后脑

Three hours following seizures there was an evident expression of c-fos proteins in the hippocampus (CA1 area), dentate gyrus, piriform cortex, dorsal part of entorhinal cortex, and amygdaloid nucleus, and there was a dramatic increase of c-fos proteins in CA3 area and the areas mentioned above except the CA1 area where c-fos proteins apparently decreased after electroacupuncture treatment.

在海马青霉素致痫模型上观察了致痫及致痫加电针后脑内c-fos蛋白的变化，结果表明致痫时海马〓区、齿状回、梨状皮层、背侧内嗅皮层、杏仁核见到较多的c-fos表达；电针后c-fos在〓区明显减少，在〓区及上述其余各区则显著增加。

The patients have used their full dentures for 1—3 months .(2) With the use of transcranial color Doppler analyzer the blood flow in the brain was measured before and 10 minutes after mastication in 17 subjects .

应用经颅彩色多谱勒超声扫描仪(Transcranial Color Doppler ，TCCD)探测牙列完整受试者及无牙颌患者在安静状态下和咀嚼无糖口香糖10分钟后脑血流速的改变，对大脑中动脉收缩期峰流速(systolic peak flow velocity ，Vs)、舒张期末峰流速(enddiastolic peak flow velocity ，Vd)、平均峰流速(mean peak flow velocity ，Vm)进行统计分析。

The maxillofacial and brain injuries were examined. Craniptomy was done to animals of impact injury 6 hours after damage, however it was done to animals of explosive injury in different time point (1 hour, 24 hours, 72 hours, 7 days, 14 days after damage). The pathological characteristics of corelative tissues were examined in detail under light and electron microscopes. The content of LPO, SOD, NO in soft tissue, brain, blood, and cerebral water content were measured, compared with control. The distribution and tissue location of NOS were studied by histochemistical and immunohistochemistic methods. Their relationship was studied to assess the pathological and physiological characteristics of maxillofacial injuries and brain damage associated.

撞击伤各组动物于伤后6h、爆炸伤各组动物于伤后不同时期（1h、24h、72h、7d、14d）开颅检查颅脑损伤情况，切取伤区肌组织、左侧脑组织、脑干标本行光镜、电镜观察；测定伤后肌组织、脑组织与血中LPO、SOD、NO含量及脑水含量变化；并使用组织化学和免疫组织化学的方法，观察伤后脑组织中NOS的分布和组织定位，研究伤后机体的病理及病理生理学改变特点，以及颅脑损伤与机体自由基反应、NO含量改变之间的关系，证实其病理作用。

Much progress has been made in recent years through studying septic shock,however brain injury is rarely involved.This study takes the models of endotoxin-shock rats as research object to simulate septic shock.The study aims are to explore anomalous signal transduction in brain injury caused by septic shock and the effect of Sini Decoction in the process;define the molecular and cellular mechanisms of reviving yang for resuscitation method used to prevent and treat brain injury in septic shock and establish the scientific foundation for modernizing traditional Chinese medicine.

研究目的：1。明确内毒素休克后脑损伤组织中炎症介质水平，中性粒细胞和脑血管内皮细胞等细胞表面黏附分子表达情况，脑血管平滑肌细胞、神经胶质细胞和神经细胞对细胞外炎症介质刺激产生的功能代谢变化以及结构改变等三者之间的规律性关系。2。

Hydrocephalus after severe brain injury is one of the important causations results in disability,and right diagnosis and therapy can improve its prognosis.

重型颅脑外伤后脑积水是造成颅脑外伤后患者致残的重要原因之，正确诊断及治疗可改善预后。

Objective To explore effects of the iron chelator on brain edema after experimental intracerebral hemorrhage in rats.

目的：探讨铁螯合剂对大鼠脑出血(intracerebralhemorrhage，ICH)后脑水肿的治疗作用。

Objective To study the mechanisms of brain edema formation following intracerebral hemorrhage and therapeutic effects of iron chelators.

目的 探讨脑出血后脑水肿的发生机制及铁络合剂的作用。

Draco starting watching Kokurou sucking his cock, because the back of his head was facing him and he didn't know Draco was awake.

Draco 开始看著克狼吸他的屌。因为克狼的后脑正对著他，所以克狼不知道 Draco 已经醒了。

Objective To observe the effect of ischemic preconditioning on mitochondrial function after cerebral ischemic reperfusion in gerbils.

目的 观察缺血预处理对沙土鼠脑缺血再灌注后脑组织线粒体功能的影响。

Objective To investigate the expression of heat shock protein 27 (HSP27) and its possible role following cerebral ischemia-reperfusion in gerbils anterior cortex.

目的 研究沙鼠脑缺血再灌注后前脑皮质内热休克蛋白27(HSP27)的表达变化并探讨其在脑缺血再灌注后脑组织中可能的作用。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。