嗜酸粒细胞

The test results of 20 blood samples detected on different hematological analyzers were analyzed by a q test which indicated that there were no statistical differences between blocks（P＞0.05）. The correlative coefficient was over 0.97 and the relative deviation was within the range of allowable error.

白细胞分类中，中性粒细胞和淋巴细胞在各比对仪器之间以及仪器与手工分类之间的相关性较好，而嗜酸粒细胞、嗜碱粒细胞和单核细胞，不论在各仪器还是仪器与手工分类之间，都存在较大的试验误差（P＜0.05，P＜0.01）。

The atopy asthma was correlated with the number of the eosinophil while the non-atopy with the neutrophil.

特应性哮喘的发病机制与诱导痰中嗜酸粒细胞数相关；非特应性哮喘与中性粒细胞数相关。

Results: 1 The capacity of engulfment of A549 cells started enhancing at the 1h of preincubation with dexamethasone, reached the highest at the 4h of preincubation,At the 8h、24h of preincubation the capacity of engulfment evidently reduced. 2 Glucocorticoid cytosolic receptors antagonist RU38486 inhibited the improved engulfment ability by dexamethasone. 3Phagocytosing apoptotic eosinophils did not change the release of IL-6 and IL-8,but can inhibited the release of IL-6 and IL-8 from A549 cells stimulated by lipopolysaccharide. 4Increasely uptaking by dexamethasone also did not agitate the release of IL-6 and IL-8 from A549 cells. Conclusion: 1Dexamethasome improve the phagocyte capacity for apoptotic eosinophils of A549 cells by glucocorticoid cytosolic receptors, and the effect presents time-dependently. 2Phagocytosis of apoptotic eosinphils can degrade inflammatory reflection, so it is likely to be critical in the control of asthma inflammation.

结果： 1 A549细胞在地塞米松诱导后1小时吞噬能力开始升高，4小时吞噬能力达最高峰，8小时、24小时吞噬能力明显下降 2加入RU38486后抑制了地塞米松所增强的吞噬能力 3凋亡嗜酸粒细胞的吞噬并不能改变A549细胞释放IL-6和IL-8，但能抑制脂多糖刺激A549细胞后IL-6、IL-8的分泌 4地塞米松增加的吞噬也不激发A549细胞释放IL-6和IL-8 结论： 1地塞米松促进A549细胞吞噬凋亡的嗜酸粒细胞是通过A549细胞糖皮质激素胞浆受体发挥作用的，并且具有时间依赖性 2 A549细胞吞噬凋亡的嗜酸粒细胞能降低炎症反应，对哮喘炎症的控制具有重要意义

Idiopathic hypereosinophilic syndrome involves a prolonged state of eosinophilia associated with organ dysfunction .

特发性高嗜酸粒细胞综合征涉及一种与器官功能障碍相关的长期高嗜酸粒细胞增多状态。

MethodsRandomly clinical observation method was adopted,the treatment group(n=152)were treated with Kechuanning,and the control group(n=146)were treated with Guilongkechuanning capsule;The symptom score,the count of eosinophils,leukomonocyte and neutrophile granulocyte,and clinical therapeutic effect were observed.

方法采用随机临床对照观察方法，咳喘宁治疗152例为治疗组，以桂龙咳喘宁胶囊治疗146例为对照组，观察两组患者治疗前后症状评分，嗜酸粒细胞、淋巴细胞、中性粒细胞计数的变化，并比较两组的临床疗效。

Eotaxin, a kind of chemokines of eosinophil found in recent years, is a kind of chemotactic agent of eosinophil.

近年发现的嗜酸粒细胞趋化因子，即Eotaxin基因，是一种嗜酸粒细胞选择性的趋化剂。

A fusion gene is reported to be responsible for the hypereosinophilia in myeloproliferative subtype of HES.

骨髓异常增殖型嗜酸细胞增多综合征因出现融合基因导致嗜酸粒细胞的增多。

Or white blood cell; a colorless blood corpuscle capable of ameboid movement, whose chief function is to protect the body against microorganisms causing disease and which may be classified in two main groups: granular (granulocytes: basophils, eosinophils, neutrophils) and nongranular(nongranulocytes: lymphocytes, monocytes).

或称白血球，为一能作变形运动的无色血细胞，其主要作用是保护机体抗御致病菌。它可以分为两大类：粒性（粒性白细胞：嗜碱粒细胞、嗜酸粒细胞、嗜中性粒细胞）和非粒性（非粒性白细胞：淋巴细胞、单核细胞）。

Eosinophils play a very important role in the pathogenesis of hypereosinophilic dermatoses.

嗜酸粒细胞在嗜酸粒细胞增多性皮肤病发病中发挥重要的作用。

Except for monocyte and basophil,the precision and accuracy of sysmex SF-3000 blood analyzer corresponds to microscope classification.

无异常提示，除单核细胞、嗜碱粒细胞外，中性粒细胞、淋巴细胞及嗜酸粒细胞，用仪器分类的结果相当于目测分类。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。