呼吸中枢

This is because it is not only the lifting of bronchospasm, but also so that the contraction of smooth muscle increased induced asthma attack and suppress the respiratory center, so that even more difficult to breathe, leading to respiratory failure.

这是因为它不但能解除支气管痉挛，还可使平滑肌收缩加剧，诱发哮喘的发作，且抑制呼吸中枢，使呼吸更加困难，导致呼吸衰竭。

The blood pressure of.1 of pathology physiology mechanism that SAS causes CVD breathes time-out to be able to bring about airframe in elevatory Morpheus anoxic, sympathetic is excessive and excited, element of serous catechu phenolic amine, kidney and hemal endodermis element are elevatory, bring about hemal easy of hemal convulsion; to shrink the function is disorder, can make blood-vessel flowing flesh happening reframes and fleshy, systemic blood-vessel obstruction adds element of the kidney when; is chronic and anoxic, system of hemal and nervous element is activationed, bring about blood pressure to lift. Obstruction of way of energy of life of patient of OSAS of disease of blood of 1.2 low oxygen increases, cause air current to interrupt, breath pauses, at the same time airframe gets used to low oxygen environment gradually, breathing centre drops to low oxygen and sensitivity of disease of blood of tall carbonic acid, breath suspends a frequency increasing, farther aggravating airframe is anoxic. 1.3 heads are self-adjusting the function drops normal person changes quickly in systematic circulation blood pressure when, the head can be passed adjust independently functional generation protects effect, make change of cerebral blood flow not big.

SAS诱发CVD的病理生理机制。1血压升高睡眠中呼吸暂停可导致机体缺氧，交感神经过度兴奋，血浆儿茶酚胺、肾素以及血管内皮素升高，导致血管痉挛；血管舒缩功能紊乱，可使血管平滑肌发生重构和肥厚，全身血管阻力增加；慢性缺氧时肾素—血管紧张素系统被激活，导致血压升高。1.2低氧血症OSAS患者气道阻力增加，造成气流中断、呼吸暂停，同时机体逐渐适应低氧环境，呼吸中枢对低氧和高碳酸血症敏感性下降，呼吸暂停次数增加，进一步加重机体缺氧。1.3脑自动调节功能下降正常人在体循环血压快速变化时，脑可通过自主调节功能产生保护效应，使脑血流量变化不大。

The results indicated that LVPO might play a role in respiratory responses to CO〓, and also suggested that LVPO might participate in the mechanism of central chemoreception.

实验表明损毁LVPO后能明显地减弱呼吸中枢对CO〓的反应性，提示LVPO在吸入CO〓所引起的兴奋反应中起着一定的作用，为LVPO参与中枢化学感受性机制提供了另一佐证

Tetrodotoxin selectively blocks the neuromuscular electrical activity, producing the effect of nondepolarizing curine and leading to failure of the respiratory drive without compromising the respiratory center.

河豚毒选择阻断神经与肌肉的电生理活动，呈非去极化型箭毒碱样作用，致呼吸动力障碍，与呼吸中枢无关，M胆碱能受体阻滞剂阿托品及中枢兴备剂均无明确使用指征，维持有效通气等综合治疗可达到满意疗效。

Asarum can boost ion concentration of potassium besides cell, leadwithering of breath and correlate breath neuron cell and palsying breathsystem.

4细辛对呼吸中枢的影响可能是由于升高了细胞外钾离子浓度，诱导呼吸相关神经元的凋亡，从而导致呼吸麻痹而死亡。

Respiratory center is composed of several groups of nerve cells which produce and regulate respiratory movement in central nervous svstem.

呼吸中枢是指在中枢神经系统里产生和调节呼吸运动的神经细胞群。

Central mechanism includes the direct influences on cardiovascular control center from respiratory center and the feedback effects from stretch receptors in the lung.

中枢机制主要是呼吸中枢对心血管控制中枢的直接影响以及肺牵张受器的回馈效应。

Low-dose intramuscular injection (5 ~ 10mg) of morphine can suppress mild respiratory center, so that by the rapid and shallow breathing slowly and deeply to ease breathing problems in patients with Deng Zheng.

肌注小剂量（5～10mg）吗啡，可以轻度抑制呼吸中枢，使呼吸由快而浅变为慢而深，缓解患者呼吸困难等症。

Birth canal head pressure congestive childbirth, can stimulate the respiratory center, easy to stimulate fetal breathing after birth and crying, and less occurrence of "respiratory distress syndrome."

产道分娩时胎头受压充血，能刺激呼吸中枢，易激发出生后胎儿呼吸和啼哭，较少发生&呼吸窘迫综合征&。

Thus, the membrane of the neurons in the respiratory center and CRCs may own TASK-1, which can sense the change of H+ or CO2 of the extracellular fluid and enhance the output of the respiration by increasing the excitability of the neurons.

因此，我们推测TASK-1可能存在于呼吸中枢和/或中枢化学感受器，当细胞外H+或CO2增加时，可通过作用于该通道而增加呼吸相关神经元的兴奋性，使呼吸活动增强。

According to the clear water experiment, aeration performance of the new equipment is good with high total oxygen transfer coefficient and oxygen utilization ratio.

曝气设备的动力效率在叶轮转速为120rpm～150rpm时取得最大值，此时氧利用率和充氧能力也具有较高值。

The environmental stability of that world - including its crushing pressures and icy darkness - means that some of its most famous inhabitants have survived for eons as evolutionary throwbacks, their bodies undergoing little change.

稳定的海底环境─包括能把人压扁的压力和冰冷的黑暗─意谓海底某些最知名的栖居生物已以演化返祖的样态活了万世，形体几无变化。