antiphlogistic Liquid can effectively prevent the the gangrene and the local tissue of the ischemia limb from seconddory infecton, and realeasethe ache of the affected limbs.

消炎液可以有效的防止坏疽肢端和缺血肢体的局部组织坏死液化后继发感染,在一定程度上缓解肢体疼痛;并促进患肢侧支循环的建立,从而减少截肢的范围。

In a way, the Antiphlogistic Liquid can release the ache of the attected limb and prompt the establishement of the compensatory circulation. As a reault the Antiphlogistic Liquid can diminish the extent of amputated extremity

消炎液可以有效的防止坏疽肢端和缺血肢体的局部组织坏死液化后继发感染,在一定程度上缓解肢体疼痛;并促进患肢侧支循环的建立,从而减少截肢的范围。

Bacteria on burn wound and alive tissue under burn wound of the 119 patients that were treated with MEBT was generally cultured and identified, when these patients were first debrided ,7 days later and before the wound healing.30 patients randomly selected from 119 patients were enrolled in the study ,bacterial quantity and identification in burn wound tissue were carried out, bacterial epidemiology data in burn ward were analyzed in the same period.

对应用MEBT治疗的119例烧伤患者的创面于首次清创时、入院后一周、创面愈合之前作常规细菌学培养、鉴定,同时抽取其中30例进行烧伤创面坏死组织及其下活组织细菌定量计数及菌种鉴定,并与同期烧伤病房环境的细菌流行病学资料进行了相关分析。

MEBT.. Method Bacteria on burn wound and alive tissue under burn wound of the 119 patients that were treated with MEBT was generally cultured and identified, when these patients were first debrided ,7 days later and before the wound healing.30 patients? randomly selected from 119 patients were enrolled in the study ,bacterial quantity and identification in burn wound tissue were carried out, bacterial epidemiology data in burn ward were analyzed in the same period.

对应用MEBT治疗的119例烧伤患者的创面于首次清创时、入院后一周、创面愈合之前作常规细菌学培养、鉴定,同时抽取其中30例进行烧伤创面坏死组织及其下活组织细菌定量计数及菌种鉴定,并与同期烧伤病房环境的细菌流行病学资料进行了相关分析。

Most important, macrophage apoptosis was decreased by 61%(P=0.034) and plaque necrosis by 34%(P=0.02) in the plaques of fat-fed low density lipoprotein receptor null Ldlr-/- mice transplanted with Stat1-/- bone marrow.

最重要的是,在脂餐喂养的、LDL受体无效的Ldlr-/-小鼠中,接受Stat1-/-骨髓移植后巨噬细胞凋亡下降61%(P=0.034),斑块中坏死减少34%(P=0.02)。

Results: The animal experimental model indicates the facial nerve' s bone canal fracture can make the facial nerve hyperaemia, strut, and putrescence, denaturalization and with the Schwann cell and the cell become more and more, taking on the restore evidence.

结果 动物实验表明:面神经骨管骨折后,面神经发生充血肿胀,并出现脱髓鞘、变性及坏死的变化,伴随神经损伤的同时出现巨噬细胞和施万细胞增生肥大及纤维母细胞的增生,呈现修复迹象。

METHODS Dinitrofluorobenzene was used to induce the contact dermatitis in mouse ear, rhIL-11 0.375-1.5 mgkg^(-1)d^(-1) sc for 10 d, the degree of skin inflammatory reaction was observed. At the same time, the serum level of tumor necrosis factor-α and interleukin-6(IL-6) was measured by radioimmunoassay; the serum level of nitrogen monoxidum was detected by biochemical method and the expression of intercellular adhesion molecule-1(ICAM-1) was detected by immunohistochemical method.

采用2,4-二硝基氟苯致小鼠耳部皮肤接触性皮炎试验模型,观察sc rhIL-11 0.375~1.5mgkg^(-1)d^(-1),连续10d后皮肤炎症反应程度,同时采用放射免疫分析法测定血清中肿瘤坏死因子-α和白细胞介素-6(IL-6)的水平,采用生物化学检测法测定血清一氧化氮的水平,采用免疫组化法测定皮肤中细胞间粘附分子(ICAM-1)的表达。

Our understanding of the pathogenesis of CP has improved in recent years, though important advances that have been made with respect to delineating the mechanisms responsible for the development of pancreatic fibrosis following repeated acute attacks of pancreatic necroinflammation (the necrosis-fibrosis concept).

虽然慢性胰腺炎重要的进展在于阐述反复急性胰腺坏死性炎症后胰囊性纤维化的发生机制方面,但最近几年我们对于慢性胰腺炎发病机理的理解已有所更新。

Our understanding of the pathogenesis of CP has improed in recent years, though important adances that hae been made with respect to delineating the mechanisms responsible for the deelopment of pancreatic fibrosis following repeated acute attacks of pancreatic necroinflammation (the necrosis-fibrosis concept).

虽然慢性胰腺炎重要的进展在于阐述反复急性胰腺坏死性炎症后胰囊性纤维化的发生机制方面,但最近几年我们对于慢性胰腺炎发病机理的理解已有所更新。

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