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Abstract] Objective:To study the synergistic induction of apoptosis by the combination of antihuman DR5(death receptor 5) monoclonal antibody(mDRA6) and cisplatin on HL60 cells.

目的:观察抗死亡受体5(Death receptor 5,DR5)单克隆抗体——mDRA6与顺铂对HL60细胞的协同杀伤作用。

The immunohistochemistric staining technique and image Analysis technique were adopted to investigate the expression of NGF and its receptor in salivary gland tissues around neoplasm,ACC and MEC with rabbit antihuman NGF TrkA and p75 polyclonal antibody.

采用免疫组织化学染色技术及图像分析技术,用兔抗人NGF、TrkA及p75多克隆抗体,对肿瘤周围涎腺组织、ACC和MEC进行神经生长因子及其受体表达研究。

The parental BHK and the transfectants stably expressing human DR4, DR5, DcR1, or DcR2 were stained with control mouse IgG or murine antihuman DR4, DR5, DcR1, or DcR2 mAbs and analyzed by flow cytometry. Dotted and solid lines correspondent to control and specific stainings, respectiely. Cell surface staining with anti-DR4, anti-DR5 and anti-Fas mAbs.

抗人TRAIL受体的单克隆抗体的特异性:用小鼠IgG或鼠抗人DR4、DR5、DcR1及DcR2单克隆抗体对未转染的BHK细胞以及分别转染了人DR4、DR5、DcR1及DcR2并稳定表达的BHK细胞进行免疫组化染色,然后做流式细胞分析,点线和实线分别对应于对照及特异性染色。

The GABAB agonist baclofen is approved as an antispasmodic, but has been used to inhibit TLESR and reflux in humans.

巴氯芬是一种B型GABA受体激动剂,被批准作为解痉剂使用,但在人类已被用于抑制TLESR及反流。

AIM: To observe the effect of Nourishing Yin Antitoxic Capsule 2 (YC2) on the expression of glucocorticoid receptor proteins in Yin deficiency rats induced by hydrocortisone, and to investigate how the YC2 relieves the side effects of Glucocorticoid.

目的:观察养阴抗毒胶囊2号(YC2)对阴虚大鼠模型肝组织糖皮质激素受体蛋白表达的和对减轻激素副作用机理。

There is up-regulation of striatum D2 receptors in lesioned side in experimental hemi-parkinsonism which show good correlation with rotation behavior induced by Apomorphine, and coincided with the pathological findings.

大鼠偏侧PD模型患侧纹状体D2受体出现上调反应,这种上调反应与APO诱导的行为学改变呈良好的相关性,与病理组织学改变相符合。

The rotations of PD rats induced by apomorphine could be inhibited by injection of CCK-8 into venous and nucleus accumbens of the lesioned sides.

静脉注射CCK-8及损毁侧伏核微量注射CCK-8可抑制PD大鼠模型由阿朴吗啡诱导的旋转行为,这种作用由CCK-A受体介导。

Chronic ions exist in organism in the form of glucose tolerance factors, which are likely to stimulate the metabolism of glucose and lipid through increasing the appetency of insulin, the number of insulin receptor and the function of P cell.

三价铬在生物体内是以葡萄糖耐量因子的形式存在的,可能是通过增强胰岛素的亲和力、胰岛素的受体数目以及β细胞的功能来促进糖和脂代谢的。

One of the problems we have seen I that when we give a drug that reduces appetitive like the Melanocortin 4 receptor agonist, that that tends to raise the blood pressure and of course that is undesirable in treating hypertensive patients to have a further rise in blood pressure so we would like to understand the molecular signaling pathways in the brain that allow for appetite, metabolic function, and blood pressure to be differentially controlled and therefore attack those different targets in treating not only obesity but also hypertension.

我们所遇到的一个难题是,当我们使用一种药物减低食欲,如黑皮质素受体4,有可能会增加血压。当然,血压进一步升高对于治疗中高血压患者是不受欢迎的。所以在高血压和肥胖的治疗中,我宁愿了解脑影响食欲、代谢功能和血压的分子信号途径,由此寻找差异性控制、进而治疗不同的目标。

Armando et al9 showed that intact female mice have higher AT2R expression in the kidney compared with ovariectomized females.

Armando等19人研究显示正常的成年雌鼠比卵巢切除后雌鼠肾脏血管紧张素II受体表达水平高。

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