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During periimplantation, the decrease of the ECL and the PHAL receptors may lead to the failure of implantation in the oviduct, and the increase of the UEA receptors at diestrus may contribute to the process of fertilization.

围植入期ECL和PHAL受体表达下降,在输卵管内形成了不利于植入的微环境,可能是胚泡正常情况下不植入输卵管的原因之一;动情期UEA受体水平显著低于动情间期,有利于受精过程的正常进行。

Upon FGF binding, specific membrane receptors dimerize and autophosphorylate, then activate several downstream signaling pathways including Ras-MAPK.

FGF通过结合细胞表面的FGF受体,使其二聚化并自体磷酸化,活化后的FGF受体具有酪氨酸激酶活性,进而活化胞内下游多条信号通路。

Expression of human β-arrestin 1 attenuated κ andδ opioid receptor subtype-mediated G protein activation and inhibition of cAMP production, while did not affect the concentration- effect relationship ofμ-agonist 〓-enkephalin in the same signalings.

arrestin 1的表达明显减弱了κ型,δ型阿片受体亚型所介导的G蛋白的激活和腺苷酸环化酶活性的抑制,但是却不影响μ型阿片受体亚型特异性激动剂〓-enkephalin刺激引起的浓度效应曲线。

However, the activation of ERKs was significantly suppressed after the COS7 cells were transfected with C289A and Q257A. The result indicated that C289A and Q257A were the stretch sensitive region.

结果提示AT1受体上位于257位的谷氨酰胺和289位的胱氨酸在机械牵张引起的AT1受体激活中起了重要作用。

RESULTS:(1) ET-1 could increase total protein production, surface area, ERKs activity and [Ca2+]i in cultured cardiomyocyte in dose-dependent manner at concentrations ranging from 10-9 to 10-7 mol/L. And this effect could be abolished by BQ123, an antagonist of ETA receptor, partly inhibited by PTX, but not by BQ788, an antagonist of ETB receptor.(2)The activation of ERKs and the increase of [Ca2+]i induced by ET-1 were obviously inhibited by PD98059, a selective ERKs kinase inhibitor, and nifedipine, a calcium channel blocker, respectively. Both antagonists partially inhibited ET-1-stimulated cardiomyocyte hypertrophic response.(3) Staurosporine, a selective PKC inhibitor, could inhibit ET-1-stimulated cardiomyocyte hypertrophic response and increase of [Ca2+]i, but not affect the activation of ERKs.

结果: ①ET-1浓度依赖性增加新生大鼠心肌细胞蛋白质含量和心肌细胞表面积、ERKs活性及[Ca2+]i浓度,以上作用可被ETA受体拮抗剂BQ123所完全抑制,被百日咳毒素部分抑制,而ETB受体拮抗剂BQ788则无效;②ERKs激酶特异性抑制剂PD98059可完全抑制ET-1激活ERKs的作用,钙通道拮抗剂硝苯地平可明显抑制ET-1介导的[Ca2+]i浓度增加,但二者皆仅部分抑制ET-1介导的心肌细胞肥大反应;③蛋白激酶C选择性抑制剂staurosporine并不能明显抑制ET-1介导的ERKs激活,但可抑制ET-1介导的的[Ca2+]i浓度增加及心肌细胞肥大反应。

In order to investigate the relationship between vulvar dystro phy, malignant tumor, and estrogen, progestin receptor, we made an analysis on estro gen and progestin receptor contents in 31 cases of vulvar dystrophy and 19 cases of malignant tumor, using the dextran-coated charcoal method. The Res u lts showed that ER content of the mixed type was higher than that of the atroph ic type.

为了探讨雌、孕激素受体与外阴营养不良、恶性肿瘤的关系,用葡聚糖-活性碳单点饱和分析法测定了31例外阴营养不良(增生型 14例、混合型10例、萎缩型7例)和19例外阴恶性肿瘤(外阴鳞形细胞癌16例)的雌、孕激素受体含量。

Tolllike receptor has been found a newly innate immunoreceptor. It is significant in immune response. Along with further investigation of TLR, infection and antiinfection immunity theory would gain a new breakthrough and play a guiding role in clinical treatment of acute leukemia complicated with infection.

To11样受体(Tolllike receptor, TLR)是新近发现的天然免疫受体,在免疫应答中的重要性受到广泛关注,对TLR研究的进一步深入,必将使感染、抗感染免疫的理论研究取得新的突破,并对急性白血病合并感染等的临床治疗起指导性作用。

Receptor system of transformation have developed from meristems to callus receptor ; and the gene transformation methods have made some progress.

香蕉转化受体系统已从茎尖发展到胚状体和愈伤组织受体系统,遗传转化方法也在不断改进,从单一的方法发展为根癌农杆菌感染法与基因枪法的结合。

The sciatic nerves on the right side of all animals were exposed and 10 cm long segments were removed from the mid-thigh level and replaced by one of the three types of grafts of Sprague-Dawley cat.

另取30只Sprague Dawley大鼠随机分成冻融法联合改良化学法受体组15只,新鲜异体神经受体15只。

Results The pD2 value (the affinity to M-receptors of agonist) of Erycibele alkaloid's miosis is 3.60±0.15 and its IOP lowering action is 3.49±0.07. In the isolated iris contracting experiment, its pD2 value is 6.38±0.12. M3 receptor antagonist is the strongest antagonist in inhibiting the miosis, IOP lowering action and isolated iris pupillary contracting action of Erycibele alkaloid. 0.01% Erycibele alkaloid can cause the decrease of cAMP and the increase of cGMP in the aqueous humor.

结果 测定丁公藤碱缩瞳作用的pD2值为3.60±0.15,降眼压作用的pD2值为3.49±0.07,离体虹膜收缩作用的pD2值为6.38±0.12.M1~M3受体拮抗剂均可拮抗丁公藤碱的缩瞳作用、降眼压作用及离体虹膜收缩作用,其中以M3受体拮抗剂的拮抗作用最强。0.01%丁公藤碱可使房水环磷酸腺苷(cyclic adenosine monophosphate,cAMP)含量降低,环磷酸鸟苷(cyclic guanosine 3,5-monophosphate;cGMP)含量升高。

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