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发病

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Objective: To evaluate the role of LPA and FIB inpathogenesis of ischemic stroke.

目的:探讨溶血磷脂酸及纤维蛋白原在缺血性卒中发病中的作用。

Objective: To analyze age, etiology and clinical character of patients with atrial fibrillation.

目的:分析心房纤颤患者的发病年龄与病因、临床特点的关系。

Objective To discuss the role of plasmatic fibrinogen in the pathogenesis of coronary heart disease.

目的 探讨血浆纤维蛋白原在冠心病发病中的作用。

It is suggested that fibrinogen in plasma takes an important role in the the pathogenesis of CHD.

血浆纤维蛋白原在冠心病的发病中可能起重要作用。

The high levels of fibrinogen are one of the risk factors in acute progressive cerebral infarction.

发病后高纤维蛋白原水平为急性进展性脑梗死的危险因素。

Objective To investigate the role of basic fibroblast growth factorin the mechanism of vascular dementia.

目的 探讨碱性成纤维细胞生长因子在血管性痴呆的发病机制中的作用。

Objective To investigate the role of platelet-derived growth factor receptor in the development of scleroderma fibrosis.

目的 探讨血小板衍生生长因子在硬皮病发病中的作用。

Objective To observe the expression of nicotinic acetylcholine receptor in the brain's fimbria/fornix transected rats, the effect of ectogenic estrogen on it, and its relation with Alzhelmer's disease.

目的 以双侧穹隆海马伞切断制作阿尔茨海默病大鼠模型,观察脑内海马CA1区和皮层区烟碱型乙酰胆碱受体表达的变化,探讨与 AD相关的发病机制,并观察雌激素的干预作用。

Objective To observe the expressive varieties of β-amyloid(A β1-40)peptide in different sections of the rat brain with fimbria/fornix transaction and to study the pathogenic mechanisms related to Alzheimer disease.

目的 采用穹隆-海马伞切断及去势的大鼠模型模拟阿尔茨海默病病人,观察模型大鼠脑内不同部位β淀粉肽1-40(Aβ1-40)表达的变化,了解A β1-40与阿尔茨海默病相关的发病机制。

Research of ultramicro- structure of anal sphincter in the cases of patients with fecal incontinence shows : shortness of smooth muscle cells of anal sphincter , destruction of normal structure of the rest cells , elongation of elastic tissue and increase of collagen fibers. Thus the most familiar cause of copracrasia is Degeneration of finespun smooth muscle of anal sphincter which mantain anal sphincter closed .One of the pathomechanism of fecal incontinence is decrease of anal resting pressure .

对大便失禁者肛门内括约肌的超微结构研究发现:肛门内括约肌的平滑肌细胞缺失、剩余细胞的正常关系破坏、弹性组织拉伸、胶原纤维成分增加,因而大便溢漏最常见的原因是维持括约肌关闭的肌肉-肛门内括约肌的纤细平滑肌的退行性病变,肛管静息压降低是大便失禁发病的一个病理学机制。

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