可以抑制的

If you can limit your exposure to an allergen, you can prevent or dampen your body's allergic reaction.

如果可以限制接触过敏原，那就可以防止或抑制身体的过敏反应。

(1) cerebral ischemical reperfusion injury rats'limbs motor function is variable. Acupuncture could promote lims'functional recovery.(2) PCNA masc cells is visible in cerebral ischemical semidarkness region. There is cell regeneration phenomenon. Acupuncture could strengthen injury region's PCNA expression, could profit injury recovery and functional reconstruction.(3) In ischemia semidarkness region for the model group and acupuncture group, PCNA masc cells percentage of 14days group is lower than 7days group. Along with the recovery of injury, cell multiplication is weaken.(4) In cerebral ischemia semidarkness region, there is VEGF masc cells and regeneration phenomenon. Acupuncture could strengthen injury region's VEGF expression, could profit protection after injury and blood vessel regenerate.(5) In ischemia semidarkness region for the model group and acupuncture group, VEGF masc cells percentage of 14days group is lower than 7days group. Along with the recovery of semidarkness region, ischemia and anoxemia state is getting improved, and VEGF is reduce.(6) As there are PCNA and VEGF masc cells in brain injured region, we could conclude that, after brain ischemical reperfusion injury, there are blood vessel regeneration phenomenon. Acupuncture could promote blood vessel regeneration, recovery blood supply sufficiently and quickly, and promote the recovery of brain injury region.(7)The VEGF masc cells percentage of inhibitor group is lower than acupuncture group. It state that the effect of acupuncture promote VEGF is partly depend on the existing of eNOS.

实验结论：(1)脑缺血再灌注损伤后大鼠的肢体运动功能发生改变，针刺可以促进肢体功能恢复；(2)脑缺损伤区可见PCNA阳性细胞，存在细胞再生现象，针刺可以增强损伤区PCNA的表达，有利于损伤的修复和功能重建；(3)针刺组和模型组14d时缺血损伤区PCNA阳性细胞百分比低于7d组，随着损伤逐渐得到修复，细胞增殖现象减弱；(4)脑缺血损伤区可见VEGF阳性细胞，存在内皮型细胞再生现象，针刺可以增强损伤区VEGF的表达，有利于脑损伤后保护和缺血区血管再生；(5)针刺组和模型组14d时缺血损伤区VEGF阳性细胞百分比低于7d组，随着缺血损伤的修复，缺血缺氧状态得到改善，产生的VEGF减少；(6)由于脑损伤区同时出现PCNA阳性细胞和VEGF阳性细胞，前者是增殖细胞的标志，后者是促进血管再生的重要因子，可以推断，脑缺血再灌注损伤后脑内存在血管再生现象，针刺可以促进损伤区的血管再生，更迅速而充分的恢复损伤区的血供，促进脑损伤区的修复；(7)抑制剂针刺组脑损伤区VEGF阳性细胞百分比与针刺组相比有不同程度的降低，说明针刺促进缺血损伤区VEGF表达部分依赖eNOS的存在。

Thus, inhibition of this mechanism could conceivably enhance the effect of norepinephrine.

因此可以想象，抑制这种机制可以增强去甲肾上腺素的作用。

Therefore,a sequence specific artificial zinc finger proteinis created when replacing the amino acids in the specific sites of C2H2 zinc finger protein and then fusing the protein to other activation or repression domains,while remaining the backbone of the zinc finger protein unchange...

ZFP可以介导靶基因的转录调控，抑制或激活特定基因的表达与配体依赖的靶基因激活或抑制；对DNA进行修饰，如人造限制性内切酶，重组酶，整合酶；抗病毒感染等。因此，人造锌指蛋白应用前景广阔，研究价值显著，是未来人类基因治疗的革命性的工具。

"Recent studies on the autoimmune origin of vitiligo also emphasize the probable implication of the immunosuppressive action of UV therapy in the repigmentation of vitiliginous plaques," the authors note."acrolimus, a new topical immunosuppressive drug developed for the treatment of atopic dermatitis, has shown some interesting results in treating vitiligo in 2 prospective studies."

研究人员指出，最近对於白斑自体免疫来源的实验，也强调了UV疗法的免疫抑制作用对於白斑再色素化可能的应用；Tacrolimus，一种新的局部免疫抑制药物，被开发用於治疗异位性皮肤炎，已经被两项前瞻性试验受瞩目的结果证实可以治疗白斑。

Sodium diacetate has the role in preventing the increasing of MDA and the decreasing of pectin in asparagus. All of the four preservatives cannot prevent the increasing of coarse fibre. The super absorbent polymer can effectively delay dewing in the package bags in a given time, but the available dosage and package material should be studied thoroughly.

双乙酸钠处理还可以较好地抑制MDA含量的增加并有延缓果胶分解的效果。4种防腐保鲜液处理均不能抑制芦笋纤维素含量的增加高吸水性树脂在一定时间内具有理想的防结露效果，但适宜使用剂量和包装方式尚需进一步研究。

In this investigation, TP could inhibit the ATPase's activity of P-gp in dose-dependent manner, indicating that TP exerted their modulation effect on MDR through inhibiting the ATPase activity of P-gp, and consequently interdicting the pumping function of P-gp on anti-cancer drug.

研究发现TP剂量依赖性的抑制ATPase活性，说明TP可以通过抑制P-gp糖蛋白的ATP酶活性的方式阻断P-gp糖蛋白对抗癌药物的溢出作用从而发挥其逆转MDR的作用。

After an analysis of the mechanisms of the effects of pressures on OMM, it is denoted that when the increase of ＂pressure＂ on the samples is the increase of effective stress, the stress acts directly on solid OM and the maturating rate will be accelerated.

综合分析结果表明，当压力的增加表现为沉积有机质受到的有效应力增加时可以促进有机质生烃；当压力的增加表现为孔隙流体压力增加时，有机质生烃过程将受到抑制；另外，这两种情况都可能使干酪根的镜质组反射率降低，易使人得出压力增加抑制有机质演化的结论。

The positive expression rates of Tissue Inhibitors of Metalloproteinase-2 protein in the cases of involuting groups were significantly higher than those in proliferating group and vascular malformation groups and normal skin group (P.05). Its difference was significant. Conclusions: Matrix MetallProtinase-2 and Tissue Inhibitors of Metalloproteinase-2 may play an important role in the pathogenesis of hemangiomas through angiogenesis. They may have no effect on vascular malformation and normal skin

血管瘤退化期，TIMP-2表达明显升高，说明 TIMP-2与血管瘤纤维化、消退有关，其机制可能是一方面通过抑制MMP-2介导的基底膜降解而在内皮细胞迁移、聚集中起作用，另一方面直接抑制内皮细胞的增殖及迁移，使内皮细胞高频率调亡，细胞外基质增多，组织出现纤维化，最终导致血管瘤的退化。2、血管畸形和正常皮肤小血管中MMP-2和TIMP-2不表达或表达较弱，说明血管畸形和正常皮肤小血管中并不存在异常的血管生成，据此可以对血管性疾病进行分型、分期，并指导临床治疗。

Using t-tset to analyze significance of difference. Results: 1. H101 could cause cytopathic effects in U251 cells from 24h and shows greater oncolysis effects when much more H101mRNA expression by enhanced virus tite or prolonged time.

结果： 1。 H101病毒在24小时就可以对体外培养的胶质母细胞瘤U251产生抑制作用，加入的病毒滴度越大抑制率越高，并且随着病毒作用时间的延长，细胞病变反应越发明显，H101mRNA的表达呈上升趋势。2。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。