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The PA2 value for α blocking were 7.46±0.20 (n=8) in rat anococcygeal muscles and 7.11±0.27 (n=8) for β blocking in guinea pig atrium.

阻断α1受体的PA2为7.46±0.20(n=8);阻断β1受体的PA2值为7.11±0.27(n=8)。

Anthropometrical measurements were performed and serum concentrations of IGF-I, insulin-like growth factor binding protein-3 (IGFBP-3), leptin, soluble leptin receptor, as well as the estimated molar excess of sOB-R over leptin were obtained. Two groups were formed.

执行人体测量,获得了胰岛素样生长因子-1、胰岛素样生长因子连接蛋白-3(IGFBP-3)、瘦素、可溶性瘦素受体的血清浓度和可溶性瘦素受体超过瘦素的估计摩尔量。

Anthropometrical measurements were performed and serum concentrations of IGF-I, insulin-like growth factor binding protein-3 (IGFBP-3), leptin, soluble leptin receptor, as well as the estimated molar excess of sOB-R oer leptin were obtained. Two groups were formed.

执行人体测量,获得了胰岛素样生长因子-1、胰岛素样生长因子连接蛋白-3(IGFBP-3)、瘦素、可溶性瘦素受体的血清浓度和可溶性瘦素受体超过瘦素的估计摩尔量。

An amide-based fluorescent acyclic receptor (II -1) consisting of anthryl group was synthesized, and the anion binding property was studied in CHCI3 by fluorescence, UV-vis and H NMR spectra.

合成了带有蒽荧光团的非环状酰胺受体分子Ⅱ-1,通过荧光、UV-vis光谱等研究了受体在CHCl_3中识别阴离子的性质。

The increased ARNA produced by intrapelvic infusion of DADLE was unaffected by pretreatment of rats with the NK-1 receptor antagonist, spantide I (0.1mg/ml, 20μl/min).

另外也发现,由肾盂灌注类鸦片拮抗剂,并不会改变因肾盂内压力增加而活化的肾压力性受体时ARNA的增加;而灌注DADLE也不会改变因肾盂灌注高张食盐水(600mM NaCl)而活化肾化学性受体时ARNA的增加。

Autoradiographic studies have demonstrated that adenosine A1 and A2 receptors are expressed in the spinal dorsal horn, both on the post-synaptic SG neurons and the central terminals of the primary afferent neurons.

放射自显影的结果表明A_1和A_2受体都分布在脊髓背角,包括突触后SG神经元和初级传入神经元的中枢突,而A_1受体集中分布于胶状质。

The mRNA and protein of CD14 and TLR4 in U937 cells were detected by semi-quantitative RT-PCR and Western blotting.

RT-PCR 和Western blotting检测U937 细胞膜受体CD_(14)和跨膜受体

And we found that carnosine increased the cell surface expression of NMDA receptor, which was abolished by clobenpropit.

我们发现肌肽能引起NMDA受体在细胞膜上表达增加,而H_3受体拮抗剂clobenpropit能完全阻断这一现象。

Then we examine whether NMDA receptor is involved in the protective effect of carnosine on Aβ42-induced neurotoxicity because of the important role of NMDA receptor in AD.

由于NMDA受体在AD病理过程中的重要地位,本实验进一步检测NMDA受体的表达变化。

Finally, we found that MK801, a complete antagonist of NMDA receptor, abolished increasing cell surface expression of NMDA receptor afforded by carnosine or clobenpropit.

最后我们还发现NMDA受体完全阻断剂MK801能阻断肌肽和clobenpropit引起的NMDA受体膜上表达增加。

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