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Results The exoression of MMP-9 was significantly increased in hyperglycemic groups after irrigating 24h,48h post 2h ischemia.

高血糖可诱导脑缺血再灌注后脑组织内的MMP-9 表达,增加血脑屏障的通透性,从而加重脑组织的损伤。

SFO stimulation induced stable and significant drinking behavior and Fos protein expression in 8 areas of the forebrain (organum vasculosum of the lamina terminalis, median preoptic nucleus, paraventricular nucleus, supraoptic nucleus, lateral hypothalamic area, perifornical dorsal area, substantia innominata and thalamic reuniens nucleus), and in 3 areas of the hindbrain (area postrema, solitary tract nucleus and lateral parabrachial nucleus).

结果显示,刺激SFO能诱发明显的饮水行为,与此同时,前脑8个部位(终板血管器官、正中视前核、室旁核、视上核、下丘脑外侧区、穹窿周核背侧区、丘脑联合核和无名质)和后脑3个部位(最后区、孤束核和臂旁外侧核)的Fos蛋白表达明显增强。

Objective To study c-fos expression and apoptosis after traumatic brain injury in rats, and to explore the role of apoptosis in the secondary brain injury ,and the relation between c-fos expression and neuroglial apoptosis.

目的研究创伤性脑损伤后脑组织c-fos表达及神经细胞凋亡,探讨神经细胞凋亡在脑继发性损害中的作用,及c-fos基因表达与凋亡的关系。

Objective To study cfos expression and apoptosis after traumatic brain injury in rats,and to explore the role of apoptosis in the secondary brain injury,and the relation between cfos expression and neuroglial apoptosis.

目的 研究创伤性脑损伤后脑组织cfos表达及神经细胞凋亡,探讨神经细胞凋亡在脑继发性损害中的作用,及cfos基因表达与凋亡的关系。

Objective To study c fos expression and apoptosis after traumatic brain injury in rats,and to explore the role of apoptosis in the secondary brain injury,and the relation between c fos expression and neuroglial apoptosis.

目的 研究创伤性脑损伤后脑组织c fos表达及神经细胞凋亡,探讨神经细胞凋亡在脑继发性损害中的作用,及c fos基因表达与凋亡的关系。

objective To study cfos expression and apoptosis after traumatic brain injury in ratsand to explore the role of apoptosis in the secondary brain injuryand the relation between cfos expression and neuroglial apoptosis.

目的 研究创伤性脑损伤后脑组织cfos表达及神经细胞凋亡,探讨神经细胞凋亡在脑继发性损害中的作用,及cfos基因表达与凋亡的关系。

FDP intervention decreases morphologic changes of mitochondria after HIBD, and may improve neuronic energy metabolism.

FDP干预治疗减轻了HIBD后脑神经细胞线粒体的形态变化并可能改善脑神经细胞的能量代谢。

After the attack of cerebral ischemia, the expression of BDNF and Sema 3A may be related to the mechanism of neuronic injury and repairment. Puerarin may have protective effects on ischemic neurons

葛根素治疗后脑源性神经营养因子的表达增加,Sema 3A的表达下降,提示葛根素对脑缺血损伤具有保护作用。

The tritocerebrum neuropile regions of the three ant castes hasn't distinct limit with the protocerebrum and the suboesophageal ganglion, appears 5-HT immunoreactivity.

后脑的髓质区同前脑和咽下神经节没有明显界限,呈现5-HT阳性反应。

Background and Purpose— Vascular endothelial growth factor and VEGF receptors play important roles during neurovascular repair after stroke.

血管内皮生长因子和血管内皮生长因子受体在卒中后脑血管修复中有重要功能。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。