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All 7 cases were characterized by deposition of dystrophic or psammatous calcifications accompanied with chronic inflammatory cells infiltration in the stroma. In some cases, there was formation of germinal centers.

特徵性形态学表现为在胶原化的纤维组织间可见散在的钙化灶或砂砾小体,间质内伴有多少不等的淋巴细胞和浆细胞浸润灶,部分病例中可见生发中心形成。

Results①The formation of posterior capsule opacification in rabbit eyes: observation in different stages after the operation showed the inflammatory response remarkable at the first three days with iris congestion; edge of anterior capsule circularity opacification at the 1st week, but no remarkable change in the posterior capsule; just gauze-like at the 2nd week; strip-like opacification at the end of the 1st month, funicular opacification after 2~3 months, while visible opacification at the 5th month after the operation.

结果:①兔眼后发性白内障形成情况:术后不同时期裂隙灯下观察可见术后前3d前房内炎症反应明显,虹膜充血;术后1周可见前囊截缘出现白色环状混浊,但后囊尚透明;术后2周后囊呈薄纱状;1个月出现线条状混浊,2~3个月成条索状混浊,术后5个月后囊明显混浊。

The re was no significant difference between the contents of SP and VIP of lung tissue in experiment group and those in control group.4. Ultrastructurally, after infected with IBV, lots of grume were observed on the surface of trachea tissue and the cilia of the trachea were massed.

免疫电镜观察结果显示,大多数肾小管上皮细胞胞浆内均见有SP及VIP阳性颗粒,在上皮细胞间还可见有DNES细胞;肺脏中异嗜性白细胞颗粒也常呈SP及VIP阳性反应,肺脏内也可见DNES细胞。

Results In the medium and high dose F0 groups, it was observed that the atrophy and incrassation of seminiferous tubule, decrease of spermatogenesis, hyperplasia of interstitial tissue, especially in high dose groups spermatozoon abnormality and nucleolus concentration in the rats testis after DU ingestion for 14 months. The changes became more severe with the prolongation of DU ingestion. Such changes occurred in filial rats (F1) after DC ingestion for 5 months. In the medium and high dose F0 groups, it was observed that a little atrophy of kidney glomerulus, hyperplasia of interstitial tissue after DC ingestion for 14 months, and kidney glomerulus fibrosis happened after DC ingestion for 20 months, such changes occurred in filial rats (F1) after DC ingestion for 5 months In the medium and high dose F0 groups, splenic germinal center and periarterial lymphatic sheaths were hyperplasia , companies with lymphopoiesis after DC ingestion for 7 months, splenic white pulp became more small and sparse after DC ingestion for 20 months.

结果 F0代的中、高剂量组大鼠摄入贫铀14个月后可见雄性的精曲小管萎缩,管壁增厚呈空虚网状,生精细胞层次减少,间质细胞增生,但仍见有精子生成;高剂量组可见到精子呈异型性改变,细胞核浓缩深染,且随着摄入时间延长改变愈趋明显;F1代大鼠摄入贫铀5个月后就有上述改变且更为严重。F0代中、高剂量组大鼠摄入贫铀14个月后肾小球轻度萎缩,间质增生明显,20个月时肾小球萎缩纤维化;F1代大鼠摄入贫铀5个月后就有上述改变。F0代中、高剂量组摄入贫铀7个月时脾脏生发中心和淋巴鞘增生,淋巴母细胞增生活跃,20个月时脾小体减少,生发中心稀疏;F1代大鼠摄入贫铀早期和晚期有类似改变。F0和F1代高剂量组摄入贫铀早期肝脏有炎症细胞浸润,晚期骨髓有核细胞减少,脂肪细胞增加。

Results there were unconsciousness, respiratory rhythm disturbance and hyporeflexia of pupil light reflex immediately after injury, and reactiveness decrease and activity retardation still existed even after resuming consciousness. at anatomical scene, subarachnoid hemorrhage or cerebroventricular haemorrhage were widespread. at an early stage, there were swelling, collapse, and axonal retraction ball formation at cortico-medulla junction, callosum, brainstem, and cerebellar white matter under microscope. but at the later stage, gitter cell proliferation and nest-like aggregation were major pathophysiological changes at focal brain tissue.

结果 伤后大鼠均即刻出现原发昏迷,其中2只于损伤后20 min内死亡,余持续时间1-30 min不等;伤后大鼠呼吸节律紊乱,瞳孔对光反射减弱或消失,醒后均有程度不等的反应性下降,肢体活动迟缓;肉眼可见广泛蛛网膜下腔出血或脑室出血;光镜下可见皮髓交界区、胼胝体区、脑干、小脑白质等部位的神经轴索有不同程度的肿胀、断裂、轴索球形成,后期有小胶质细胞增生,局部呈巢样聚集。

Routine CT and MR examinations can only detect infracortical cerebral arteriosclerosis, whereas CT perfusion mapping images can detect evident slowed down cerebral focal blood flow Pictures 3, 4, 7, 8

常规CT和MR检查仅可见皮层下动脉硬化性脑病,CT灌注图像可见明显的脑局部血流缓慢(图3,4,7,8)。

Routine CT and MR examinations can only detect infracortical cerebral arteriosclerosis, whereas CT perfusion mapping images can detect evident slowed down cerebral focal blood flow

常规CT和MR检查仅可见皮层下动脉硬化性脑病,CT灌注图像可见明显的脑局部血流缓慢。

Routine CT and MR examinations can only detect infracortical cerebral arteriosclerosis, whereas CT perfusion mapping image s can detect evident slowed down cerebral focal blood flow

常规CT和MR检查仅可见皮层下动脉硬化性脑病,CT灌注图像可见明显的脑局部血流缓慢。

Routine CT and MR examinations can only detect infracortical cerebral arteriosclerosis, whereas CT perfusion mapping images can detect evident slow ed down cerebral focal blood flow

常规CT和MR检查仅可见皮层下动脉硬化性脑病,CT灌注图像可见明显的脑局部血流缓慢。

Routine CT and MR examinations can only detect infracortical cerebral arteriosclerosis, whereas CT perfusion map ping images can detect evident slowed down cerebral focal blood flow

常规CT和MR检查仅可见皮层下动脉硬化性脑病,CT灌注图像可见明显的脑局部血流缓慢。

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